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Literature DB >> 30287420

Right time, right place-DNA damage and DNA replication checkpoints collectively safeguard S phase.

Lorenzo Galanti¹, Boris Pfander¹.

Abstract

Mesh：

Substances：
DNA, Fungal

Year: 2018 PMID： 30287420 PMCID： PMC6213279 DOI： 10.15252/embj.2018100681

Source DB: PubMed Journal: EMBO J ISSN： 0261-4189 Impact factor: 11.598

× No keyword cloud information.

15 in total

1. Mrc1 transduces signals of DNA replication stress to activate Rad53.

Authors: A A Alcasabas; A J Osborn; J Bachant; F Hu; P J Werler; K Bousset; K Furuya; J F Diffley; A M Carr; S J Elledge
Journal: Nat Cell Biol Date: 2001-11 Impact factor: 28.824

2. The RAD6 DNA damage tolerance pathway operates uncoupled from the replication fork and is functional beyond S phase.

Authors: Georgios I Karras; Stefan Jentsch
Journal: Cell Date: 2010-04-16 Impact factor: 41.582

3. Multiple mechanisms control chromosome integrity after replication fork uncoupling and restart at irreparable UV lesions.

Authors: Massimo Lopes; Marco Foiani; José M Sogo
Journal: Mol Cell Date: 2006-01-06 Impact factor: 17.970

Review 4. The checkpoint response to replication stress.

Authors: Dana Branzei; Marco Foiani
Journal: DNA Repair (Amst) Date: 2009-05-23

5. Mrc1 and Rad9 cooperate to regulate initiation and elongation of DNA replication in response to DNA damage.

Authors: Julien Bacal; María Moriel-Carretero; Benjamin Pardo; Antoine Barthe; Sushma Sharma; Andrei Chabes; Armelle Lengronne; Philippe Pasero
Journal: EMBO J Date: 2018-08-29 Impact factor: 11.598

6. Redox-sensitive alteration of replisome architecture safeguards genome integrity.

Authors: Kumar Somyajit; Rajat Gupta; Hana Sedlackova; Kai John Neelsen; Fena Ochs; Maj-Britt Rask; Chunaram Choudhary; Jiri Lukas
Journal: Science Date: 2017-11-10 Impact factor: 47.728

Review 7. Eukaryotic DNA damage checkpoint activation in response to double-strand breaks.

Authors: Karen Finn; Noel Francis Lowndes; Muriel Grenon
Journal: Cell Mol Life Sci Date: 2011-11-15 Impact factor: 9.261

8. Activation of Rad53 kinase in response to DNA damage and its effect in modulating phosphorylation of the lagging strand DNA polymerase.

Authors: A Pellicioli; C Lucca; G Liberi; F Marini; M Lopes; P Plevani; A Romano; P P Di Fiore; M Foiani
Journal: EMBO J Date: 1999-11-15 Impact factor: 11.598

Right time, right place-DNA damage and DNA replication checkpoints collectively safeguard S phase.

1. Mrc1 transduces signals of DNA replication stress to activate Rad53.

2. The RAD6 DNA damage tolerance pathway operates uncoupled from the replication fork and is functional beyond S phase.

3. Multiple mechanisms control chromosome integrity after replication fork uncoupling and restart at irreparable UV lesions.

Review 4. The checkpoint response to replication stress.

5. Mrc1 and Rad9 cooperate to regulate initiation and elongation of DNA replication in response to DNA damage.

6. Redox-sensitive alteration of replisome architecture safeguards genome integrity.

Review 7. Eukaryotic DNA damage checkpoint activation in response to double-strand breaks.

8. Activation of Rad53 kinase in response to DNA damage and its effect in modulating phosphorylation of the lagging strand DNA polymerase.

9. Ubiquitin-dependent DNA damage bypass is separable from genome replication.

10. Checkpoint-dependent phosphorylation of Exo1 modulates the DNA damage response.

Review 1. The multi-functionality of UHRF1: epigenome maintenance and preservation of genome integrity.

Review 2. Homologous Recombination: To Fork and Beyond.

Review 3. On the Interplay of the DNA Replication Program and the Intra-S Phase Checkpoint Pathway.