| Literature DB >> 32220291 |
Tiong Yang Tan1, Jiří Sedmík2, Mark P Fitzgerald3, Rivka Sukenik Halevy4, Liam P Keegan2, Ingo Helbig3, Lina Basel-Salmon5, Lior Cohen6, Rachel Straussberg7, Wendy K Chung8, Mayada Helal8, Reza Maroofian9, Henry Houlden9, Jane Juusola10, Simon Sadedin11, Lynn Pais12, Katherine B Howell13, Susan M White14, John Christodoulou14, Mary A O'Connell15.
Abstract
The RNA editing enzyme ADAR2 is essential for the recoding of brain transcripts. Impaired ADAR2 editing leads to early-onset epilepsy and premature death in a mouse model. Here, we report bi-allelic variants in ADARB1, the gene encoding ADAR2, in four unrelated individuals with microcephaly, intellectual disability, and epilepsy. In one individual, a homozygous variant in one of the double-stranded RNA-binding domains (dsRBDs) was identified. In the others, variants were situated in or around the deaminase domain. To evaluate the effects of these variants on ADAR2 enzymatic activity, we performed in vitro assays with recombinant proteins in HEK293T cells and ex vivo assays with fibroblasts derived from one of the individuals. We demonstrate that these ADAR2 variants lead to reduced editing activity on a known ADAR2 substrate. We also demonstrate that one variant leads to changes in splicing of ADARB1 transcript isoforms. These findings reinforce the importance of RNA editing in brain development and introduce ADARB1 as a genetic etiology in individuals with intellectual disability, microcephaly, and epilepsy.Entities:
Keywords: ADAR2; RNA editing; epilepsy; intellectual disability; microcephaly; migrating focal seizures
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Year: 2020 PMID: 32220291 PMCID: PMC7118584 DOI: 10.1016/j.ajhg.2020.02.015
Source DB: PubMed Journal: Am J Hum Genet ISSN: 0002-9297 Impact factor: 11.025