Literature DB >> 31866242

Cumulative Burden of Colorectal Cancer-Associated Genetic Variants Is More Strongly Associated With Early-Onset vs Late-Onset Cancer.

Alexi N Archambault1, Yu-Ru Su2, Jihyoun Jeon3, Minta Thomas2, Yi Lin2, David V Conti4, Aung Ko Win5, Lori C Sakoda6, Iris Lansdorp-Vogelaar7, Elisabeth F P Peterse7, Ann G Zauber8, David Duggan9, Andreana N Holowatyj10, Jeroen R Huyghe2, Hermann Brenner11, Michelle Cotterchio12, Stéphane Bézieau13, Stephanie L Schmit14, Christopher K Edlund4, Melissa C Southey15, Robert J MacInnis16, Peter T Campbell17, Jenny Chang-Claude18, Martha L Slattery19, Andrew T Chan20, Amit D Joshi21, Mingyang Song22, Yin Cao23, Michael O Woods24, Emily White25, Stephanie J Weinstein26, Cornelia M Ulrich10, Michael Hoffmeister27, Stephanie A Bien2, Tabitha A Harrison2, Jochen Hampe28, Christopher I Li2, Clemens Schafmayer29, Kenneth Offit30, Paul D Pharoah31, Victor Moreno32, Annika Lindblom33, Alicja Wolk34, Anna H Wu4, Li Li35, Marc J Gunter36, Andrea Gsur37, Temitope O Keku38, Rachel Pearlman39, D Timothy Bishop40, Sergi Castellví-Bel41, Leticia Moreira41, Pavel Vodicka42, Ellen Kampman43, Graham G Giles44, Demetrius Albanes45, John A Baron46, Sonja I Berndt45, Stefanie Brezina37, Stephan Buch10, Daniel D Buchanan47, Antonia Trichopoulou48, Gianluca Severi49, María-Dolores Chirlaque50, Maria-José Sánchez51, Domenico Palli52, Tilman Kühn53, Neil Murphy54, Amanda J Cross55, Andrea N Burnett-Hartman56, Stephen J Chanock45, Albert de la Chapelle57, Douglas F Easton58, Faye Elliott40, Dallas R English44, Edith J M Feskens43, Liesel M FitzGerald59, Phyllis J Goodman60, John L Hopper61, Thomas J Hudson62, David J Hunter63, Eric J Jacobs17, Corinne E Joshu64, Sébastien Küry65, Sanford D Markowitz34, Roger L Milne16, Elizabeth A Platz64, Gad Rennert66, Hedy S Rennert66, Fredrick R Schumacher67, Robert S Sandler38, Daniela Seminara68, Catherine M Tangen60, Stephen N Thibodeau69, Amanda E Toland57, Franzel J B van Duijnhoven43, Kala Visvanathan64, Ludmila Vodickova42, John D Potter2, Satu Männistö70, Korbinian Weigl71, Jane Figueiredo72, Vicente Martín73, Susanna C Larsson74, Patrick S Parfrey75, Wen-Yi Huang26, Heinz-Josef Lenz76, Jose E Castelao77, Manuela Gago-Dominguez78, Victor Muñoz-Garzón79, Christoph Mancao80, Christopher A Haiman4, Lynne R Wilkens81, Erin Siegel82, Elizabeth Barry83, Ban Younghusband24, Bethany Van Guelpen84, Sophia Harlid85, Anne Zeleniuch-Jacquotte1, Peter S Liang86, Mengmeng Du8, Graham Casey87, Noralane M Lindor88, Loic Le Marchand81, Steven J Gallinger89, Mark A Jenkins5, Polly A Newcomb90, Stephen B Gruber91, Robert E Schoen92, Heather Hampel39, Douglas A Corley93, Li Hsu94, Ulrike Peters95, Richard B Hayes96.   

Abstract

BACKGROUND & AIMS: Early-onset colorectal cancer (CRC, in persons younger than 50 years old) is increasing in incidence; yet, in the absence of a family history of CRC, this population lacks harmonized recommendations for prevention. We aimed to determine whether a polygenic risk score (PRS) developed from 95 CRC-associated common genetic risk variants was associated with risk for early-onset CRC.
METHODS: We studied risk for CRC associated with a weighted PRS in 12,197 participants younger than 50 years old vs 95,865 participants 50 years or older. PRS was calculated based on single nucleotide polymorphisms associated with CRC in a large-scale genome-wide association study as of January 2019. Participants were pooled from 3 large consortia that provided clinical and genotyping data: the Colon Cancer Family Registry, the Colorectal Transdisciplinary Study, and the Genetics and Epidemiology of Colorectal Cancer Consortium and were all of genetically defined European descent. Findings were replicated in an independent cohort of 72,573 participants.
RESULTS: Overall associations with CRC per standard deviation of PRS were significant for early-onset cancer, and were stronger compared with late-onset cancer (P for interaction = .01); when we compared the highest PRS quartile with the lowest, risk increased 3.7-fold for early-onset CRC (95% CI 3.28-4.24) vs 2.9-fold for late-onset CRC (95% CI 2.80-3.04). This association was strongest for participants without a first-degree family history of CRC (P for interaction = 5.61 × 10-5). When we compared the highest with the lowest quartiles in this group, risk increased 4.3-fold for early-onset CRC (95% CI 3.61-5.01) vs 2.9-fold for late-onset CRC (95% CI 2.70-3.00). Sensitivity analyses were consistent with these findings.
CONCLUSIONS: In an analysis of associations with CRC per standard deviation of PRS, we found the cumulative burden of CRC-associated common genetic variants to associate with early-onset cancer, and to be more strongly associated with early-onset than late-onset cancer, particularly in the absence of CRC family history. Analyses of PRS, along with environmental and lifestyle risk factors, might identify younger individuals who would benefit from preventive measures.
Copyright © 2020 AGA Institute. Published by Elsevier Inc. All rights reserved.

Entities:  

Keywords:  Colon Cancer; EOCRC; Penetrance; SNP

Mesh:

Year:  2019        PMID: 31866242      PMCID: PMC7103489          DOI: 10.1053/j.gastro.2019.12.012

Source DB:  PubMed          Journal:  Gastroenterology        ISSN: 0016-5085            Impact factor:   33.883


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