Literature DB >> 30697902

Genome-wide association study of inhaled corticosteroid response in admixed children with asthma.

Natalia Hernandez-Pacheco1,2, Niloufar Farzan3,4, Ben Francis5, Leila Karimi6, Katja Repnik7,8, Susanne J Vijverberg3,4, Patricia Soares9, Maximilian Schieck10,11, Mario Gorenjak7, Erick Forno12, Celeste Eng13, Sam S Oh13, Lina Pérez-Méndez14,15, Vojko Berce7,16, Roger Tavendale17, Lesly-Anne Samedy13,18, Scott Hunstman13, Donglei Hu13, Kelley Meade19, Harold J Farber20, Pedro C Avila21,22, Denise Serebrisky23, Shannon M Thyne24, Emerita Brigino-Buenaventura25, William Rodriguez-Cintron26, Saunak Sen27, Rajesh Kumar28,29, Michael Lenoir30, Jose R Rodriguez-Santana31, Juan C Celedón12, Somnath Mukhopadhyay9,17, Uroš Potočnik7,8, Munir Pirmohamed32, Katia M Verhamme6, Michael Kabesch10, Colin N A Palmer17, Daniel B Hawcutt5,33, Carlos Flores1,15,34, Anke H Maitland-van der Zee3,4,35, Esteban G Burchard13,18, Maria Pino-Yanes1,2,15.   

Abstract

BACKGROUND: Inhaled corticosteroids (ICS) are the most widely prescribed and effective medication to control asthma symptoms and exacerbations. However, many children still have asthma exacerbations despite treatment, particularly in admixed populations, such as Puerto Ricans and African Americans. A few genome-wide association studies (GWAS) have been performed in European and Asian populations, and they have demonstrated the importance of the genetic component in ICS response.
OBJECTIVE: We aimed to identify genetic variants associated with asthma exacerbations in admixed children treated with ICS and to validate previous GWAS findings.
METHODS: A meta-analysis of two GWAS of asthma exacerbations was performed in 1347 admixed children treated with ICS (Hispanics/Latinos and African Americans), analysing 8.7 million genetic variants. Those with P ≤ 5 × 10-6 were followed up for replication in 1697 asthmatic patients from six European studies. Associations of ICS response described in published GWAS were followed up for replication in the admixed populations.
RESULTS: A total of 15 independent variants were suggestively associated with asthma exacerbations in admixed populations (P ≤ 5 × 10-6 ). One of them, located in the intergenic region of APOBEC3B and APOBEC3C, showed evidence of replication in Europeans (rs5995653, P = 7.52 × 10-3 ) and was also associated with change in lung function after treatment with ICS (P = 4.91 × 10-3 ). Additionally, the reported association of the L3MBTL4-ARHGAP28 genomic region was confirmed in admixed populations, although a different variant was identified. CONCLUSIONS AND CLINICAL RELEVANCE: This study revealed the novel association of APOBEC3B and APOBEC3C with asthma exacerbations in children treated with ICS and replicated previously identified genomic regions. This contributes to the current knowledge about the multiple genetic markers determining responsiveness to ICS which could lead in the future the clinical identification of those asthma patients who are not able to respond to such treatment.
© 2019 John Wiley & Sons Ltd.

Entities:  

Keywords:  African American; Latino; childhood asthma; exacerbations; pharmacogenomics

Mesh:

Substances:

Year:  2019        PMID: 30697902      PMCID: PMC7054824          DOI: 10.1111/cea.13354

Source DB:  PubMed          Journal:  Clin Exp Allergy        ISSN: 0954-7894            Impact factor:   5.018


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