Literature DB >> 33303529

Genome-wide association study of asthma exacerbations despite inhaled corticosteroid use.

Natalia Hernandez-Pacheco1,2, Susanne J Vijverberg3,4,5, Esther Herrera-Luis2, Jiang Li6, Yang Yie Sio7, Raquel Granell8, Almudena Corrales1,9, Cyrielle Maroteau10, Ryan Lethem8, Javier Perez-Garcia2, Niloufar Farzan3,4,11, Katja Repnik12,13, Mario Gorenjak12, Patricia Soares14,15, Leila Karimi16, Maximilian Schieck17,18, Lina Pérez-Méndez1,9,19, Vojko Berce12,20, Roger Tavendale21, Celeste Eng22, Olaia Sardon23,24, Inger Kull25, Somnath Mukhopadhyay14,21, Munir Pirmohamed26, Katia M C Verhamme16, Esteban G Burchard22,27, Michael Kabesch17, Daniel B Hawcutt28,29, Erik Melén25,30, Uroš Potočnik12,13, Fook Tim Chew7, Kelan G Tantisira6,31, Steve Turner32, Colin N Palmer21, Carlos Flores1,9,33,34, Maria Pino-Yanes35,2,9,34,36, Anke H Maitland-van der Zee3,4,5,36.   

Abstract

RATIONALE: Substantial variability in response to asthma treatment with inhaled corticosteroids (ICS) has been described among individuals and populations, suggesting the contribution of genetic factors. Nonetheless, only a few genes have been identified to date. We aimed to identify genetic variants associated with asthma exacerbations despite ICS use in European children and young adults and to validate the findings in non-Europeans. Moreover, we explored whether a gene-set enrichment analysis could suggest potential novel asthma therapies.
METHODS: A genome-wide association study (GWAS) of asthma exacerbations was tested in 2681 children of European descent treated with ICS from eight studies. Suggestive association signals were followed up for replication in 538 European asthma patients. Further evaluation was performed in 1773 non-Europeans. Variants revealed by published GWAS were assessed for replication. Additionally, gene-set enrichment analysis focused on drugs was performed.
RESULTS: 10 independent variants were associated with asthma exacerbations despite ICS treatment in the discovery phase (p≤5×10-6). Of those, one variant at the CACNA2D3-WNT5A locus was nominally replicated in Europeans (rs67026078; p=0.010), but this was not validated in non-European populations. Five other genes associated with ICS response in previous studies were replicated. Additionally, an enrichment of associations in genes regulated by trichostatin A treatment was found.
CONCLUSIONS: The intergenic region of CACNA2D3 and WNT5A was revealed as a novel locus for asthma exacerbations despite ICS treatment in European populations. Genes associated were related to trichostatin A, suggesting that this drug could regulate the molecular mechanisms involved in treatment response.
Copyright ©ERS 2021.

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Year:  2021        PMID: 33303529      PMCID: PMC8122045          DOI: 10.1183/13993003.03388-2020

Source DB:  PubMed          Journal:  Eur Respir J        ISSN: 0903-1936            Impact factor:   16.671


  65 in total

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6.  Exacerbation-prone asthma in the context of race and ancestry in Asthma Clinical Research Network trials.

Authors:  Nicole L Grossman; Victor E Ortega; Tonya S King; Eugene R Bleecker; Elizabeth A Ampleford; Leonard B Bacharier; Michael D Cabana; Juan C Cardet; Tara F Carr; Mario Castro; Loren C Denlinger; Joshua L Denson; Nicolas Fandino; Anne M Fitzpatrick; Gregory A Hawkins; Fernando Holguin; Jerry A Krishnan; Stephen C Lazarus; Sharmilee M Nyenhuis; Wanda Phipatanakul; Sima K Ramratnam; Sally Wenzel; Stephen P Peters; Deborah A Meyers; Michael E Wechsler; Elliot Israel
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Authors:  Ralf Jp van der Valk; Liesbeth Duijts; Nicolas J Timpson; Muhammad T Salam; Marie Standl; John A Curtin; Jon Genuneit; Marjan Kerhof; Eskil Kreiner-Møller; Alejandro Cáceres; Anna Gref; Liming L Liang; H Rob Taal; Emmanuelle Bouzigon; Florence Demenais; Rachel Nadif; Carole Ober; Emma E Thompson; Karol Estrada; Albert Hofman; André G Uitterlinden; Cornélia van Duijn; Fernando Rivadeneira; Xia Li; Sandrah P Eckel; Kiros Berhane; W James Gauderman; Raquel Granell; David M Evans; Beate St Pourcain; Wendy McArdle; John P Kemp; George Davey Smith; Carla Mt Tiesler; Claudia Flexeder; Angela Simpson; Clare S Murray; Oliver Fuchs; Dirkje S Postma; Klaus Bønnelykke; Maties Torrent; Martin Andersson; Patrick Sleiman; Hakon Hakonarson; William O Cookson; Miriam F Moffatt; Lavinia Paternoster; Erik Melén; Jordi Sunyer; Hans Bisgaard; Gerard H Koppelman; Markus Ege; Adnan Custovic; Joachim Heinrich; Frank D Gilliland; Alexander J Henderson; Vincent Wv Jaddoe; Johan C de Jongste
Journal:  J Allergy Clin Immunol       Date:  2013-12-06       Impact factor: 10.793

Review 8.  Mapping asthma-associated variants in admixed populations.

Authors:  Tesfaye B Mersha
Journal:  Front Genet       Date:  2015-09-29       Impact factor: 4.599

Review 9.  Genomic Predictors of Asthma Phenotypes and Treatment Response.

Authors:  Natalia Hernandez-Pacheco; Maria Pino-Yanes; Carlos Flores
Journal:  Front Pediatr       Date:  2019-02-05       Impact factor: 3.418

10.  HaploReg v4: systematic mining of putative causal variants, cell types, regulators and target genes for human complex traits and disease.

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2.  Multiomics analysis identifies BIRC3 as a novel glucocorticoid response-associated gene.

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Review 3.  Genetic Determinants of Poor Response to Treatment in Severe Asthma.

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Review 5.  Gene-environment interactions in childhood asthma revisited; expanding the interaction concept.

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