Literature DB >> 23541188

The neuropsychology of normal aging and preclinical Alzheimer's disease.

Richard J Caselli1, Dona E C Locke2, Amylou C Dueck3, David S Knopman4, Bryan K Woodruff5, Charlene Hoffman-Snyder5, Rosa Rademakers6, Adam S Fleisher7, Eric M Reiman7.   

Abstract

OBJECTIVE: A National Institute on Aging-sponsored work group on preclinical Alzheimer's disease (AD) articulated the need to characterize cognitive differences between normal aging and preclinical AD.
METHODS: Seventy-one apolipoprotein E (APOE) ε4 homozygotes, 194 ε3/ε4 heterozygotes, and 356 ε4 noncarriers age 21 to 87 years who were cognitively healthy underwent neuropsychological testing every 2 years. Longitudinal trajectories of test scores were compared between APOE subgroups.
RESULTS: There was a significant effect of age on all cognitive domains in both APOE ε4 carriers and noncarriers. A significant effect of APOE ε4 gene dose was confined to the memory domain and the Dementia Rating Scale. Cross-sectional comparisons did not discriminate the groups.
CONCLUSIONS: Although cognitive aging patterns are similar in APOE ε4 carriers and noncarriers, preclinical AD is characterized by a significant ε4 gene dose effect that impacts memory and is detectable longitudinally. Preclinical neuropsychological testing strategies should emphasize memory-sensitive measures and longitudinal design.
Copyright © 2014 The Alzheimer's Association. Published by Elsevier Inc. All rights reserved.

Entities:  

Keywords:  Age-related memory loss; Apolipoprotein E; Cognitive aging; Longitudinal testing; Mild cognitive impairment; Preclinical Alzheimer's disease

Mesh:

Substances:

Year:  2013        PMID: 23541188      PMCID: PMC3700591          DOI: 10.1016/j.jalz.2013.01.004

Source DB:  PubMed          Journal:  Alzheimers Dement        ISSN: 1552-5260            Impact factor:   21.566


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