Literature DB >> 22678500

Genetic factors for choroidal neovascularization associated with high myopia.

Nicolas Leveziel1, Yi Yu, Robyn Reynolds, Albert Tai, Weihua Meng, Violaine Caillaux, Patrick Calvas, Bernard Rosner, François Malecaze, Eric H Souied, Johanna M Seddon.   

Abstract

PURPOSE: Nonsyndromic high myopia, defined by a refractive error greater than -6 diopters (D), is associated with an increased risk of macular choroidal neovascularization (CNV), a vision-threatening complication. The aim of this study was to investigate whether genetic factors associated with age-related macular degeneration (AMD) are related to myopic CNV.
METHODS: We conducted a case-control study, including 71 cases with myopic CNV and 196 myopic controls without CNV, from Creteil and Toulouse, France, and Boston, MA. Single nucleotide polymorphisms (SNPs) from 15 genes reported to be related to AMD were selected for association testing in this study.
RESULTS: In univariate analysis, the rs10033900 SNP located in CFI was associated with myopic CNV (P = 0.0011), and a SNP in APOE was also related (P = 0.041). After adjustment for age, sex, and degree of myopia, SNPs in three genes were significantly associated, including CFI (odds ratio [OR] 2.1, 95% confidence interval [CI] 1.3-3.37, P = 0.0023), COL8A1 (OR 1.88, 95% CI 1.18-2.98, P = 0.0076), and CFH (OR 1.65, 95% CI 1.02-2.66, P = 0.04). After correction for multiple testing, only CFI remained significantly related to high myopic CNV (P = 0.045).
CONCLUSIONS: We report the first genetic associations with choroidal neovascularization (CNV) in a high myopic Caucasian population. One SNP (rs10033900) in the CFI gene, which encodes a protein involved in the inflammatory pathway, was significantly associated with myopic CNV in multivariate analysis after correction for multiple testing. This SNP is a plausible biological marker associated with CNV outgrowth among high myopic patients. Results generate hypotheses about potential loci related to CNV in high myopia, and larger studies are needed to expand on these findings.

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Year:  2012        PMID: 22678500      PMCID: PMC3410690          DOI: 10.1167/iovs.12-9538

Source DB:  PubMed          Journal:  Invest Ophthalmol Vis Sci        ISSN: 0146-0404            Impact factor:   4.799


  63 in total

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5.  Linkage replication of the MYP12 locus in common myopia.

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6.  A genome-wide scan maps a novel high myopia locus to 5p15.

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7.  A PAX6 gene polymorphism is associated with genetic predisposition to extreme myopia.

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9.  Genetic association of apolipoprotein E with age-related macular degeneration.

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10.  Complement C3 variant and the risk of age-related macular degeneration.

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  12 in total

Review 1.  Current and emerging treatment options for myopic choroidal neovascularization.

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Review 2.  Anti-VEGF treatment for myopic choroid neovascularization: from molecular characterization to update on clinical application.

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3.  Oxidative Stress Levels in Aqueous Humor from High Myopic Patients.

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4.  Evaluation of 10 AMD Associated Polymorphisms as a Cause of Choroidal Neovascularization in Highly Myopic Eyes.

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6.  Computational Approach to Investigating Key GO Terms and KEGG Pathways Associated with CNV.

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Review 7.  Myopic choroidal neovascularisation: current concepts and update on clinical management.

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Review 8.  Anti-VEGF Therapy and the Retina: An Update.

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Review 9.  Anti-vascular endothelial growth factor therapy for the treatment of myopic choroidal neovascularization.

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10.  Long-Term Natural Outcomes of Simple Hemorrhage Associated with Lacquer Crack in High Myopia: A Risk Factor for Myopic CNV?

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Journal:  J Ophthalmol       Date:  2018-01-24       Impact factor: 1.909

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