Literature DB >> 22441582

Longitudinal imaging of Alzheimer pathology using [11C]PIB, [18F]FDDNP and [18F]FDG PET.

Rik Ossenkoppele1, Nelleke Tolboom, Jessica C Foster-Dingley, Sofie F Adriaanse, Ronald Boellaard, Maqsood Yaqub, Albert D Windhorst, Frederik Barkhof, Adriaan A Lammertsma, Philip Scheltens, Wiesje M van der Flier, Bart N M van Berckel.   

Abstract

PURPOSE: [(11)C]PIB and [(18)F]FDDNP are PET tracers for in vivo detection of the neuropathology underlying Alzheimer's disease (AD). [(18)F]FDG is a glucose analogue and its uptake reflects metabolic activity. The purpose of this study was to examine longitudinal changes in these tracers in patients with AD or mild cognitive impairment (MCI) and in healthy controls.
METHODS: Longitudinal, paired, dynamic [(11)C]PIB and [(18)F]FDDNP (90 min each) and static [(18)F]FDG (15 min) PET scans were obtained in 11 controls, 12 MCI patients and 8 AD patients. The mean interval between baseline and follow-up was 2.5 years (range 2.0-4.0 years). Parametric [(11)C]PIB and [(18)F]FDDNP images of binding potential (BP(ND)) and [(18)F]FDG standardized uptake value ratio (SUVr) images were generated.
RESULTS: A significant increase in global cortical [(11)C]PIB BP(ND) was found in MCI patients, but no changes were observed in AD patients or controls. Subsequent regional analysis revealed that this increase in [(11)C]PIB BP(ND) in MCI patients was most prominent in the lateral temporal lobe (p < 0.05). For [(18)F]FDDNP, no changes in global BP(ND) were found. [(18)F]FDG uptake was reduced at follow-up in the AD group only, especially in frontal, parietal and lateral temporal lobes (all p < 0.01). Changes in global [(11)C]PIB binding (ρ = -0.42, p < 0.05) and posterior cingulate [(18)F]FDG uptake (ρ = 0.54, p < 0.01) were correlated with changes in Mini-Mental-State Examination score over time across groups, whilst changes in [(18)F]FDDNP binding (ρ = -0.18, p = 0.35) were not.
CONCLUSION: [(11)C]PIB and [(18)F]FDG track molecular changes in different stages of AD. We found increased amyloid load in MCI patients and progressive metabolic impairment in AD patients. [(18)F]FDDNP seems to be less useful for examining disease progression.

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Year:  2012        PMID: 22441582     DOI: 10.1007/s00259-012-2102-3

Source DB:  PubMed          Journal:  Eur J Nucl Med Mol Imaging        ISSN: 1619-7070            Impact factor:   9.236


  39 in total

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6.  Differential association of [11C]PIB and [18F]FDDNP binding with cognitive impairment.

Authors:  N Tolboom; W M van der Flier; M Yaqub; T Koene; R Boellaard; A D Windhorst; P Scheltens; A A Lammertsma; B N M van Berckel
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7.  Relationship of cerebrospinal fluid markers to 11C-PiB and 18F-FDDNP binding.

Authors:  Nelleke Tolboom; Wiesje M van der Flier; Maqsood Yaqub; Ronald Boellaard; Nicolaas A Verwey; Marinus A Blankenstein; Albert D Windhorst; Philip Scheltens; Adriaan A Lammertsma; Bart N M van Berckel
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10.  Follow-up of [11C]PIB uptake and brain volume in patients with Alzheimer disease and controls.

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Journal:  Neurology       Date:  2009-09-02       Impact factor: 9.910

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6.  The relationship between CSF biomarkers and cerebral metabolism in early-onset Alzheimer's disease.

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7.  In vivo characterization of a bigenic fluorescent mouse model of Alzheimer's disease with neurodegeneration.

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Review 8.  Molecular imaging of Alzheimer disease pathology.

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Review 9.  ¹⁸F-FDG PET for the early diagnosis of Alzheimer's disease dementia and other dementias in people with mild cognitive impairment (MCI).

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10.  Is verbal episodic memory in elderly with amyloid deposits preserved through altered neuronal function?

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