Literature DB >> 19383812

Utility of p16 immunohistochemistry for the identification of Lynch syndrome.

Artemio Payá1, Cristina Alenda, Lucía Pérez-Carbonell, Estefanía Rojas, José-Luis Soto, Carmen Guillén, Adela Castillejo, Victor M Barberá, Alfredo Carrato, Antoni Castells, Xavier Llor, Montserrat Andreu, Jim Koh, Greg H Enders, Susana Benlloch, Rodrigo Jover.   

Abstract

PURPOSE: Immunohistochemistry for mismatch repair proteins has shown utility in the identification of Lynch syndrome, but majority of tumors with loss of MLH1 expression are due to sporadic hypermethylation of the MLH1 promoter. These tumors can also show epigenetic silencing of other genes, such as p16. The aim of our study is to evaluate the utility of p16 immunohistochemistry in the prediction of MLH1 germline mutations. EXPERIMENTAL
DESIGN: p16 immunohistochemistry was appropriately evaluated in 79 colorectal cancers with loss of MLH1 expression. Methylation of MLH1 and p16 were quantitatively studied using real-time PCR assay Methylight. BRAF V600E mutation in tumor tissue was also investigated. Genetic testing for germline mutation of MLH1 was made on 52 patients.
RESULTS: Loss of p16 expression was seen in 21 of 79 samples (26.6%). There was found statistically significant association between p16 expression and p16 methylation (P < 0.001), MLH1 methylation (P < 0.001), and BRAF mutation (P < 0.005). All tumors with loss of p16 expression showed hypermethylation of p16 (21 of 21), 95.2% (20 of 21) showed MLH1 methylation, and 71.4% (15 of 21) were mutated for BRAF V600E. Mutational analysis showed pathogenic germline mutations in 8 of the patients, harboring 10 tumors. All 10 of these tumors showed normal staining of p16 in the immunochemical analysis.
CONCLUSIONS: p16 immunohistochemistry is a good surrogate marker for p16 and MLH1 epigenetic silencing due to hypermethylation, and is useful as screening tool in the selection of patients for genetic testing in Lynch syndrome.

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Year:  2009        PMID: 19383812      PMCID: PMC2825754          DOI: 10.1158/1078-0432.CCR-08-3116

Source DB:  PubMed          Journal:  Clin Cancer Res        ISSN: 1078-0432            Impact factor:   12.531


  40 in total

1.  p16(Ink4a) inhibits histologic progression and angiogenic signaling in min colon tumors.

Authors:  Steven L Gibson; Amelie Boquoi; Tina Chen; Norman E Sharpless; Colleen Brensinger; Greg H Enders
Journal:  Cancer Biol Ther       Date:  2005-12-09       Impact factor: 4.742

2.  Mismatch repair status in the prediction of benefit from adjuvant fluorouracil chemotherapy in colorectal cancer.

Authors:  R Jover; P Zapater; A Castells; X Llor; M Andreu; J Cubiella; V Piñol; R M Xicola; L Bujanda; J M Reñé; J Clofent; X Bessa; J D Morillas; D Nicolás-Pérez; A Payá; C Alenda
Journal:  Gut       Date:  2005-11-18       Impact factor: 23.059

3.  CpG island methylator phenotype underlies sporadic microsatellite instability and is tightly associated with BRAF mutation in colorectal cancer.

Authors:  Daniel J Weisenberger; Kimberly D Siegmund; Mihaela Campan; Joanne Young; Tiffany I Long; Mark A Faasse; Gyeong Hoon Kang; Martin Widschwendter; Deborah Weener; Daniel Buchanan; Hoey Koh; Lisa Simms; Melissa Barker; Barbara Leggett; Joan Levine; Myungjin Kim; Amy J French; Stephen N Thibodeau; Jeremy Jass; Robert Haile; Peter W Laird
Journal:  Nat Genet       Date:  2006-06-25       Impact factor: 38.330

4.  Precision and performance characteristics of bisulfite conversion and real-time PCR (MethyLight) for quantitative DNA methylation analysis.

Authors:  Shuji Ogino; Takako Kawasaki; Mohan Brahmandam; Mami Cantor; Gregory J Kirkner; Donna Spiegelman; G Mike Makrigiorgos; Daniel J Weisenberger; Peter W Laird; Massimo Loda; Charles S Fuchs
Journal:  J Mol Diagn       Date:  2006-05       Impact factor: 5.568

5.  Evaluation of a large, population-based sample supports a CpG island methylator phenotype in colon cancer.

Authors:  Wade S Samowitz; Hans Albertsen; Jennifer Herrick; Theodore R Levin; Carol Sweeney; Maureen A Murtaugh; Roger K Wolff; Martha L Slattery
Journal:  Gastroenterology       Date:  2005-09       Impact factor: 22.682

6.  Screening for Lynch syndrome (hereditary nonpolyposis colorectal cancer) among endometrial cancer patients.

Authors:  Heather Hampel; Wendy Frankel; Jenny Panescu; Janet Lockman; Kaisa Sotamaa; Daniel Fix; Ilene Comeras; Jennifer La Jeunesse; Hidewaki Nakagawa; Judith A Westman; Thomas W Prior; Mark Clendenning; Pamela Penzone; Janet Lombardi; Patti Dunn; David E Cohn; Larry Copeland; Lynne Eaton; Jeffrey Fowler; George Lewandowski; Luis Vaccarello; Jeffrey Bell; Gary Reid; Albert de la Chapelle
Journal:  Cancer Res       Date:  2006-08-01       Impact factor: 12.701

7.  CpG island methylator phenotype (CIMP) of colorectal cancer is best characterised by quantitative DNA methylation analysis and prospective cohort studies.

Authors:  S Ogino; M Cantor; T Kawasaki; M Brahmandam; G J Kirkner; D J Weisenberger; M Campan; P W Laird; M Loda; C S Fuchs
Journal:  Gut       Date:  2006-01-11       Impact factor: 23.059

8.  Accuracy of revised Bethesda guidelines, microsatellite instability, and immunohistochemistry for the identification of patients with hereditary nonpolyposis colorectal cancer.

Authors:  Virgínia Piñol; Antoni Castells; Montserrat Andreu; Sergi Castellví-Bel; Cristina Alenda; Xavier Llor; Rosa M Xicola; Francisco Rodríguez-Moranta; Artemio Payá; Rodrigo Jover; Xavier Bessa
Journal:  JAMA       Date:  2005-04-27       Impact factor: 56.272

9.  Screening for the Lynch syndrome (hereditary nonpolyposis colorectal cancer).

Authors:  Heather Hampel; Wendy L Frankel; Edward Martin; Mark Arnold; Karamjit Khanduja; Philip Kuebler; Hidewaki Nakagawa; Kaisa Sotamaa; Thomas W Prior; Judith Westman; Jenny Panescu; Dan Fix; Janet Lockman; Ilene Comeras; Albert de la Chapelle
Journal:  N Engl J Med       Date:  2005-05-05       Impact factor: 91.245

10.  COSMIC 2005.

Authors:  S Forbes; J Clements; E Dawson; S Bamford; T Webb; A Dogan; A Flanagan; J Teague; R Wooster; P A Futreal; M R Stratton
Journal:  Br J Cancer       Date:  2006-01-30       Impact factor: 7.640

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  8 in total

Review 1.  Prognostic significance of CDKN2A (p16) promoter methylation and loss of expression in 902 colorectal cancers: Cohort study and literature review.

Authors:  Kaori Shima; Katsuhiko Nosho; Yoshifumi Baba; Mami Cantor; Jeffrey A Meyerhardt; Edward L Giovannucci; Charles S Fuchs; Shuji Ogino
Journal:  Int J Cancer       Date:  2011-03-01       Impact factor: 7.396

2.  Methylation analysis of MLH1 improves the selection of patients for genetic testing in Lynch syndrome.

Authors:  Lucía Pérez-Carbonell; Cristina Alenda; Artemio Payá; Adela Castillejo; Víctor M Barberá; Carmen Guillén; Estefanía Rojas; Nuria Acame; Francisco J Gutiérrez-Aviñó; Antoni Castells; Xavier Llor; Montserrat Andreu; José-Luis Soto; Rodrigo Jover
Journal:  J Mol Diagn       Date:  2010-05-20       Impact factor: 5.568

3.  Identification of Lynch syndrome among patients with colorectal cancer.

Authors:  Leticia Moreira; Francesc Balaguer; Noralane Lindor; Albert de la Chapelle; Heather Hampel; Lauri A Aaltonen; John L Hopper; Loic Le Marchand; Steven Gallinger; Polly A Newcomb; Robert Haile; Stephen N Thibodeau; Shanaka Gunawardena; Mark A Jenkins; Daniel D Buchanan; John D Potter; John A Baron; Dennis J Ahnen; Victor Moreno; Montserrat Andreu; Maurizio Ponz de Leon; Anil K Rustgi; Antoni Castells
Journal:  JAMA       Date:  2012-10-17       Impact factor: 56.272

4.  Immunohistochemical staining for p16 and BRAFV600E is useful to distinguish between sporadic and hereditary (Lynch syndrome-related) microsatellite instable colorectal carcinomas.

Authors:  Florence Boissière-Michot; Hélène Frugier; Alexandre Ho-Pun-Cheung; Evelyne Lopez-Crapez; Jacqueline Duffour; Frédéric Bibeau
Journal:  Virchows Arch       Date:  2016-05-25       Impact factor: 4.064

5.  Co-occurrence of nonsense mutations in MSH6 and MSH2 in Lynch syndrome families evidencing that not all truncating mutations are equal.

Authors:  Carla Pinto; Manuela Pinheiro; Ana Peixoto; Catarina Santos; Isabel Veiga; Patrícia Rocha; Pedro Pinto; Paula Lopes; Manuela Baptista; Rui Henrique; Manuel R Teixeira
Journal:  J Hum Genet       Date:  2015-10-08       Impact factor: 3.172

6.  Prognostic and clinicopathological value of p16 protein aberrant expression in colorectal cancer: A PRISMA-compliant Meta-analysis.

Authors:  Ning Zhou; Quankai Gu
Journal:  Medicine (Baltimore)       Date:  2018-03       Impact factor: 1.889

7.  Multi-Gene Expression in Anthracosis of the Lungs as One of the Risk Factors for Non-Small Cell Lung Cancer

Authors:  Hamidreza Jamaati; Naghmeh Bahrami; Payam Tabarsi; Adnan Khosravi; Arda Kiani; Atefeh Abedini; Rahim Ahmadi; Somayeh Sharifynia; Abdolreza Mohamadnia
Journal:  Asian Pac J Cancer Prev       Date:  2017-11-26

Review 8.  Diagnosis of Lynch Syndrome and Strategies to Distinguish Lynch-Related Tumors from Sporadic MSI/dMMR Tumors.

Authors:  Julie Leclerc; Catherine Vermaut; Marie-Pierre Buisine
Journal:  Cancers (Basel)       Date:  2021-01-26       Impact factor: 6.639

  8 in total

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