Literature DB >> 18650181

Promoter hypermethylation of the MEG3 (DLK1/MEG3) imprinted gene in multiple myeloma.

Leonidas Benetatos1, Aggeliki Dasoula, Eleftheria Hatzimichael, Ioannis Georgiou, Maria Syrrou, Konstantinos L Bourantas.   

Abstract

BACKGROUND: Methylation represents the most studied epigenetic modification and results in the silencing of genes involved in various processes such as differentiation and cell-cycle regulation. MEG3 represents an imprinted gene maternally expressed in humans that encodes a nontranslated product. In this survey, we studied the methylation status of the specific gene in multiple myeloma (MM). PATIENTS AND METHODS: Twenty-one patients with MM (17 with immunoglobulin [Ig] G, 3 with IgA, and 1 with IgM) were evaluated using methylation-specific polymerase chain reaction (after DNA bisulphite modification).
RESULTS: Promoter hypermethylation was observed in 12 (57.14%) bone marrow samples and in 9 of 14 (64.28%) available peripheral blood samples. A correlation with disease stage was also observed and also with the disease subtype (IgG, 64.7%; IgA, 0; IgM, 100%).
CONCLUSION: We conclude that promoter hypermethylation of the differentially methylated region of the MEG3 imprinted gene is observed in patients with MM.

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Year:  2008        PMID: 18650181     DOI: 10.3816/CLM.2008.n.021

Source DB:  PubMed          Journal:  Clin Lymphoma Myeloma        ISSN: 1557-9190


  39 in total

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7.  Study of DNA methylation patterns of imprinted genes in children born after assisted reproductive technologies reveals no imprinting errors: A pilot study.

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10.  The Dysregulation of the DLK1-MEG3 Locus in Islets From Patients With Type 2 Diabetes Is Mimicked by Targeted Epimutation of Its Promoter With TALE-DNMT Constructs.

Authors:  Vasumathi Kameswaran; Maria L Golson; Mireia Ramos-Rodríguez; Kristy Ou; Yue J Wang; Jia Zhang; Lorenzo Pasquali; Klaus H Kaestner
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