Literature DB >> 8445732

Cleavage at a novel site in the NS4A region by the yellow fever virus NS2B-3 proteinase is a prerequisite for processing at the downstream 4A/4B signalase site.

C Lin1, S M Amberg, T J Chambers, C M Rice.   

Abstract

Flavivirus proteins are produced by co- and posttranslational proteolytic processing of a large polyprotein by both host- and virus-encoded proteinases. The viral serine proteinase, which consists of NS2B and NS3, is responsible for cleavage of at least four dibasic sites (2A/2B, 2B/3, 3/4A, and 4B/5) in the nonstructural region. Since the amino acid sequence preceding NS4B shares characteristics with signal peptides used for translocation of nascent polypeptides into the lumen of the endoplasmic reticulum, it has been proposed that cleavage at the 4A/4B site is mediated by a cellular signal peptidase. In this report, cell-free translation and in vivo transient expression assays were used to study processing in the NS4 region of the yellow fever virus polyprotein. With a construct which contained NS4B preceded by 17 residues constituting the putative signal peptide (sig4B), membrane-dependent cleavage at the 4A/4B site was demonstrated in vitro. Surprisingly, processing of NS4A-4B was not observed in cell-free translation studies, and in vivo expression of several yellow fever virus polyproteins revealed that the 4A/4B cleavage occurred only during coexpression of NS2B and the proteinase domain of NS3. Examination of mutant derivatives of the NS3 proteinase domain demonstrated that cleavage at the 4A/4B site correlated with expression of an active NS2B-3 proteinase. From these results, we propose a model in which the signalase cleavage generating the N terminus of NS4B requires a prior NS2B-3 proteinase-mediated cleavage at a novel site (called the 4A/2K site) which is conserved among flaviviruses and located 23 residues upstream of the signalase site. In support of this model, mutations at the 4A/4B signalase site did not eliminate processing in the NS4 region. In contrast, substitutions at the 4A/2K site, which were engineered to block NS2B-3 proteinase-mediated cleavage, eliminated signalase cleavage at the 4A/4B site. In addition, the size of the 3(502)-4A product generated by trans processing of a truncated polyprotein, 3(502)-5(356), was consistent with cleavage at the 4A/2K site rather than at the downstream 4A/4B signalase site.

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Year:  1993        PMID: 8445732      PMCID: PMC240389     

Source DB:  PubMed          Journal:  J Virol        ISSN: 0022-538X            Impact factor:   5.103


  38 in total

1.  Both nonstructural proteins NS2B and NS3 are required for the proteolytic processing of dengue virus nonstructural proteins.

Authors:  B Falgout; M Pethel; Y M Zhang; C J Lai
Journal:  J Virol       Date:  1991-05       Impact factor: 5.103

2.  Flavivirus enzyme-substrate interactions studied with chimeric proteinases: identification of an intragenic locus important for substrate recognition.

Authors:  F Preugschat; E M Lenches; J H Strauss
Journal:  J Virol       Date:  1991-09       Impact factor: 5.103

3.  Processing of the yellow fever virus nonstructural polyprotein: a catalytically active NS3 proteinase domain and NS2B are required for cleavages at dibasic sites.

Authors:  T J Chambers; A Grakoui; C M Rice
Journal:  J Virol       Date:  1991-11       Impact factor: 5.103

4.  The sensitivity of cell-associated dengue virus proteins to trypsin and the detection of trypsin-resistant fragments of the nonstructural glycoprotein NS1.

Authors:  M R Cauchi; E A Henchal; P J Wright
Journal:  Virology       Date:  1991-02       Impact factor: 3.616

Review 5.  Flavivirus genome organization, expression, and replication.

Authors:  T J Chambers; C S Hahn; R Galler; C M Rice
Journal:  Annu Rev Microbiol       Date:  1990       Impact factor: 15.500

6.  Evidence that the N-terminal domain of nonstructural protein NS3 from yellow fever virus is a serine protease responsible for site-specific cleavages in the viral polyprotein.

Authors:  T J Chambers; R C Weir; A Grakoui; D W McCourt; J F Bazan; R J Fletterick; C M Rice
Journal:  Proc Natl Acad Sci U S A       Date:  1990-11       Impact factor: 11.205

7.  Cleavage of structural proteins during the assembly of the head of bacteriophage T4.

Authors:  U K Laemmli
Journal:  Nature       Date:  1970-08-15       Impact factor: 49.962

8.  Rapid and efficient site-specific mutagenesis without phenotypic selection.

Authors:  T A Kunkel
Journal:  Proc Natl Acad Sci U S A       Date:  1985-01       Impact factor: 11.205

9.  Nucleotide sequence of yellow fever virus: implications for flavivirus gene expression and evolution.

Authors:  C M Rice; E M Lenches; S R Eddy; S J Shin; R L Sheets; J H Strauss
Journal:  Science       Date:  1985-08-23       Impact factor: 47.728

10.  In vitro synthesis of West Nile virus proteins indicates that the amino-terminal segment of the NS3 protein contains the active centre of the protease which cleaves the viral polyprotein after multiple basic amino acids.

Authors:  G Wengler; G Czaya; P M Färber; J H Hegemann
Journal:  J Gen Virol       Date:  1991-04       Impact factor: 3.891

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  82 in total

1.  Mutagenesis of the NS2B-NS3-mediated cleavage site in the flavivirus capsid protein demonstrates a requirement for coordinated processing.

Authors:  S M Amberg; C M Rice
Journal:  J Virol       Date:  1999-10       Impact factor: 5.103

2.  Mutations in the yellow fever virus nonstructural protein NS2A selectively block production of infectious particles.

Authors:  Beate M Kümmerer; Charles M Rice
Journal:  J Virol       Date:  2002-05       Impact factor: 5.103

3.  High fidelity of yellow fever virus RNA polymerase.

Authors:  Konstantin V Pugachev; Farshad Guirakhoo; Simeon W Ocran; Fred Mitchell; Megan Parsons; Caroline Penal; Soheila Girakhoo; Svetlana O Pougatcheva; Juan Arroyo; Dennis W Trent; Thomas P Monath
Journal:  J Virol       Date:  2004-01       Impact factor: 5.103

4.  Structural and functional parameters of the flaviviral protease: a promising antiviral drug target.

Authors:  Sergey A Shiryaev; Alex Y Strongin
Journal:  Future Virol       Date:  2010-09-01       Impact factor: 1.831

5.  Mutagenesis of the signal sequence of yellow fever virus prM protein: enhancement of signalase cleavage In vitro is lethal for virus production.

Authors:  E Lee; C E Stocks; S M Amberg; C M Rice; M Lobigs
Journal:  J Virol       Date:  2000-01       Impact factor: 5.103

Review 6.  Flavivirus RNA synthesis in vitro.

Authors:  Radhakrishnan Padmanabhan; Ratree Takhampunya; Tadahisa Teramoto; Kyung H Choi
Journal:  Methods       Date:  2015-08-10       Impact factor: 3.608

7.  An N-terminal amphipathic helix in dengue virus nonstructural protein 4A mediates oligomerization and is essential for replication.

Authors:  Omer Stern; Yu-Fu Hung; Olga Valdau; Yakey Yaffe; Eva Harris; Silke Hoffmann; Dieter Willbold; Ella H Sklan
Journal:  J Virol       Date:  2013-01-16       Impact factor: 5.103

8.  Isolation and characterization of noncytopathic pestivirus mutants reveals a role for nonstructural protein NS4B in viral cytopathogenicity.

Authors:  L Qu; L K McMullan; C M Rice
Journal:  J Virol       Date:  2001-11       Impact factor: 5.103

9.  NS2B-3 proteinase-mediated processing in the yellow fever virus structural region: in vitro and in vivo studies.

Authors:  S M Amberg; A Nestorowicz; D W McCourt; C M Rice
Journal:  J Virol       Date:  1994-06       Impact factor: 5.103

10.  Both NS3 and NS4A are required for proteolytic processing of hepatitis C virus nonstructural proteins.

Authors:  C Failla; L Tomei; R De Francesco
Journal:  J Virol       Date:  1994-06       Impact factor: 5.103

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