Literature DB >> 35524799

Intrinsic differences in rod and cone membrane composition: implications for cone degeneration.

Daniela M Verra1, Perrine Spinnhirny1, Cristina Sandu1, Stéphane Grégoire2,3,4, Niyazi Acar2,3,4, Olivier Berdeaux2,3,4, Lionel Brétillon2,3,4, Janet R Sparrow5, David Hicks6.   

Abstract

PURPOSE: In many retinal pathological conditions, rod and cone degeneration differs. For example, the early-onset maculopathy Stargardts disease type 1 (STGD1) is typified by loss of cones while rods are often less affected. We wanted to examine whether there exist intrinsic membrane differences between rods and cones that might explain such features.
METHODS: Abca4 mRNA and protein levels were quantified in rod- and cone-enriched samples from wild-type and Nrl-/- mice retinas; rod- and cone-enriched outer segments (ROS and COS respectively) were prepared from pig retinas, and total lipids were analyzed by flame ionization, chromatography, and tandem mass spectrometry. Immunohistochemical staining of cone-rich rodent Arvicanthis ansorgei retinas was conducted, and ultra-high performance liquid chromatography of lipid species in porcine ROS and COS was performed.
RESULTS: Abca4 mRNA and Abca4 protein content was significantly higher (50-300%) in cone compared to rod-enriched samples. ROS and COS displayed dramatic differences in several lipids, including very long chain poly-unsaturated fatty acids (VLC-PUFAs), especially docosahexaenoic acid (DHA, 22:6n-3): ROS 20.6% DHA, COS 3.3% (p < 0.001). VLC-PUFAs (> 50 total carbons) were virtually absent from COS. COS were impoverished (> 6× less) in phosphatidylethanolamine compared to ROS. ELOVL4 ("ELOngation of Very Long chain fatty acids 4") antibody labelled Arvicanthis cones only very weakly compared to rods. Finally, there were large amounts (905 a.u.) of the bisretinoid A2PE in ROS, whereas it was much lower (121 a.u., ~ 7.5-fold less) in COS fractions. In contrast, COS contained fivefold higher amounts of all-trans-retinal dimer (115 a.u. compared to 22 a.u. in rods).
CONCLUSIONS: Compared to rods, cones expressed higher levels of Abca4 mRNA and Abca4 protein, were highly impoverished in PUFA (especially DHA) and phosphatidylethanolamine, and contained significant amounts of all-trans-retinal dimer. Based on these and other data, we propose that in contrast to rods, cones are preferentially vulnerable to stress and may die through direct cellular toxicity in pathologies such as STGD1.
© 2022. The Author(s), under exclusive licence to Springer-Verlag GmbH Germany, part of Springer Nature.

Entities:  

Keywords:  Abca4; Cones; Immunochemistry; Liquid chromatography; Membrane lipids; Rods; Stargardt disease

Mesh:

Substances:

Year:  2022        PMID: 35524799     DOI: 10.1007/s00417-022-05684-9

Source DB:  PubMed          Journal:  Graefes Arch Clin Exp Ophthalmol        ISSN: 0721-832X            Impact factor:   3.535


  74 in total

1.  ABCR expression in foveal cone photoreceptors and its role in Stargardt macular dystrophy.

Authors:  L L Molday; A R Rabin; R S Molday
Journal:  Nat Genet       Date:  2000-07       Impact factor: 38.330

2.  A photoreceptor cell-specific ATP-binding transporter gene (ABCR) is mutated in recessive Stargardt macular dystrophy.

Authors:  R Allikmets
Journal:  Nat Genet       Date:  1997-09       Impact factor: 38.330

3.  Distribution of cones in human and monkey retina: individual variability and radial asymmetry.

Authors:  C A Curcio; K R Sloan; O Packer; A E Hendrickson; R E Kalina
Journal:  Science       Date:  1987-05-01       Impact factor: 47.728

Review 4.  Light and macular degeneration: a biophysical and clinical perspective.

Authors:  M A Mainster
Journal:  Eye (Lond)       Date:  1987       Impact factor: 3.775

5.  Decreased thickness and integrity of the macular elastic layer of Bruch's membrane correspond to the distribution of lesions associated with age-related macular degeneration.

Authors:  N H Victor Chong; Jason Keonin; Phil J Luthert; Christina I Frennesson; David M Weingeist; Rachel L Wolf; Robert F Mullins; Gregory S Hageman
Journal:  Am J Pathol       Date:  2005-01       Impact factor: 4.307

6.  Visual acuity loss and clinical observations in a large series of patients with Stargardt disease.

Authors:  Ygal Rotenstreich; Gerald A Fishman; Robert J Anderson
Journal:  Ophthalmology       Date:  2003-06       Impact factor: 12.079

7.  Relationship between the foveal avascular zone and foveal pit morphology.

Authors:  Adam M Dubis; Benjamin R Hansen; Robert F Cooper; Joseph Beringer; Alfredo Dubra; Joseph Carroll
Journal:  Invest Ophthalmol Vis Sci       Date:  2012-03-21       Impact factor: 4.799

8.  A longitudinal study of stargardt disease: clinical and electrophysiologic assessment, progression, and genotype correlations.

Authors:  Kaoru Fujinami; Noemi Lois; Alice E Davidson; Donna S Mackay; Chris R Hogg; Edwin M Stone; Kazushige Tsunoda; Kazuo Tsubota; Catey Bunce; Anthony G Robson; Anthony T Moore; Andrew R Webster; Graham E Holder; Michel Michaelides
Journal:  Am J Ophthalmol       Date:  2013-03-15       Impact factor: 5.258

9.  Lipofuscin and autofluorescence metrics in progressive STGD.

Authors:  R Theodore Smith; Nuno L Gomes; Gaetano Barile; Mihai Busuioc; Noah Lee; Andrew Laine
Journal:  Invest Ophthalmol Vis Sci       Date:  2009-04-22       Impact factor: 4.799

10.  Disease sequence from mutant rhodopsin allele to rod and cone photoreceptor degeneration in man.

Authors:  A V Cideciyan; D C Hood; Y Huang; E Banin; Z Y Li; E M Stone; A H Milam; S G Jacobson
Journal:  Proc Natl Acad Sci U S A       Date:  1998-06-09       Impact factor: 11.205

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