Literature DB >> 34792616

CHRFAM7A expression in mice increases resiliency after injury.

Todd W Costantini1, Raul Coimbra2, Jessica L Weaver3, Brian P Eliceiri3.   

Abstract

INTRODUCTION: The CHRNA7 gene encodes the α-7 nicotinic acetylcholine receptor (α7nAchR) that regulates anti-inflammatory responses to injury; however, only humans express a variant gene called CHRFAM7A that alters the function of α7nAChR; CHRFAM7A expression predominates in bone marrow and monocytes/macrophages where the CHRFAM7A/CHRNA7 ratio is highly variable between individuals. We have previously shown in transgenic mice that CHRFAM7A increased emergency myelopoiesis from the bone marrow and monocyte/macrophage expression in lungs.
MATERIALS AND METHODS: CHRFAM7A transgenic mice are compared to age- and gender-matched wild-type (WT) siblings. We utilized a model of sepsis using LPS injection to measure survival. Lung vascular permeability was measured after severe burn injury in WT vs. CHRFAM7A transgenic mice. Bone marrow CHRFAM7A expression was evaluated using adoptive transfer of CHRFAM7A transgenic bone marrow into WT mice.
RESULTS: Here, we demonstrate that CHRFAM7A expression results in an anti-inflammatory phenotype with an improved survival to LPS and decreased acute lung injury in a severe cutaneous burn model compared to WT.
CONCLUSIONS: These data suggest that the relative expression of CHRFAM7A may alter resiliency to injury and contribute to individual variability in the human systemic inflammatory response (SIRS) to injury.
© 2021. The Author(s), under exclusive licence to Springer Nature Switzerland AG.

Entities:  

Keywords:  burn; human gene; lung permeability; sepsis; survival

Mesh:

Substances:

Year:  2021        PMID: 34792616      PMCID: PMC8758545          DOI: 10.1007/s00011-021-01519-1

Source DB:  PubMed          Journal:  Inflamm Res        ISSN: 1023-3830            Impact factor:   4.575


  6 in total

1.  Targeting α-7 nicotinic acetylcholine receptor in the enteric nervous system: a cholinergic agonist prevents gut barrier failure after severe burn injury.

Authors:  Todd W Costantini; Michael Krzyzaniak; Gerald A Cheadle; James G Putnam; Ann-Marie Hageny; Nicole Lopez; Brian P Eliceiri; Vishal Bansal; Raul Coimbra
Journal:  Am J Pathol       Date:  2012-06-08       Impact factor: 4.307

2.  A Human-Specific α7-Nicotinic Acetylcholine Receptor Gene in Human Leukocytes: Identification, Regulation and the Consequences of CHRFAM7A Expression.

Authors:  Todd W Costantini; Xitong Dang; Maryana V Yurchyshyna; Raul Coimbra; Brian P Eliceiri; Andrew Baird
Journal:  Mol Med       Date:  2015-04-03       Impact factor: 6.354

Review 3.  Molecular control of activation and priming in macrophages.

Authors:  Christopher K Glass; Gioacchino Natoli
Journal:  Nat Immunol       Date:  2016-01       Impact factor: 25.606

4.  CHRFAM7A reduces monocyte/macrophage migration and colony formation in vitro.

Authors:  Theresa W Chan; Simone Langness; Olga Cohen; Brian P Eliceiri; Andrew Baird; Todd W Costantini
Journal:  Inflamm Res       Date:  2020-04-17       Impact factor: 4.575

5.  Uniquely human CHRFAM7A gene increases the hematopoietic stem cell reservoir in mice and amplifies their inflammatory response.

Authors:  Todd W Costantini; Theresa W Chan; Olga Cohen; Simone Langness; Sabrina Treadwell; Elliot Williams; Brian P Eliceiri; Andrew Baird
Journal:  Proc Natl Acad Sci U S A       Date:  2019-04-03       Impact factor: 11.205

6.  The human-specific duplicated α7 gene inhibits the ancestral α7, negatively regulating nicotinic acetylcholine receptor-mediated transmitter release.

Authors:  Carolina Martín-Sánchez; Eva Alés; Santiago Balseiro-Gómez; Gema Atienza; Francisco Arnalich; Anna Bordas; José L Cedillo; María Extremera; Arturo Chávez-Reyes; Carmen Montiel
Journal:  J Biol Chem       Date:  2021-01-28       Impact factor: 5.157

  6 in total

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