Literature DB >> 30201514

An admixture mapping meta-analysis implicates genetic variation at 18q21 with asthma susceptibility in Latinos.

Christopher R Gignoux1, Dara G Torgerson2, Maria Pino-Yanes3, Lawrence H Uricchio4, Joshua Galanter5, Lindsey A Roth2, Celeste Eng2, Donglei Hu2, Elizabeth A Nguyen2, Scott Huntsman2, Rasika A Mathias6, Rajesh Kumar7, Jose Rodriguez-Santana8, Neeta Thakur2, Sam S Oh2, Meghan McGarry9, Andres Moreno-Estrada10, Karla Sandoval10, Cheryl A Winkler11, Max A Seibold12, Badri Padhukasahasram13, David V Conti14, Harold J Farber15, Pedro Avila16, Emerita Brigino-Buenaventura17, Michael Lenoir18, Kelley Meade19, Denise Serebrisky20, Luisa N Borrell21, William Rodriguez-Cintron22, Shannon Thyne2, Bonnie R Joubert23, Isabelle Romieu24, Albert M Levin13, Juan-Jose Sienra-Monge25, Blanca Estela Del Rio-Navarro25, Weiniu Gan26, Benjamin A Raby27, Scott T Weiss27, Eugene Bleecker28, Deborah A Meyers28, Fernando J Martinez29, W James Gauderman14, Frank Gilliland14, Stephanie J London23, Carlos D Bustamante10, Dan L Nicolae30, Carole Ober31, Saunak Sen32, Kathleen Barnes6, L Keoki Williams33, Ryan D Hernandez34, Esteban G Burchard35.   

Abstract

BACKGROUND: Asthma is a common but complex disease with racial/ethnic differences in prevalence, morbidity, and response to therapies.
OBJECTIVE: We sought to perform an analysis of genetic ancestry to identify new loci that contribute to asthma susceptibility.
METHODS: We leveraged the mixed ancestry of 3902 Latinos and performed an admixture mapping meta-analysis for asthma susceptibility. We replicated associations in an independent study of 3774 Latinos, performed targeted sequencing for fine mapping, and tested for disease correlations with gene expression in the whole blood of more than 500 subjects from 3 racial/ethnic groups.
RESULTS: We identified a genome-wide significant admixture mapping peak at 18q21 in Latinos (P = 6.8 × 10-6), where Native American ancestry was associated with increased risk of asthma (odds ratio [OR], 1.20; 95% CI, 1.07-1.34; P = .002) and European ancestry was associated with protection (OR, 0.86; 95% CI, 0.77-0.96; P = .008). Our findings were replicated in an independent childhood asthma study in Latinos (P = 5.3 × 10-3, combined P = 2.6 × 10-7). Fine mapping of 18q21 in 1978 Latinos identified a significant association with multiple variants 5' of SMAD family member 2 (SMAD2) in Mexicans, whereas a single rare variant in the same window was the top association in Puerto Ricans. Low versus high SMAD2 blood expression was correlated with case status (13.4% lower expression; OR, 3.93; 95% CI, 2.12-7.28; P < .001). In addition, lower expression of SMAD2 was associated with more frequent exacerbations among Puerto Ricans with asthma.
CONCLUSION: Ancestry at 18q21 was significantly associated with asthma in Latinos and implicated multiple ancestry-informative noncoding variants upstream of SMAD2 with asthma susceptibility. Furthermore, decreased SMAD2 expression in blood was strongly associated with increased asthma risk and increased exacerbations.
Copyright © 2018. Published by Elsevier Inc.

Entities:  

Keywords:  Asthma; Latinos; SMAD2; admixture mapping; asthma exacerbations; gene expression; meta-analysis; rare variation; targeted sequencing

Mesh:

Substances:

Year:  2018        PMID: 30201514      PMCID: PMC6927816          DOI: 10.1016/j.jaci.2016.08.057

Source DB:  PubMed          Journal:  J Allergy Clin Immunol        ISSN: 0091-6749            Impact factor:   10.793


  47 in total

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