Literature DB >> 29938395

LCCC 1025: a phase II study of everolimus, trastuzumab, and vinorelbine to treat progressive HER2-positive breast cancer brain metastases.

Amanda E D Van Swearingen1, Marni B Siegel1, Allison M Deal1, Maria J Sambade1, Alan Hoyle1, D Neil Hayes1,2, Heejoon Jo1, Paul Little1, Elizabeth Claire Dees1, Hyman Muss1, Trevor Jolly1, Timothy M Zagar1, Nirali Patel1, C Ryan Miller1, Joel S Parker1, J Keith Smith1, Julie Fisher3, Nikita Shah4, Lisle Nabell5, Rita Nanda6, Patrick Dillon7, Vandana Abramson8, Lisa A Carey1, Carey K Anders9.   

Abstract

PURPOSE: HER2 + breast cancer (BC) is an aggressive subtype with high rates of brain metastases (BCBM). Two-thirds of HER2 + BCBM demonstrate activation of the PI3K/mTOR pathway driving resistance to anti-HER2 therapy. This phase II study evaluated everolimus (E), a brain-permeable mTOR inhibitor, trastuzumab (T), and vinorelbine (V) in patients with HER2 + BCBM. PATIENTS AND METHODS: Eligible patients had progressive HER2 + BCBM. The primary endpoint was intracranial response rate (RR); secondary objectives were CNS clinical benefit rate (CBR), extracranial RR, time to progression (TTP), overall survival (OS), and targeted sequencing of tumors from enrolled patients. A two-stage design distinguished intracranial RR of 5% versus 20%.
RESULTS: 32 patients were evaluable for toxicity, 26 for efficacy. Intracranial RR was 4% (1 PR). CNS CBR at 6 mos was 27%; at 3 mos 65%. Median intracranial TTP was 3.9 mos (95% CI 2.2-5). OS was 12.2 mos (95% CI 0.6-20.2). Grade 3-4 toxicities included neutropenia (41%), anemia (16%), and stomatitis (16%). Mutations in TP53 and PIK3CA were common in BCBM. Mutations in the PI3K/mTOR pathway were not associated with response. ERBB2 amplification was higher in BCBM compared to primary BC; ERBB2 amplification in the primary BC trended toward worse OS.
CONCLUSION: While intracranial RR to ETV was low in HER2 + BCBM patients, one-third achieved CNS CBR; TTP/OS was similar to historical control. No new toxicity signals were observed. Further analysis of the genomic underpinnings of BCBM to identify tractable prognostic and/or predictive biomarkers is warranted. CLINICAL TRIAL: (NCT01305941).

Entities:  

Keywords:  Brain metastases; Breast cancer; Metastases; PI3K, MEK; Targeted therapy

Mesh:

Substances:

Year:  2018        PMID: 29938395      PMCID: PMC6779035          DOI: 10.1007/s10549-018-4852-5

Source DB:  PubMed          Journal:  Breast Cancer Res Treat        ISSN: 0167-6806            Impact factor:   4.872


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