Literature DB >> 29452179

Massively parallel C. elegans tracking provides multi-dimensional fingerprints for phenotypic discovery.

Michele Perni1, Pavan K Challa1, Julius B Kirkegaard2, Ryan Limbocker1, Mandy Koopman3, Maarten C Hardenberg3, Pietro Sormanni1, Thomas Müller1, Kadi L Saar1, Lianne W Y Roode1, Johnny Habchi1, Giulia Vecchi1, Nilumi Fernando1, Samuel Casford1, Ellen A A Nollen3, Michele Vendruscolo4, Christopher M Dobson5, Tuomas P J Knowles6.   

Abstract

BACKGROUND: The nematode worm C. elegans is a model organism widely used for studies of genetics and of human disease. The health and fitness of the worms can be quantified in different ways, such as by measuring their bending frequency, speed or lifespan. Manual assays, however, are time consuming and limited in their scope providing a strong motivation for automation. NEW
METHOD: We describe the development and application of an advanced machine vision system for characterising the behaviour of C. elegans, the Wide Field-of-View Nematode Tracking Platform (WF-NTP), which enables massively parallel data acquisition and automated multi-parameter behavioural profiling of thousands of worms simultaneously.
RESULTS: We screened more than a million worms from several established models of neurodegenerative disorders and characterised the effects of potential therapeutic molecules for Alzheimer's and Parkinson's diseases. By using very large numbers of animals we show that the sensitivity and reproducibility of behavioural assays is very greatly increased. The results reveal the ability of this platform to detect even subtle phenotypes. COMPARISON WITH EXISTING
METHODS: The WF-NTP method has substantially greater capacity compared to current automated platforms that typically either focus on characterising single worms at high resolution or tracking the properties of populations of less than 50 animals.
CONCLUSIONS: The WF-NTP extends significantly the power of existing automated platforms by combining enhanced optical imaging techniques with an advanced software platform. We anticipate that this approach will further extend the scope and utility of C. elegans as a model organism.
Copyright © 2018. Published by Elsevier B.V.

Entities:  

Keywords:  Alzheimer’s Disease; Automation; Drug discovery; Neuroscience; Parkinson’s Disease

Mesh:

Year:  2018        PMID: 29452179     DOI: 10.1016/j.jneumeth.2018.02.005

Source DB:  PubMed          Journal:  J Neurosci Methods        ISSN: 0165-0270            Impact factor:   2.390


  21 in total

1.  Assessing motor-related phenotypes of Caenorhabditis elegans with the wide field-of-view nematode tracking platform.

Authors:  Mandy Koopman; Quentin Peter; Renée I Seinstra; Michele Perni; Michele Vendruscolo; Christopher M Dobson; Tuomas P J Knowles; Ellen A A Nollen
Journal:  Nat Protoc       Date:  2020-05-20       Impact factor: 13.491

2.  Image recognition based on deep learning in Haemonchus contortus motility assays.

Authors:  Martin Žofka; Linh Thuy Nguyen; Eva Mašátová; Petra Matoušková
Journal:  Comput Struct Biotechnol J       Date:  2022-05-13       Impact factor: 6.155

3.  An economical and highly adaptable optogenetics system for individual and population-level manipulation of Caenorhabditis elegans.

Authors:  M Koopman; L Janssen; E A A Nollen
Journal:  BMC Biol       Date:  2021-08-24       Impact factor: 7.431

4.  Fast Fluorescence Lifetime Imaging Reveals the Aggregation Processes of α-Synuclein and Polyglutamine in Aging Caenorhabditis elegans.

Authors:  Romain F Laine; Tessa Sinnige; Kai Yu Ma; Amanda J Haack; Chetan Poudel; Peter Gaida; Nathan Curry; Michele Perni; Ellen A A Nollen; Christopher M Dobson; Michele Vendruscolo; Gabriele S Kaminski Schierle; Clemens F Kaminski
Journal:  ACS Chem Biol       Date:  2019-06-27       Impact factor: 4.634

5.  Expression of the amyloid-β peptide in a single pair of C. elegans sensory neurons modulates the associated behavioural response.

Authors:  Tessa Sinnige; Prashanth Ciryam; Samuel Casford; Christopher M Dobson; Mario de Bono; Michele Vendruscolo
Journal:  PLoS One       Date:  2019-05-31       Impact factor: 3.240

6.  Exogenous misfolded protein oligomers can cross the intestinal barrier and cause a disease phenotype in C. elegans.

Authors:  Michele Perni; Benedetta Mannini; Catherine K Xu; Janet R Kumita; Christopher M Dobson; Fabrizio Chiti; Michele Vendruscolo
Journal:  Sci Rep       Date:  2021-07-13       Impact factor: 4.379

7.  Two human metabolites rescue a C. elegans model of Alzheimer's disease via a cytosolic unfolded protein response.

Authors:  Priyanka Joshi; Michele Perni; Ryan Limbocker; Benedetta Mannini; Sam Casford; Sean Chia; Johnny Habchi; Johnathan Labbadia; Christopher M Dobson; Michele Vendruscolo
Journal:  Commun Biol       Date:  2021-07-07

8.  Infertility induced by auxin in PX627 Caenorhabditis elegans does not affect mitochondrial functions and aging parameters.

Authors:  Benjamin Dilberger; Stefan Baumanns; Salome T Spieth; Uwe Wenzel; Gunter P Eckert
Journal:  Aging (Albany NY)       Date:  2020-06-08       Impact factor: 5.682

9.  Powerful and interpretable behavioural features for quantitative phenotyping of Caenorhabditis elegans.

Authors:  Avelino Javer; Lidia Ripoll-Sánchez; André E X Brown
Journal:  Philos Trans R Soc Lond B Biol Sci       Date:  2018-09-10       Impact factor: 6.237

10.  Trodusquemine enhances Aβ42 aggregation but suppresses its toxicity by displacing oligomers from cell membranes.

Authors:  Ryan Limbocker; Sean Chia; Francesco S Ruggeri; Michele Perni; Roberta Cascella; Gabriella T Heller; Georg Meisl; Benedetta Mannini; Johnny Habchi; Thomas C T Michaels; Pavan K Challa; Minkoo Ahn; Samuel T Casford; Nilumi Fernando; Catherine K Xu; Nina D Kloss; Samuel I A Cohen; Janet R Kumita; Cristina Cecchi; Michael Zasloff; Sara Linse; Tuomas P J Knowles; Fabrizio Chiti; Michele Vendruscolo; Christopher M Dobson
Journal:  Nat Commun       Date:  2019-01-15       Impact factor: 14.919

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