Literature DB >> 29201508

How localized is pathologically localized prostate cancer? The use of secondary circulating prostate cells as a marker of minimal residual disease and their association with patient outcome.

Nigel P Murray1, Socrates Aedo1, Cynthia Fuentealba2, Eduardo Reyes3, Omar Jacob2.   

Abstract

OBJECTIVE: To determine the prognostic value of secondary circulating prostate cells (CPCs) in men with pT2 prostate cancer treated with radical prostatectomy.
MATERIAL AND METHODS: Prospective observational study was performed in men with pathologically confined prostate cancer who had been treated with radical prostatectomy. CPCs were obtained by differential gel centrifugation from 8 mL venous blood and identified by standard immunocytochemistry using anti-Prostate Specific Antigen (PSA) monoclonal antibody. A positive test was defined as ≥1 PSA staining cell/blood sample. Biochemical failure was defined as a serum PSA >0.2 ng/mL. Age, PSA at diagnosis, pT2a versus pT2b/c, Gleason score and the presence/absence of CPCs were compared with patient outcomes using Kaplan-Meier curves and Cox's hazard model.
RESULTS: Hundred and ninety-one men participated in the study, 107 (44.0%) had pT2b/c disease, 25 (13.1%) had a Gleason score ≥7, and 39 (20.4%) were positive for CPCs. Biochemical failure occurred in 39 (20.4%) patients which was associated with a Gleason score ≥ 7 and CPCs (+). Survival rates at 3, 5 and 10 years for men with CPC (-) and CPC (+) were 100%, 100% and 89.6%, and 74.4%, 64.1% and 18.5% respectively (HR: 18.70). The median time to failure was 5.1 years in CPC (+) men versus 8.1 years in CPC (-) patients.
CONCLUSION: Secondary CPC is a marker for minimal residual disease and it is associated with a worse prognosis. The lead time to failure over serum PSA is approximately 5 years. However they do not define whether the failure is local or systemic.

Entities:  

Keywords:  Biochemical failure; circulating prostate cells; minimal residual disease; pathologically organ confined; prostate cancer; radical prostatectomy

Year:  2017        PMID: 29201508      PMCID: PMC5687208          DOI: 10.5152/tud.2017.60251

Source DB:  PubMed          Journal:  Turk J Urol        ISSN: 2149-3235


  18 in total

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2.  Anatomic radical retropubic prostatectomy-long-term recurrence-free survival rates for localized prostate cancer.

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5.  Clinical and pathological characteristics of patients presenting with biochemical progression after radical retropubic prostatectomy for pathologically organ-confined prostate cancer.

Authors:  F Pinto; T Prayer-Galetti; M Gardiman; E Sacco; M Ciaccia; S Fracalanza; G Betto; F Pagano
Journal:  Urol Int       Date:  2006       Impact factor: 2.089

6.  Biochemical failure after radical prostatectomy in men with pathologic organ-confined disease: pT2a versus pT2b.

Authors:  Stephen J Freedland; Alan W Partin; Jonathan I Epstein; Patrick C Walsh
Journal:  Cancer       Date:  2004-04-15       Impact factor: 6.860

7.  Phase III postoperative adjuvant radiotherapy after radical prostatectomy compared with radical prostatectomy alone in pT3 prostate cancer with postoperative undetectable prostate-specific antigen: ARO 96-02/AUO AP 09/95.

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Review 8.  The timing of salvage radiotherapy after radical prostatectomy: a systematic review.

Authors:  Christopher R King
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9.  Ten-year survival after radical prostatectomy: specimen Gleason score is the predictor in organ-confined prostate cancer.

Authors:  Fernando J Bianco; David P Wood; Michael L Cher; Isaac J Powell; Julia W Souza; J Edson Pontes
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10.  Secondary circulating prostate cells predict biochemical failure in prostate cancer patients after radical prostatectomy and without evidence of disease.

Authors:  Nigel P Murray; Eduardo Reyes; Nelson Orellana; Cynthia Fuentealba; Leonardo Bádinez; Ruben Olivares; José Porcell; Ricardo Dueñas
Journal:  ScientificWorldJournal       Date:  2013-03-31
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Review 1.  The presence of secondary circulating prostate tumour cells determines the risk of biochemical relapse for patients with low- and intermediate-risk prostate cancer who are treated only with external radiotherapy.

Authors:  Nigel P Murray; Socrates Aedo; Cynthia Fuentealba; Eduardo Reyes; Simone Minzer; Aníbal Salazar
Journal:  Ecancermedicalscience       Date:  2018-06-20
  1 in total

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