Literature DB >> 29095058

Protective roles of intestinal microbiota derived short chain fatty acids in Alzheimer's disease-type beta-amyloid neuropathological mechanisms.

Lap Ho1, Kenjiro Ono2, Mayumi Tsuji3, Paolo Mazzola4, Risham Singh1, Giulio M Pasinetti1.   

Abstract

BACKGROUND: Dietary fibers are metabolized by gastrointestinal (GI) bacteria into short-chain fatty acids (SCFAs). We investigated the potential role of these SCFAs in β-amyloid (Aβ) mediated pathological processes that play key roles in Alzheimer's disease (AD) pathogenesis. RESEARCH DESIGN AND METHODS: Multiple complementary assays were used to investigate individual SCFAs for their dose-responsive effects in interfering with the assembly of Aβß1-40 and Aβ1-42 peptides into soluble neurotoxic Aβ aggregates.
RESULTS: We found that several select SCFAs are capable of potently inhibiting Aβ aggregations, in vitro.
CONCLUSION: Our studies support the hypothesis that intestinal microbiota may help protect against AD, in part, by supporting the generation of select SCFAs, which interfere with the formation of toxic soluble Aβ aggregates.

Entities:  

Keywords:  Alzheimer’s disease; beta-amyloid (Aβ); fibrils; microbial; microbiome; microbiota; neurodegeneration; protein misfolding

Mesh:

Substances:

Year:  2017        PMID: 29095058      PMCID: PMC5958896          DOI: 10.1080/14737175.2018.1400909

Source DB:  PubMed          Journal:  Expert Rev Neurother        ISSN: 1473-7175            Impact factor:   4.618


  33 in total

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  65 in total

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Review 3.  The dichotomous role of the gut microbiome in exacerbating and ameliorating neurodegenerative disorders.

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Review 6.  The role of short-chain fatty acids in central nervous system diseases.

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Review 7.  Gut Microbiota Composition and Epigenetic Molecular Changes Connected to the Pathogenesis of Alzheimer's Disease.

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9.  Gut microbial involvement in Alzheimer's disease pathogenesis.

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10.  Effects of Probiotic Supplementation on Short Chain Fatty Acids in the AppNL-G-F Mouse Model of Alzheimer's Disease.

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