| Literature DB >> 29022589 |
Meng How Tan1,2,3, Qin Li1, Raghuvaran Shanmugam2,3, Robert Piskol1, Jennefer Kohler1, Amy N Young1, Kaiwen Ivy Liu3, Rui Zhang1, Gokul Ramaswami1, Kentaro Ariyoshi4, Ankita Gupte5, Liam P Keegan6,7, Cyril X George8, Avinash Ramu9,10, Ni Huang9,10, Elizabeth A Pollina1, Dena S Leeman1, Alessandra Rustighi11, Y P Sharon Goh12, Ajay Chawla12, Giannino Del Sal11, Gary Peltz13, Anne Brunet1, Donald F Conrad9,10, Charles E Samuel8, Mary A O'Connell6,7, Carl R Walkley5,14, Kazuko Nishikura4, Jin Billy Li1.
Abstract
Adenosine-to-inosine (A-to-I) RNA editing is a conserved post-transcriptional mechanism mediated by ADAR enzymes that diversifies the transcriptome by altering selected nucleotides in RNA molecules. Although many editing sites have recently been discovered, the extent to which most sites are edited and how the editing is regulated in different biological contexts are not fully understood. Here we report dynamic spatiotemporal patterns and new regulators of RNA editing, discovered through an extensive profiling of A-to-I RNA editing in 8,551 human samples (representing 53 body sites from 552 individuals) from the Genotype-Tissue Expression (GTEx) project and in hundreds of other primate and mouse samples. We show that editing levels in non-repetitive coding regions vary more between tissues than editing levels in repetitive regions. Globally, ADAR1 is the primary editor of repetitive sites and ADAR2 is the primary editor of non-repetitive coding sites, whereas the catalytically inactive ADAR3 predominantly acts as an inhibitor of editing. Cross-species analysis of RNA editing in several tissues revealed that species, rather than tissue type, is the primary determinant of editing levels, suggesting stronger cis-directed regulation of RNA editing for most sites, although the small set of conserved coding sites is under stronger trans-regulation. In addition, we curated an extensive set of ADAR1 and ADAR2 targets and showed that many editing sites display distinct tissue-specific regulation by the ADAR enzymes in vivo. Further analysis of the GTEx data revealed several potential regulators of editing, such as AIMP2, which reduces editing in muscles by enhancing the degradation of the ADAR proteins. Collectively, our work provides insights into the complex cis- and trans-regulation of A-to-I editing.Entities:
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Year: 2017 PMID: 29022589 PMCID: PMC5723435 DOI: 10.1038/nature24041
Source DB: PubMed Journal: Nature ISSN: 0028-0836 Impact factor: 49.962