Literature DB >> 28882951

The Immunoreactivity of PI3K/AKT Pathway After Prenatal Hypoxic Damage.

Huisun Wang1, Yoonyoung Chung2, Sun-Kyoung Yu3, Yonghyun Jun4.   

Abstract

BACKGROUND/AIM: There is no consensus on the effect of hypoxia on neurogenesis. In this study, we investigated the immunoreactivity of BDNF and PI3K/Akt signaling after uterine artery ligation in pregnant rats.
MATERIALS AND METHODS: Unilateral uterine artery ligation was performed at 16 days of gestation (dg). Fetuses from one horn with ligated artery were allocated to the hypoxic group. Immunohistochemistry was performed with primary antibodies; NeuN, BDNF, PI3K, Akt and phospho-Akt (pAkt).
RESULTS: The densities of NeuN- and BDNF-immunoreactive (IR) cells in the cerebral cortex were lower in the hypoxic fetuses than in the controls at 21 dg. The density of PI3K and pAkt-IR cells in the cortex of the hypoxic group significantly decreased. The results in dentate gyrus were similar to the results in the cerebral cortex.
CONCLUSION: Prenatal hypoxia reduced Akt phosphorylation, which affected neuronal survival in the cortex and dentate gyrus. Copyright
© 2017, International Institute of Anticancer Research (Dr. George J. Delinasios), All rights reserved.

Entities:  

Keywords:  Akt; PI3K; Prenatal hypoxia; neurogenesis; pAkt

Mesh:

Substances:

Year:  2017        PMID: 28882951      PMCID: PMC5656858          DOI: 10.21873/invivo.11139

Source DB:  PubMed          Journal:  In Vivo        ISSN: 0258-851X            Impact factor:   2.155


  31 in total

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9.  Apoptosis and neurogenesis after transient hypoxia in the developing rat brain.

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Authors:  J Du; L Feng; F Yang; B Lu
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  3 in total

1.  Hypoxia of Rats Subjected to Carotid Artery Ligation Results in Impaired Neurogenesis and Reduced Number of Cortical Neurons.

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Journal:  In Vivo       Date:  2019 Mar-Apr       Impact factor: 2.155

2.  Differential Expression of Vascular Endothelial Growth Factor in the Cortex and Hippocampus upon Cerebral Hypoperfusion.

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Review 3.  Role of Prenatal Hypoxia in Brain Development, Cognitive Functions, and Neurodegeneration.

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