Literature DB >> 28696585

The presence and severity of nonalcoholic steatohepatitis is associated with specific changes in circulating bile acids.

Puneet Puri1, Kalyani Daita1, Andrew Joyce2, Faridoddin Mirshahi1, Prasanna K Santhekadur1, Sophie Cazanave1, Velimir A Luketic1, Mohammad S Siddiqui1, Sherry Boyett1, Hae-Ki Min1, Divya P Kumar1, Rohit Kohli3, Huiping Zhou1, Phillip B Hylemon1, Melissa J Contos1, Michael Idowu1, Arun J Sanyal1.   

Abstract

The histologic spectrum of nonalcoholic fatty liver disease (NAFLD) includes fatty liver (NAFL) and steatohepatitis (NASH), which can progress to cirrhosis in up to 20% of NASH patients. Bile acids (BA) are linked to the pathogenesis and therapy of NASH. We (1) characterized the plasma BA profile in biopsy-proven NAFL and NASH and compared to controls and (2) related the plasma BA profile to liver histologic features, disease activity, and fibrosis. Liquid chromatography/mass spectrometry quantified BAs. Descriptive statistics, paired and multiple group comparisons, and regression analyses were performed. Of 86 patients (24 controls, 25 NAFL, and 37 NASH; mean age 51.8 years and body mass index 31.9 kg/m2 ), 66% were women. Increased total primary BAs and decreased secondary BAs (both P < 0.05) characterized NASH. Total conjugated primary BAs were significantly higher in NASH versus NAFL (P = 0.047) and versus controls (P < 0.0001). NASH had higher conjugated to unconjugated chenodeoxycholate (P = 0.04), cholate (P = 0.0004), and total primary BAs (P < 0.0001). The total cholate to chenodeoxycholate ratio was significantly higher in NAFLD without (P = 0.005) and with (P = 0.02) diabetes. Increased key BAs were associated with higher grades of steatosis (taurocholate), lobular (glycocholate) and portal inflammation (taurolithocholate), and hepatocyte ballooning (taurocholate). Conjugated cholate and taurocholate directly and secondary to primary BA ratio inversely correlated to NAFLD activity score. A higher ratio of total secondary to primary BA decreased (odds ratio, 0.57; P = 0.004) and higher conjugated cholate increased the likelihood of significant fibrosis (F≥2) (P = 0.007).
Conclusion: NAFLD is associated with significantly altered circulating BA composition, likely unaffected by type 2 diabetes, and correlated with histological features of NASH; these observations provide the foundation for future hypothesis-driven studies of specific effects of BAs on specific aspects of NASH. (Hepatology 2018;67:534-548).
© 2017 by the American Association for the Study of Liver Diseases.

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Year:  2017        PMID: 28696585      PMCID: PMC5764808          DOI: 10.1002/hep.29359

Source DB:  PubMed          Journal:  Hepatology        ISSN: 0270-9139            Impact factor:   17.425


  38 in total

1.  Hepatocellular ballooning in NASH.

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Journal:  J Hepatol       Date:  2010-06-25       Impact factor: 25.083

2.  Sources of fatty acids stored in liver and secreted via lipoproteins in patients with nonalcoholic fatty liver disease.

Authors:  Kerry L Donnelly; Coleman I Smith; Sarah J Schwarzenberg; Jose Jessurun; Mark D Boldt; Elizabeth J Parks
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3.  Identification of a nuclear receptor for bile acids.

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Authors:  Jason M Ridlon; Phillip B Hylemon
Journal:  J Lipid Res       Date:  2011-10-20       Impact factor: 5.922

5.  Nonalcoholic steatohepatitis is the second leading etiology of liver disease among adults awaiting liver transplantation in the United States.

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Journal:  Gastroenterology       Date:  2013-05-30       Impact factor: 22.682

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10.  Potential of fecal microbiota for early-stage detection of colorectal cancer.

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Journal:  Mol Syst Biol       Date:  2014-11-28       Impact factor: 11.429

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  86 in total

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Journal:  J Lipid Res       Date:  2020-07-22       Impact factor: 5.922

2.  Serum bile acid patterns are associated with the presence of NAFLD in twins, and dose-dependent changes with increase in fibrosis stage in patients with biopsy-proven NAFLD.

Authors:  Cyrielle Caussy; Cynthia Hsu; Seema Singh; Shirin Bassirian; James Kolar; Claire Faulkner; Nikhil Sinha; Ricki Bettencourt; Naveen Gara; Mark A Valasek; Bernd Schnabl; Lisa Richards; David A Brenner; Alan F Hofmann; Rohit Loomba
Journal:  Aliment Pharmacol Ther       Date:  2018-12-02       Impact factor: 8.171

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Review 4.  Bile acid receptors FXR and TGR5 signaling in fatty liver diseases and therapy.

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5.  Exploring new treatment paradigms for alcoholic hepatitis by extrapolating from NASH and cholestasis.

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6.  Serum metabolites detect the presence of advanced fibrosis in derivation and validation cohorts of patients with non-alcoholic fatty liver disease.

Authors:  Cyrielle Caussy; Veeral H Ajmera; Puneet Puri; Cynthia Li-Shin Hsu; Shirin Bassirian; Mania Mgdsyan; Seema Singh; Claire Faulkner; Mark A Valasek; Emily Rizo; Lisa Richards; David A Brenner; Claude B Sirlin; Arun J Sanyal; Rohit Loomba
Journal:  Gut       Date:  2018-12-19       Impact factor: 23.059

Review 7.  Role and effective therapeutic target of gut microbiota in NAFLD/NASH.

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Journal:  Exp Ther Med       Date:  2019-07-17       Impact factor: 2.447

Review 8.  Gut Microbiota in Liver Disease: What Do We Know and What Do We Not Know?

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Journal:  Physiology (Bethesda)       Date:  2020-07-01

9.  Sterol 12α-Hydroxylase Aggravates Dyslipidemia by Activating the Ceramide/mTORC1/SREBP-1C Pathway via FGF21 and FGF15.

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Journal:  Gene Expr       Date:  2019-03-19

10.  Dysregulated FXR-FGF19 signaling and choline metabolism are associated with gut dysbiosis and hyperplasia in a novel pig model of pediatric NASH.

Authors:  Gabriella V Hernandez; Victoria A Smith; Megan Melnyk; Matthew A Burd; Kimberly A Sprayberry; Mark S Edwards; Daniel G Peterson; Darin C Bennet; Rob K Fanter; Daniel A Columbus; Juan P Steibel; Hunter Glanz; Chad Immoos; Margaret S Rice; Tasha M Santiago-Rodriguez; Jason Blank; Jennifer J VanderKelen; Christopher L Kitts; Brian D Piccolo; Michael R La Frano; Douglas G Burrin; Magdalena Maj; Rodrigo Manjarin
Journal:  Am J Physiol Gastrointest Liver Physiol       Date:  2020-01-31       Impact factor: 4.052

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