Literature DB >> 26551737

VEGFR-1 activation-induced MMP-9-dependent invasion in hepatocellular carcinoma.

Tao Li1, Yuhua Zhu1, Lihui Han2, Wanhua Ren1, Hui Liu1, Chengyong Qin1.   

Abstract

AIM: VEGFR-1 can promote invasion through epithelial-mesenchymal transition induction in hepatocellular carcinoma (HCC). This study aims to elucidate VEGFR-1 impact on proteolytic enzymes profile involved with invasion. MATERIALS &
METHODS: The effect on cell invasion was evaluated by invasive and migration assays with and without VEGFR-1 activation. The mechanism was investigated by real-time PCR, western blot and gelatin zymography using inhibitors for MMP-9. In total, 95 HCC patients were enrolled for its clinical value evaluation.
RESULTS: VEGFR-1 activation induced invasion in HCC cells with an increase in the expression and activity of MMP-9 and Snail. MMP-9 blockage effectively inhibited VEGFR-1-induced invasion. High coexpression of both in HCC predicted a worse clinical outcome.
CONCLUSION: Data show a novel VEGFR-1 activation-to-MMP-9 mechanism promoting HCC invasion.

Entities:  

Keywords:  MMP-9; Snail; VEGFR-1; hepatocellular carcinoma; invasion

Mesh:

Substances:

Year:  2015        PMID: 26551737      PMCID: PMC4976842          DOI: 10.2217/fon.15.263

Source DB:  PubMed          Journal:  Future Oncol        ISSN: 1479-6694            Impact factor:   3.404


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