Literature DB >> 26245235

Strontium ranelate analgesia in arthritis models is associated to decreased cytokine release and opioid-dependent mechanisms.

Rodolfo de Melo Nunes1, Morgana Ramos Martins1, Francisco Saraiva da Silva Junior1, Ana Caroline Rocha de Melo Leite1, Virgínia Claudia Carneiro Girão2, Fernando de Queiroz Cunha3, Aryana Lushese Lima Feitosa Marinho1, Ana Carolina Matias Dinelly Pinto1, Francisco Airton Castro Rocha4.   

Abstract

OBJECTIVE: We investigated the anti-inflammatory activity of strontium ranelate (SR) in arthritis models.
MATERIALS AND METHODS: Rats received 1 mg zymosan (Zy) or saline intra-articularly. Other groups were subjected to anterior cruciate ligament transection in the right knee, as an osteoarthritis (OA) model, or a sham procedure. Joint pain was assessed using the articular incapacitation and paw-pressure tests. Cell influx and cytokines were measured in joint exudates. TREATMENT: Groups received either SR (30-300 mg/kg per os) or saline.
RESULTS: SR dose-dependently and significantly inhibited joint pain in both Zy and OA models, while not altering cell influx. Naloxone administration significantly reversed SR analgesia. SR significantly reduced levels of Interleukin-1β and tumor necrosis factor-α in Zy arthritis, whereas those of cytokine-induced neutrophil chemoattractant (CINC)-1 were not altered.
CONCLUSIONS: SR provides analgesia in arthritis that is associated to inhibition of the release of inflammatory cytokines into inflamed joints. This effect is abrogated by administration of the opioid antagonist naloxone.

Entities:  

Keywords:  Arthritis; Cytokines; Pain; Strontium ranelate; Zymosan

Mesh:

Substances:

Year:  2015        PMID: 26245235     DOI: 10.1007/s00011-015-0860-7

Source DB:  PubMed          Journal:  Inflamm Res        ISSN: 1023-3830            Impact factor:   4.575


  24 in total

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