Literature DB >> 25086402

Meniscal transection rather than excision increases pain behavior and structural damage in experimental osteoarthritis in mice.

A C R de Melo Leite1, M A A Teotonio2, V C C Girão3, M M de Lima Pompeu4, R de Melo Nunes5, T M Cunha6, A C M D Pinto7, F de Queiroz Cunha8, F A C Rocha9.   

Abstract

OBJECTIVE: To evaluate pain behavior and structural damage in mice subjected to either meniscal transection or removal.
METHODS: Mice (10/group) were subjected to transection of the medial collateral and anterior cruciate ligaments (ACLT/MCLT) followed by either transection (meniscotomy) or removal (meniscectomy) of the medial meniscus. A control group was subjected only to transection of the ligaments. Pain was assessed using the electronic pressure-meter paw test. Cell influx, measured in joint exudates, and joint histopathology were assessed after 49 days. Four other groups subjected to meniscotomy received indomethacin, the inducible nitric oxide synthase (iNOS) inhibitor 1400W, morphine or the vehicles.
RESULTS: Both meniscotomy and meniscectomy groups displayed persistent and significant increase in pain behavior as compared to controls, being significantly more severe in the former. Cell influx was more intense in the meniscotomy as compared to the meniscectomy group. Structural damage at the tibia, but not at the femur, was also more severe in the meniscotomy group. Indomethacin and 1400W partially but significantly reduced pain whereas morphine abrogated pain behavior in meniscotomized mice.
CONCLUSION: Meniscal transection rather than resection promotes more severe pain and structural damage in mice. Administration of opioids, cyclooxygenase and nitric oxide (NO) synthase inhibitors provide analgesia in this model. Careful description of the structures damaged is crucial when reporting experimental osteoarthritis (OA).
Copyright © 2014 Osteoarthritis Research Society International. Published by Elsevier Ltd. All rights reserved.

Entities:  

Keywords:  Knee; Meniscus; Mice; Osteoarthritis; Pain

Mesh:

Year:  2014        PMID: 25086402     DOI: 10.1016/j.joca.2014.07.019

Source DB:  PubMed          Journal:  Osteoarthritis Cartilage        ISSN: 1063-4584            Impact factor:   6.576


  5 in total

1.  Strontium ranelate analgesia in arthritis models is associated to decreased cytokine release and opioid-dependent mechanisms.

Authors:  Rodolfo de Melo Nunes; Morgana Ramos Martins; Francisco Saraiva da Silva Junior; Ana Caroline Rocha de Melo Leite; Virgínia Claudia Carneiro Girão; Fernando de Queiroz Cunha; Aryana Lushese Lima Feitosa Marinho; Ana Carolina Matias Dinelly Pinto; Francisco Airton Castro Rocha
Journal:  Inflamm Res       Date:  2015-08-06       Impact factor: 4.575

Review 2.  Mechanisms of Osteoarthritic Pain. Studies in Humans and Experimental Models.

Authors:  Annett Eitner; Gunther O Hofmann; Hans-Georg Schaible
Journal:  Front Mol Neurosci       Date:  2017-11-03       Impact factor: 5.639

3.  Anti-IL-20 monoclonal antibody inhibited inflammation and protected against cartilage destruction in murine models of osteoarthritis.

Authors:  Yu-Hsiang Hsu; Ya-Yu Yang; Man-Hsiang Huwang; Yun-Han Weng; I-Ming Jou; Po-Tin Wu; Tain-Yu Lin; Li-Wha Wu; Ming-Shi Chang
Journal:  PLoS One       Date:  2017-04-20       Impact factor: 3.240

4.  Cell sources of inflammatory mediators present in bone marrow areas inside the meniscus.

Authors:  Francisco Airton Castro Rocha; Virgínia Claudia Carneiro Girão; Rodolfo de Melo Nunes; Ana Carolina Matias Dinelly Pinto; Bruno Vidal; João Eurico Fonseca
Journal:  PLoS One       Date:  2019-12-20       Impact factor: 3.240

5.  Refining surgical models of osteoarthritis in mice and rats alters pain phenotype but not joint pathology.

Authors:  Peter R W Gowler; Paul I Mapp; James J Burston; Mohsen Shahtaheri; David A Walsh; Victoria Chapman
Journal:  PLoS One       Date:  2020-09-29       Impact factor: 3.240

  5 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.