Literature DB >> 24522924

Limited hepatitis B virus replication space in the chronically hepatitis C virus-infected liver.

S F Wieland1, S Asabe, R E Engle, R H Purcell, F V Chisari.   

Abstract

We compared the kinetics and magnitude of hepatitis B virus (HBV) infection in hepatitis C virus (HCV)-naive and chronically HCV-infected chimpanzees in whose livers type I interferon-stimulated gene (ISG) expression is strongly induced. HBV infection was delayed and attenuated in the HCV-infected animals, and the number of HBV-infected hepatocytes was drastically reduced. These results suggest that establishment of HBV infection and its replication space is limited by the antiviral effects of type I interferon in the chronically HCV-infected liver.

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Year:  2014        PMID： 24522924      PMCID： PMC3993806          DOI： 10.1128/JVI.03553-13

Source DB:  PubMed          Journal:  J Virol        ISSN： 0022-538X            Impact factor:   5.103


  30 in total

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4.  Genomic analysis of the host response to hepatitis B virus infection.

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7.  Viral and immunological determinants of hepatitis C virus clearance, persistence, and disease.

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6.  Early and Late Hepatitis B Reactivation After IFN- or DAA-based Therapy of Recurrent Hepatitis C in Anti-HBc-positive Liver Transplant Recipients.

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9.  Modeling host interactions with hepatitis B virus using primary and induced pluripotent stem cell-derived hepatocellular systems.

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