Literature DB >> 24122181

Quinacrine treatment trial for sporadic Creutzfeldt-Jakob disease.

Michael D Geschwind1, Amy L Kuo, Katherine S Wong, Aissa Haman, Gillian Devereux, Benjamin J Raudabaugh, David Y Johnson, Charles C Torres-Chae, Ron Finley, Paul Garcia, Julie N Thai, Hugo Q Cheng, John M Neuhaus, Sven A Forner, Jacque L Duncan, Katherine L Possin, Stephen J Dearmond, Stanley B Prusiner, Bruce L Miller.   

Abstract

OBJECTIVE: To determine whether oral quinacrine increases survival in sporadic Creutzfeldt-Jakob disease (sCJD).
METHODS: This NIH/National Institute on Aging-funded, double-blinded, placebo-controlled, stratified randomization treatment trial was conducted at the University of California, San Francisco from February 2005 through May 2009 (ClinicalTrials.gov, NCT00183092). Subjects were randomized (50:50) to quinacrine (300 mg daily) or placebo with inpatient evaluations at baseline, and planned for months 2, 6, and 12. Subjects returning for their month-2 visit were offered open-label quinacrine. The primary outcome was survival from randomization to month 2.
RESULTS: Of 425 patients referred, 69 subjects enrolled, 54 subjects were randomized to active drug or placebo, and 51 subjects with sCJD were included in survival analyses. Survival for the randomized portion of the trial (first 2 months) showed no significant difference between the 2 groups (log-rank statistic, p = 0.43; Cox proportional relative hazard = 1.43, quinacrine compared with placebo, 95% confidence interval = 0.58, 3.53). The quinacrine-treated group, however, declined less on 2 of 3 functional scales, the modified Rankin and Clinical Dementia Rating, than the placebo group during the first 2 months.
CONCLUSION: This interventional study provides Class I evidence that oral quinacrine at 300 mg per day does not improve 2-month survival of patients with sCJD, compared with placebo. Importantly, this study shows that double-blinded, placebo-controlled, randomized treatment trials are possible in prion disease. Furthermore, the quantitative data collected on the course of sCJD will be useful for future trials. CLASSIFICATION OF EVIDENCE: This study provides Class I evidence that quinacrine does not improve survival for people with sCJD when given orally at a dose of 300 mg per day for 2 months.

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Year:  2013        PMID: 24122181      PMCID: PMC4211922          DOI: 10.1212/WNL.0b013e3182a9f3b4

Source DB:  PubMed          Journal:  Neurology        ISSN: 0028-3878            Impact factor:   9.910


  32 in total

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Review 4.  Emerging pharmacotherapies for Creutzfeldt-Jakob disease.

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6.  Quinacrine is mainly metabolized to mono-desethyl quinacrine by CYP3A4/5 and its brain accumulation is limited by P-glycoprotein.

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7.  Clinical trials for prion disease: difficult challenges, but hope for the future.

Authors:  Michael D Geschwind
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9.  Safety and efficacy of quinacrine in human prion disease (PRION-1 study): a patient-preference trial.

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10.  Continuous quinacrine treatment results in the formation of drug-resistant prions.

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Journal:  PLoS Pathog       Date:  2009-11-26       Impact factor: 6.823

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Review 2.  Biology and Genetics of PrP Prion Strains.

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4.  Clinical Trial Simulations Based on Genetic Stratification and the Natural History of a Functional Outcome Measure in Creutzfeldt-Jakob Disease.

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Review 5.  Developing Therapeutics for PrP Prion Diseases.

Authors:  Kurt Giles; Steven H Olson; Stanley B Prusiner
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6.  Melanin or a Melanin-Like Substance Interacts with the N-Terminal Portion of Prion Protein and Inhibits Abnormal Prion Protein Formation in Prion-Infected Cells.

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8.  Quinacrine promotes replication and conformational mutation of chronic wasting disease prions.

Authors:  Jifeng Bian; Hae-Eun Kang; Glenn C Telling
Journal:  Proc Natl Acad Sci U S A       Date:  2014-04-07       Impact factor: 11.205

Review 9.  The Autophagy-Lysosomal Pathway in Neurodegeneration: A TFEB Perspective.

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