Literature DB >> 23738717

An analysis of PLAG1 and HMGA2 rearrangements in salivary duct carcinoma and examination of the role of precursor lesions.

Armita Bahrami1, Bayardo Perez-Ordonez, James D Dalton, Ilan Weinreb.   

Abstract

AIMS: Salivary duct carcinoma (SDC) often arises in pleomorphic adenoma (PA). Putative precursors, including low-grade cribriform cystadenocarcinoma (LGCCC) and ductal carcinoma in-situ (DCIS), are more controversial. Rearrangement of PLAG1 or HMGA2 is seen in 50-70% of PAs, but this has not been investigated in SDC. Using a large collection of SDCs from a single institution, we aimed to study these genes by fluorescence in-situ hybridization (FISH), and to correlate the presence of precursor lesions/intraductal proliferations with gene alterations. METHODS AND
RESULTS: Forty-four SDCs were stained for smooth muscle actin, CK14, and p63, and examined with PLAG1 and HMGA2 FISH. Eight cases were SDC ex-PA; ten had a hyalinized nodule (HN), which is suspicious for PA; six arose in association with LGCCC; and twenty were 'de-novo' SDCs. Ten cases had PLAG1 rearrangement/amplification (22.7%) and eight had HMGA2 (18.2%) rearrangement/amplification. The positive cases were four SDC ex-PAs, eight SDCs with an HN, and five 'de-novo' SDCs. Twenty-three SDC ex-PAs were present in total (52.3%). All six SDC ex-LGCCCs were FISH-negative. Myoepithelial staining surrounded all LGCCCs, and demonstrated DCIS in 17 cases. Eleven DCIS lesions were in SDC ex-PAs or FISH-positive 'de-novo' SDCs. These cases represent 'cancerization' of ducts. Only six FISH-negative 'de-novo' SDCs showed DCIS.
CONCLUSIONS: A large proportion of SDCs arise in PAs (with or without residual evidence of a PA). A small proportion of SDCs arise in LGCCCs. Cases showing DCIS often represent cancerization.
© 2013 John Wiley & Sons Ltd.

Entities:  

Keywords:  HMGA2; PLAG1; ductal carcinoma in situ; low-grade cribriform cystadenocarcinoma; pleomorphic adenoma; precursor; salivary duct carcinoma

Mesh:

Substances:

Year:  2013        PMID: 23738717     DOI: 10.1111/his.12152

Source DB:  PubMed          Journal:  Histopathology        ISSN: 0309-0167            Impact factor:   5.087


  16 in total

1.  Consistent PLAG1 and HMGA2 abnormalities distinguish carcinoma ex-pleomorphic adenoma from its de novo counterparts.

Authors:  Nora Katabi; Ronald Ghossein; Alan Ho; Snjezana Dogan; Lei Zhang; Yun-Shao Sung; Cristina R Antonescu
Journal:  Hum Pathol       Date:  2014-09-07       Impact factor: 3.466

2.  Recurrent RET Gene Rearrangements in Intraductal Carcinomas of Salivary Gland.

Authors:  Ilan Weinreb; Justin A Bishop; Simion I Chiosea; Raja R Seethala; Bayardo Perez-Ordonez; Lei Zhang; Yun-Shao Sung; Chun-Liang Chen; Adel Assaad; Bahram R Oliai; Cristina R Antonescu
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3.  Low-grade cribriform cystadenocarcinoma of the palatal gland: A case report.

Authors:  Shoichiro Kokabu; Junya Nojima; Hidekazu Kayano; Testuya Yoda
Journal:  Oncol Lett       Date:  2015-07-23       Impact factor: 2.967

4.  Histological, immunohistological and molecular characteristics of intraductal precursor of carcinoma ex pleomorphic adenoma support a multistep carcinogenic process.

Authors:  Stephan Ihrler; Orlando Guntinas-Lichius; Abbas Agaimy; Agnes Wolf; Martin Mollenhauer
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Review 6.  Recent advances in the diagnostic pathology of salivary carcinomas.

Authors:  Roderick H W Simpson; Alena Skálová; Silvana Di Palma; Ilmo Leivo
Journal:  Virchows Arch       Date:  2014-08-30       Impact factor: 4.064

7.  Subsets of salivary duct carcinoma defined by morphologic evidence of pleomorphic adenoma, PLAG1 or HMGA2 rearrangements, and common genetic alterations.

Authors:  Simion I Chiosea; Lester D R Thompson; Ilan Weinreb; Julie E Bauman; Alyssa M Mahaffey; Caitlyn Miller; Robert L Ferris; William E Gooding
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Review 8.  Salivary duct carcinoma: An aggressive salivary gland malignancy with opportunities for targeted therapy.

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Review 9.  Low-grade intraductal carcinoma of salivary glands: A systematic review of this rare entity.

Authors:  Francesco Giovacchini; Caterina Bensi; Stefano Belli; Maria Elena Laurenti; Martina Mandarano; Daniele Paradiso; Michele Giansanti; Antonio Tullio
Journal:  J Oral Biol Craniofac Res       Date:  2018-11-25

10.  Integrated data analysis reveals uterine leiomyoma subtypes with distinct driver pathways and biomarkers.

Authors:  Miika Mehine; Eevi Kaasinen; Hanna-Riikka Heinonen; Netta Mäkinen; Kati Kämpjärvi; Nanna Sarvilinna; Mervi Aavikko; Anna Vähärautio; Annukka Pasanen; Ralf Bützow; Oskari Heikinheimo; Jari Sjöberg; Esa Pitkänen; Pia Vahteristo; Lauri A Aaltonen
Journal:  Proc Natl Acad Sci U S A       Date:  2016-01-19       Impact factor: 11.205

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