Literature DB >> 23411883

Visual acuity changes in patients with leber congenital amaurosis and mutations in CEP290.

J Jason McAnany1, Mohamed A Genead, Saloni Walia, Arlene V Drack, Edwin M Stone, Robert K Koenekoop, Elias I Traboulsi, Alison Smith, Richard G Weleber, Samuel G Jacobson, Gerald A Fishman.   

Abstract

OBJECTIVE: To evaluate changes in visual acuity (VA) over time in patients with Leber congenital amaurosis (LCA) and mutations in the CEP290 gene.
METHODS: Visual acuity was determined at the initial and most recent visits of 43 patients with LCA and CEP290 mutations. The main outcome measures included the best-corrected VA at the initial and most recent visits, as well as the correlation between age and VA.
RESULTS: At the initial visit, 14 patients had measurable chart VA in the better-seeing eye, 25 patients had nonmeasurable chart VA, and 4 young patients did not have VA assessed. At the most recent visit, 15 patients had measurable chart VA and 28 had nonmeasurable chart VA. The average interval between the 2 visits was 10.4 years (range, 2-47 years). For patients with measurable chart VA, the median logMAR value at the initial visit (0.75; range, 0.10-2.30) and at the most recent visit (0.70; range, 0.10-2.00) did not differ significantly (P> .05). There was no significant relationship between VA and age.
CONCLUSIONS: Patients with LCA and CEP290 mutations had a wide spectrum of VA that was not related to age or length of follow-up. Severe VA loss was observed in most, but not all, patients in the first decade. These data will help clinicians provide counseling on VA changes in patients with CEP290 mutations and could be of value for future treatment trials.

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Year:  2013        PMID: 23411883      PMCID: PMC3688627          DOI: 10.1001/2013.jamaophthalmol.354

Source DB:  PubMed          Journal:  JAMA Ophthalmol        ISSN: 2168-6165            Impact factor:   7.389


  17 in total

1.  The natural history of Leber's congenital amaurosis. Age-related findings in 35 patients.

Authors:  K L Heher; E I Traboulsi; I H Maumenee
Journal:  Ophthalmology       Date:  1992-02       Impact factor: 12.079

2.  Mutations in CEP290, which encodes a centrosomal protein, cause pleiotropic forms of Joubert syndrome.

Authors:  Enza Maria Valente; Jennifer L Silhavy; Francesco Brancati; Giuseppe Barrano; Suguna Rani Krishnaswami; Marco Castori; Madeline A Lancaster; Eugen Boltshauser; Loredana Boccone; Lihadh Al-Gazali; Elisa Fazzi; Sabrina Signorini; Carrie M Louie; Emanuele Bellacchio; Enrico Bertini; Bruno Dallapiccola; Joseph G Gleeson
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6.  Mutations in the CEP290 (NPHP6) gene are a frequent cause of Leber congenital amaurosis.

Authors:  Anneke I den Hollander; Robert K Koenekoop; Suzanne Yzer; Irma Lopez; Maarten L Arends; Krysta E J Voesenek; Marijke N Zonneveld; Tim M Strom; Thomas Meitinger; Han G Brunner; Carel B Hoyng; L Ingeborgh van den Born; Klaus Rohrschneider; Frans P M Cremers
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Review 10.  Follow-up and diagnostic reappraisal of 75 patients with Leber's congenital amaurosis.

Authors:  S R Lambert; A Kriss; D Taylor; R Coffey; M Pembrey
Journal:  Am J Ophthalmol       Date:  1989-06-15       Impact factor: 5.258

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2.  Full-Field Pupillary Light Responses, Luminance Thresholds, and Light Discomfort Thresholds in CEP290 Leber Congenital Amaurosis Patients.

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Review 7.  Leber congenital amaurosis/early-onset severe retinal dystrophy: clinical features, molecular genetics and therapeutic interventions.

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8.  Splice-Modulating Oligonucleotide QR-110 Restores CEP290 mRNA and Function in Human c.2991+1655A>G LCA10 Models.

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