Literature DB >> 23123105

Long-term follow-up of patients with hypereosinophilic syndrome treated with Alemtuzumab, an anti-CD52 antibody.

Paolo Strati1, Jorge Cortes, Stefan Faderl, Hagop Kantarjian, Srdan Verstovsek.   

Abstract

BACKGROUND: Relapsing, refractory patients with idiopathic hypereosinophilic syndrome (I-HES) and chronic eosinophilic leukemia-not otherwise specified (CEL-NOS) do not have many effective, durable therapeutic options. Alemtuzumab, an anti-CD52 antibody, has been reported to be an effective therapy due to inherent expression of CD52 on eosinophils.
METHODS: A retrospective chart review of 12 patients treated with alemtuzumab at our center until 2012.
RESULTS: Ten (83%) of 12 patients achieved complete hematologic response (CHR) after a median of 1 week for a median duration of 66 weeks, with the elimination of disease-related symptoms; 2 patients achieved partial hematologic remission hematologic remission (PHR). Patients with CHR who received alemtuzumab maintenance (n = 5) had a significantly longer time to progression than those patients who were only observed (n = 5) (P = .01). Eleven patients relapsed (only one while on maintenance), and 6 were rechallenged with alemtuzumab. Five (83%) achieved second CHR after a median of 3.5 weeks, for a median duration of 123 weeks. Again, those given maintenance (n = 3) had a longer time to progression than those who were only observed (P = .04). Adverse effects were mostly related to infusion reactions and lymphopenia-related viral infections (despite antibiotic prophylaxis). One patient developed Epstein-Barr virus-related lymphoma.
CONCLUSIONS: Alemtuzumab is an effective treatment for patients with relapsed, refractory idiopathic hypereosinophilic syndrome and chronic eosinophilic leukemia-not otherwise specified, in terms of both CHR achievement (even after repeated rechallenges) and duration (particularly if provided as a maintenance therapy). Common adverse effects are related to infusion reactions and immunosuppression.
Copyright © 2013 Elsevier Inc. All rights reserved.

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Year:  2012        PMID: 23123105      PMCID: PMC4445419          DOI: 10.1016/j.clml.2012.09.018

Source DB:  PubMed          Journal:  Clin Lymphoma Myeloma Leuk        ISSN: 2152-2669


  18 in total

1.  Alemtuzumab therapy for refractory idiopathic hypereosinophilic syndrome.

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3.  Alemtuzumab therapy for refractory idiopathic hypereosinophilic syndrome with abnormal T cells: a case report.

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Review 5.  Eosinophilia: secondary, clonal and idiopathic.

Authors:  Ayalew Tefferi; Mrinal M Patnaik; Animesh Pardanani
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6.  Surface and mRNA expression of the CD52 antigen by human eosinophils but not by neutrophils.

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7.  The efficacy of imatinib mesylate in patients with FIP1L1-PDGFRalpha-positive hypereosinophilic syndrome. Results of a multicenter prospective study.

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8.  Alemtuzumab therapy for hypereosinophilic syndrome and chronic eosinophilic leukemia.

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Authors:  J H Butterfield; C R Weiler
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  13 in total

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Review 2.  Biological Modulators in Eosinophilic Diseases.

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Review 6.  Management of hypereosinophilia in tropical settings.

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7.  An International, Retrospective Study of Off-Label Biologic Use in the Treatment of Hypereosinophilic Syndromes.

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9.  CD52-targeted depletion by Alemtuzumab ameliorates allergic airway hyperreactivity and lung inflammation.

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Review 10.  Approach to Eosinophilia Presenting With Pulmonary Symptoms.

Authors:  Chen E Rosenberg; Paneez Khoury
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