Literature DB >> 22773231

Factors affecting the treatment of multiple colorectal adenomas.

Emanuele D L Urso1, Riccardo Nascimbeni, Salvatore Pucciarelli, Marco Agostini, Claudio Casella, Dario Moneghini, Diego Di Lorenzo, Isacco Maretto, Maribel Sullivan, Isabella Mammi, Alessandra Viel, Donato Nitti.   

Abstract

BACKGROUND: Currently, no guidelines exist for the treatment of patients with multiple colorectal adenomas (MCRAs) (>10 but <100 synchronous nondiminutive polyps of the large bowel). This retrospective study aimed to investigate the clinical and molecular factors related to different treatments for MCRAs.
METHODS: Patients with MCRAs were consecutively enrolled from January 2003 to June 2011. Sequencing of their APC and MutYH genes was performed. The clinical, molecular, and family histories of the patients were collected using the Progeny database. The patient treatments were divided into three groups of increasing clinical weight: endoscopic polypectomy, segmental resection, and total colectomy. A logistic regression analysis of clinicomolecular factors related to different treatment options was performed.
RESULTS: The study comprised 80 patients (32 women, 40%) with a median age of 53 years (range 13-74 years). The median number of polyps was 33 (range 10-90).The cases included 62 diffuse polyposis, 18 segmental polyposis coli and synchronous colorectal carcinomas (CRC; 34 cases, 43%). The pathogenetic mutations were biallelic MutYH (n = 19, 24%) and APC (n = 4, 5%). The mean follow-up period was 74 months (median 43 months, range 1-468 months). Endoscopic polypectomy was performed in 25 cases (31%), segmental resection in 16 cases (20%), and total colectomy in 39 cases (49%). The logistics regression analysis, considering all the patients, showed that the number of polyps, the presence of CRC, and mutation were correlated with more intensive treatment. For the patients without CRC, only the number of polyps was correlated with the severity of the treatment (p > 0.0166). "On the ROC (receiver operating characteristic) curve, 25 was the number of polyps that best discriminated between surgical and endoscopic therapy.
CONCLUSIONS: The majority of patients with MCRAs undergo surgery. For patients without CRC, only the number of polyps, and not the presence of a disease-causing mutation, is correlated with increased heaviness of treatment. Patients with more than 25 polyps are more likely to undergo a surgical resection.

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Year:  2012        PMID: 22773231     DOI: 10.1007/s00464-012-2421-2

Source DB:  PubMed          Journal:  Surg Endosc        ISSN: 0930-2794            Impact factor:   4.584


  30 in total

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3.  Laparoscopic surgery versus open surgery for colon cancer: short-term outcomes of a randomised trial.

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4.  Rectum-sparing surgery may be appropriate for biallelic MutYH-associated polyposis.

Authors:  Riccardo Nascimbeni; Salvatore Pucciarelli; Diego Di Lorenzo; Emanuele Urso; Claudio Casella; Marco Agostini; Donato Nitti; Bruno Salerni
Journal:  Dis Colon Rectum       Date:  2010-12       Impact factor: 4.585

5.  Inherited variants of MYH associated with somatic G:C-->T:A mutations in colorectal tumors.

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6.  Pathological features of colorectal carcinomas in MYH-associated polyposis.

Authors:  A M O'Shea; S P Cleary; M A Croitoru; H Kim; T Berk; N Monga; R H Riddell; A Pollett; S Gallinger
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7.  Adenomas are detected more often in morning than in afternoon colonoscopy.

Authors:  Madhusudhan R Sanaka; Fnu Deepinder; Prashanthi N Thota; Rocio Lopez; Carol A Burke
Journal:  Am J Gastroenterol       Date:  2009-06-02       Impact factor: 10.864

8.  MYH mutations in patients with attenuated and classic polyposis and with young-onset colorectal cancer without polyps.

Authors:  Liang Wang; Linnea M Baudhuin; Lisa A Boardman; Kelle J Steenblock; Gloria M Petersen; Kevin C Halling; Amy J French; Ruth A Johnson; Lawrence J Burgart; Kari Rabe; Noralane M Lindor; Stephen N Thibodeau
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9.  Missed adenomas during colonoscopic surveillance in individuals with Lynch Syndrome (hereditary nonpolyposis colorectal cancer).

Authors:  Elena M Stoffel; D Kim Turgeon; David H Stockwell; Lili Zhao; Daniel P Normolle; Missy K Tuck; Robert S Bresalier; Norman E Marcon; John A Baron; Mack T Ruffin; Dean E Brenner; Sapna Syngal
Journal:  Cancer Prev Res (Phila)       Date:  2008-11

10.  APC or MUTYH mutations account for the majority of clinically well-characterized families with FAP and AFAP phenotype and patients with more than 30 adenomas.

Authors:  B Filipe; C Baltazar; C Albuquerque; S Fragoso; P Lage; I Vitoriano; S Mão de Ferro; I Claro; P Rodrigues; P Fidalgo; P Chaves; M Cravo; C Nobre Leitão
Journal:  Clin Genet       Date:  2009-09       Impact factor: 4.438

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