Literature DB >> 22251309

Capturing the fragile X premutation phenotypes: a collaborative effort across multiple cohorts.

Jessica Ezzell Hunter1, Stephanie Sherman, Jim Grigsby, Cary Kogan, Kim Cornish.   

Abstract

OBJECTIVE: To capture the neuropsychological profile among male carriers of the FMR1 premutation allele (55-200 CGG repeats) who do not meet diagnostic criteria for the late-onset fragile X-associated tremor/ataxia syndrome, FXTAS.
METHOD: We have initiated a multicenter collaboration that includes 3 independent cohorts, totaling 100 carriers of the premutation and 216 noncarriers. The initial focus of this collaboration has been on executive function. Four executive function scores are shared among the 3 cohorts (Controlled Oral Word Association Test, Stroop Color-Word Test, and Wechsler backward digit span and letter-number sequencing) whereas additional executive function scores are available for specific cohorts (Behavior Dyscontrol Scale, Hayling Sentence Completion Test Part B, and Wisconsin Card Sorting Test). Raw scores were analyzed by using statistical models that adjust for cohort-specific effects as well as age and education.
RESULTS: Carriers scored significantly lower compared to noncarriers on the Stroop Color-Word Test (p = .01), Hayling Sentence Completion Test Part B (p < .01), and Behavioral Dyscontrol Scale (p = .03), with the Hayling displaying a significant age-related decline (p = .01), as assessed by an age and repeat length-group interaction. Follow-up analysis of the collective data did not identify any specific age groups or repeat length ranges (i.e., low premutation = 55-70 repeats, midpremutation = 71-100 repeats, high premutation = 101-199 repeats) that were associated with an increased risk of executive function deficits.
CONCLUSIONS: Preliminary analyses do not indicate global executive function impairment among male carriers without FXTAS compared to noncarriers. However, impairment in inhibitory capacity may be present among a subset of carriers, though the risk factors for this group do not appear to be related to age or repeat length.

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Year:  2012        PMID: 22251309      PMCID: PMC3295926          DOI: 10.1037/a0026799

Source DB:  PubMed          Journal:  Neuropsychology        ISSN: 0894-4105            Impact factor:   3.295


  47 in total

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5.  Fragile-X carrier screening and the prevalence of premutation and full-mutation carriers in Israel.

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8.  Absence of expression of the FMR-1 gene in fragile X syndrome.

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Review 10.  Is there evidence for neuropsychological and neurobehavioral phenotypes among adults without FXTAS who carry the FMR1 premutation? A review of current literature.

Authors:  Jessica Ezzell Hunter; Ann Abramowitz; Michele Rusin; Stephanie L Sherman
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7.  COGNITIVE DYSFUNCTION IN FMR1 PREMUTATION CARRIERS.

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8.  White matter disease and cognitive impairment in FMR1 premutation carriers.

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9.  Eye movements reveal impaired inhibitory control in adult male fragile X premutation carriers asymptomatic for FXTAS.

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10.  Phenotypes of hypofrontality in older female fragile X premutation carriers.

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