Literature DB >> 21525160

Increased prevalence of carpal tunnel syndrome in albright hereditary osteodystrophy.

Andrew W Joseph1, Ashley H Shoemaker, Emily L Germain-Lee.   

Abstract

CONTEXT: Albright hereditary osteodystrophy (AHO) is a rare genetic disorder characterized by phenotypic abnormalities including brachydactyly/brachymetacarpia, short stature, and sc ossifications. Carpal tunnel syndrome (CTS) is a chief complaint in many patients with AHO.
OBJECTIVE: The objective of the study was to investigate the prevalence of CTS in patients with AHO.
DESIGN: This was a cross-sectional study.
SETTING: The study was conducted at the Clinical Research Center (Institute of Clinical and Translational Medicine), Johns Hopkins University School of Medicine and Albright Clinic, Kennedy Krieger Institute. PARTICIPANTS: Thirty-three subjects with a diagnosis of AHO participated in the study. MAIN OUTCOME MEASURES: We assessed for the presence and location of hand tingling, numbness, pain, weakness, flick sign, difficulty with fine motor skills, severe hand or nail biting, and nocturnal symptoms in the setting of normocalcemia and a euthyroid state. Patients were considered to have CTS if they were positive for three of these symptoms. All subjects were analyzed for mutations in the GNAS gene.
RESULTS: Twenty-two subjects (67%) had a clinical diagnosis of CTS (95% confidence interval 0.48, 0.82). Twenty-eight of 33 subjects were confirmed to have mutations in GNAS, of whom 68% had CTS (95% confidence interval 0.48, 0.84). There were 14 children in this study; 36% had a clinical diagnosis of CTS. Body mass index, brachydactyly/brachymetacarpia, prior GH treatment, and specific GNAS mutations were not associated with CTS.
CONCLUSIONS: We report a high prevalence of CTS in both adults and children with AHO. The diagnosis of CTS should be considered when evaluating a patient with AHO because the intervention for CTS could improve overall function and quality of life in these patients.

Entities:  

Mesh:

Year:  2011        PMID: 21525160      PMCID: PMC3135204          DOI: 10.1210/jc.2011-0013

Source DB:  PubMed          Journal:  J Clin Endocrinol Metab        ISSN: 0021-972X            Impact factor:   5.958


  50 in total

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Review 2.  Editorial: Pseudohypoparathyroidism and mechanisms of resistance toward multiple hormones: molecular evidence to clinical presentation.

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3.  Median neuropathy (carpal-tunnel syndrome) in acromegaly. A sign of endocrine overactivity.

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5.  Growth hormone deficiency in pseudohypoparathyroidism type 1a: another manifestation of multihormone resistance.

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6.  Stimulatory G protein directly regulates hypertrophic differentiation of growth plate cartilage in vivo.

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8.  Carpal tunnel syndrome and gynaecomastia during growth hormone treatment of elderly men with low circulating IGF-I concentrations.

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9.  The carpal tunnel syndrome: diagnostic utility of the history and physical examination findings.

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Journal:  J Pediatr Orthop       Date:  1989 Nov-Dec       Impact factor: 2.324

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Review 4.  An update on the clinical and molecular characteristics of pseudohypoparathyroidism.

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5.  A positive genotype-phenotype correlation in a large cohort of patients with Pseudohypoparathyroidism Type Ia and Pseudo-pseudohypoparathyroidism and 33 newly identified mutations in the GNAS gene.

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Review 6.  Management of pseudohypoparathyroidism.

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Review 7.  Imprinting disorders in humans: a review.

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Review 8.  Recommendations for Diagnosis and Treatment of Pseudohypoparathyroidism and Related Disorders: An Updated Practical Tool for Physicians and Patients.

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9.  Classic and Non-Classic Features in Pseudohypoparathyroidism: Case Study and Brief Literature Review.

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Review 10.  Diagnosis and management of pseudohypoparathyroidism and related disorders: first international Consensus Statement.

Authors:  Giovanna Mantovani; Murat Bastepe; David Monk; Luisa de Sanctis; Susanne Thiele; Alessia Usardi; S Faisal Ahmed; Roberto Bufo; Timothée Choplin; Gianpaolo De Filippo; Guillemette Devernois; Thomas Eggermann; Francesca M Elli; Kathleen Freson; Aurora García Ramirez; Emily L Germain-Lee; Lionel Groussin; Neveen Hamdy; Patrick Hanna; Olaf Hiort; Harald Jüppner; Peter Kamenický; Nina Knight; Marie-Laure Kottler; Elvire Le Norcy; Beatriz Lecumberri; Michael A Levine; Outi Mäkitie; Regina Martin; Gabriel Ángel Martos-Moreno; Masanori Minagawa; Philip Murray; Arrate Pereda; Robert Pignolo; Lars Rejnmark; Rebecca Rodado; Anya Rothenbuhler; Vrinda Saraff; Ashley H Shoemaker; Eileen M Shore; Caroline Silve; Serap Turan; Philip Woods; M Carola Zillikens; Guiomar Perez de Nanclares; Agnès Linglart
Journal:  Nat Rev Endocrinol       Date:  2018-08       Impact factor: 43.330

  10 in total

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