Literature DB >> 21489305

Discovering functional modules by identifying recurrent and mutually exclusive mutational patterns in tumors.

Christopher A Miller1, Stephen H Settle, Erik P Sulman, Kenneth D Aldape, Aleksandar Milosavljevic.   

Abstract

BACKGROUND: Assays of multiple tumor samples frequently reveal recurrent genomic aberrations, including point mutations and copy-number alterations, that affect individual genes. Analyses that extend beyond single genes are often restricted to examining pathways, interactions and functional modules that are already known.
METHODS: We present a method that identifies functional modules without any information other than patterns of recurrent and mutually exclusive aberrations (RME patterns) that arise due to positive selection for key cancer phenotypes. Our algorithm efficiently constructs and searches networks of potential interactions and identifies significant modules (RME modules) by using the algorithmic significance test.
RESULTS: We apply the method to the TCGA collection of 145 glioblastoma samples, resulting in extension of known pathways and discovery of new functional modules. The method predicts a role for EP300 that was previously unknown in glioblastoma. We demonstrate the clinical relevance of these results by validating that expression of EP300 is prognostic, predicting survival independent of age at diagnosis and tumor grade.
CONCLUSIONS: We have developed a sensitive, simple, and fast method for automatically detecting functional modules in tumors based solely on patterns of recurrent genomic aberration. Due to its ability to analyze very large amounts of diverse data, we expect it to be increasingly useful when applied to the many tumor panels scheduled to be assayed in the near future.

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Year:  2011        PMID: 21489305      PMCID: PMC3102606          DOI: 10.1186/1755-8794-4-34

Source DB:  PubMed          Journal:  BMC Med Genomics        ISSN: 1755-8794            Impact factor:   3.063


  24 in total

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Review 2.  Statistical significance in biological sequence analysis.

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4.  Reverse engineering of regulatory networks in human B cells.

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5.  Intrinsic gene expression profiles of gliomas are a better predictor of survival than histology.

Authors:  Lonneke A M Gravendeel; Mathilde C M Kouwenhoven; Olivier Gevaert; Johan J de Rooi; Andrew P Stubbs; J Elza Duijm; Anneleen Daemen; Fonnet E Bleeker; Linda B C Bralten; Nanne K Kloosterhof; Bart De Moor; Paul H C Eilers; Peter J van der Spek; Johan M Kros; Peter A E Sillevis Smitt; Martin J van den Bent; Pim J French
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8.  Comprehensive genomic characterization defines human glioblastoma genes and core pathways.

Authors: 
Journal:  Nature       Date:  2008-09-04       Impact factor: 49.962

9.  A probabilistic model of local sequence alignment that simplifies statistical significance estimation.

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Journal:  PLoS Comput Biol       Date:  2008-05-30       Impact factor: 4.475

10.  Functional copy-number alterations in cancer.

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  59 in total

1.  Algorithmic methods to infer the evolutionary trajectories in cancer progression.

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Review 2.  Identifying Epistasis in Cancer Genomes: A Delicate Affair.

Authors:  Joris van de Haar; Sander Canisius; Michael K Yu; Emile E Voest; Lodewyk F A Wessels; Trey Ideker
Journal:  Cell       Date:  2019-05-30       Impact factor: 41.582

3.  Combinatorial Detection of Conserved Alteration Patterns for Identifying Cancer Subnetworks.

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4.  Simultaneous inference of cancer pathways and tumor progression from cross-sectional mutation data.

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Journal:  J Comput Biol       Date:  2015-03-18       Impact factor: 1.479

5.  A weighted exact test for mutually exclusive mutations in cancer.

Authors:  Mark D M Leiserson; Matthew A Reyna; Benjamin J Raphael
Journal:  Bioinformatics       Date:  2016-09-01       Impact factor: 6.937

6.  Efficient algorithms to discover alterations with complementary functional association in cancer.

Authors:  Rebecca Sarto Basso; Dorit S Hochbaum; Fabio Vandin
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7.  Identifying overlapping mutated driver pathways by constructing gene networks in cancer.

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Journal:  BMC Bioinformatics       Date:  2015-03-18       Impact factor: 3.169

8.  Identifying collateral and synthetic lethal vulnerabilities within the DNA-damage response.

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Journal:  BMC Bioinformatics       Date:  2021-05-15       Impact factor: 3.169

Review 9.  Integrative approaches for finding modular structure in biological networks.

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Journal:  Nat Rev Genet       Date:  2013-10       Impact factor: 53.242

Review 10.  The Emerging Potential for Network Analysis to Inform Precision Cancer Medicine.

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Journal:  J Mol Biol       Date:  2018-06-15       Impact factor: 5.469

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