Literature DB >> 19373111

A novel locus for arterial hypertension on chromosome 1p36 maps to a metabolic syndrome trait cluster in the Sorbs, a Slavic population isolate in Germany.

Katrin Hoffmann1, Christian Planitz, Franz Rüschendorf, Bertram Müller-Myhsok, Hans H Stassen, Barbara Lucke, Manuel Mattheisen, Michael Stumvoll, Rolf Bochmann, Martin Zschornack, Thomas F Wienker, Peter Nürnberg, André Reis, Friedrich C Luft, Tom H Lindner.   

Abstract

OBJECTIVE: Genome-wide linkage studies and genome-wide association studies have not as yet identified major genes contributing to primary hypertension in the general population. This state-of-affairs suggests considerable heterogeneity with small contributing effects for primary hypertension, or other complex genetic traits, in outbred populations. Isolated populations, as recent data from Iceland and French Canada suggest, could offer a solution to this problem.
METHODS: We studied a Slavic isolate in Germany, the Sorbs, and genotyped 1040 polymorphic microsatellite markers in 87 multigeneration families.
RESULTS: Our genome-wide linkage scan revealed a locus on chromosome 1p36.13 at D1S3669-D1S2826 (40.95 cM Marshfield coordinates; logarithm of the odds = 3.45, nominal P = 0.00003) that reached genome-wide significance (P = 0.004), indicating the increased power in isolated populations. The chromosome 1 locus maps to a region in which traits such as diabetes, hyperlipidemia, obesity and BMI cluster.
CONCLUSION: Our results suggest that this locus contributes to the metabolic syndrome, and that further attention in this and other populations is warranted.

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Year:  2009        PMID: 19373111     DOI: 10.1097/HJH.0b013e328328123d

Source DB:  PubMed          Journal:  J Hypertens        ISSN: 0263-6352            Impact factor:   4.844


  8 in total

1.  Mapping eQTLs in the Norfolk Island genetic isolate identifies candidate genes for CVD risk traits.

Authors:  Miles C Benton; Rod A Lea; Donia Macartney-Coxson; Melanie A Carless; Harald H Göring; Claire Bellis; Michelle Hanna; David Eccles; Geoffrey K Chambers; Joanne E Curran; Jacquie L Harper; John Blangero; Lyn R Griffiths
Journal:  Am J Hum Genet       Date:  2013-12-05       Impact factor: 11.025

2.  Genetic variation in the Sorbs of eastern Germany in the context of broader European genetic diversity.

Authors:  Krishna R Veeramah; Anke Tönjes; Peter Kovacs; Arnd Gross; Daniel Wegmann; Patrick Geary; Daniela Gasperikova; Iwar Klimes; Markus Scholz; John Novembre; Michael Stumvoll
Journal:  Eur J Hum Genet       Date:  2011-05-11       Impact factor: 4.246

3.  EphA2 Expression Regulates Inflammation and Fibroproliferative Remodeling in Atherosclerosis.

Authors:  Alexandra C Finney; Steven D Funk; Jonette M Green; Arif Yurdagul; Mohammad Atif Rana; Rebecca Pistorius; Miriam Henry; Andrew Yurochko; Christopher B Pattillo; James G Traylor; Jin Chen; Matthew D Woolard; Christopher G Kevil; A Wayne Orr
Journal:  Circulation       Date:  2017-05-09       Impact factor: 29.690

Review 4.  Progress and future aspects in genetics of human hypertension.

Authors:  Qi Zhao; Tanika N Kelly; Changwei Li; Jiang He
Journal:  Curr Hypertens Rep       Date:  2013-12       Impact factor: 5.369

5.  Genetic association study of prolylcarboxypeptidase polymorphisms with susceptibility to essential hypertension in the Yi minority of China: A case-control study based on an isolated population.

Authors:  Yanrui Wu; Hongju Yang; Chunjie Xiao
Journal:  J Renin Angiotensin Aldosterone Syst       Date:  2020 Apr-Jun       Impact factor: 1.636

6.  Haplotype-based association study between PRCP gene polymorphisms and essential hypertension in Hani minority group from a remote region of China.

Authors:  Yanrui Wu; Xingming Pan; Xiaoxiao Jin
Journal:  J Renin Angiotensin Aldosterone Syst       Date:  2020 Oct-Dec       Impact factor: 1.636

7.  Are isolated populations better for studying genes that predispose to hypertension?

Authors:  Anna F Dominiczak; Christian Delles; Sandosh Padmanabhan
Journal:  J Hypertens       Date:  2009-05       Impact factor: 4.844

8.  Type II diabetes and impaired glucose tolerance due to severe hyperinsulinism in patients with 1p36 deletion syndrome and a Prader-Willi-like phenotype.

Authors:  Stefano Stagi; Elisabetta Lapi; Marilena Pantaleo; Francesco Chiarelli; Salvatore Seminara; Maurizio de Martino
Journal:  BMC Med Genet       Date:  2014-01-30       Impact factor: 2.103

  8 in total

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