Literature DB >> 19176451

Human papillomavirus type 18 DNA load and 2-year cumulative diagnoses of cervical intraepithelial neoplasia grades 2-3.

Long Fu Xi1, Laura A Koutsky, Philip E Castle, Cosette M Wheeler, Denise A Galloway, Constance Mao, Jesse Ho, Nancy B Kiviat.   

Abstract

BACKGROUND: The clinical relevance of the amount of human papillomavirus type 18 (HPV18) DNA in cervical tissue (ie, HPV18 DNA load) is unknown.
METHODS: Study subjects were 303 women who were HPV18 positive at enrollment into the Atypical Squamous Cells of Undetermined Significance (ASC-US) and Low-Grade Squamous Intraepithelial Lesion (LSIL) Triage Study. HPV18 DNA load, expressed as copies of HPV18 per nanogram of cellular DNA, at enrollment was quantitatively measured. Subjects were followed up semiannually for a period of 2 years for detection of cervical intraepithelial neoplasia 2-3 (CIN2-3). A linear regression model was used to examine associations of CIN2-3 with HPV18 DNA load. All statistical tests were two-sided.
RESULTS: CIN2-3 was confirmed in 92 of 303 (30.4%) HPV18-positive women. Among women without CIN2-3, HPV18 DNA load was positively associated with increasing severity of cervical cytology at enrollment (Ptrend < .001). However, among those with CIN2-3, HPV18 DNA load was not associated with severity of cervical cytology at enrollment (Ptrend = .33). The ratios of geometric means of HPV18 DNA load at enrollment among women with CIN2-3, relative to those without, were 6.06 (95% confidence interval [CI] = 0.31 to 117.92) for those with normal cytology at enrollment, 0.50 (95% CI = 0.10 to 2.44) for those with ASC-US, 0.11 (95% CI = 0.03 to 0.46) for those with LSIL, and 0.07 (95% CI = 0.01 to 0.80) for those with high-grade squamous intraepithelial lesion (HSIL). After adjusting for age and coinfection with other high-risk HPVs, a statistically significant association of lower HPV18 DNA load with CIN2-3 was observed among women with LSIL or HSIL at enrollment (P = .02). Within the 2-year period, HPV18 DNA load was unrelated to the timing of CIN2-3 diagnosis. Overall results were similar when the outcome was CIN3.
CONCLUSIONS: HPV18 DNA load was higher for women with LSIL or HSIL at enrollment with no evidence of CIN2-3 during the 2-year follow-up period than it was for women with CIN2-3. Thus, testing for high levels of HPV18 DNA does not appear to be clinically useful.

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Year:  2009        PMID: 19176451      PMCID: PMC2724855          DOI: 10.1093/jnci/djn461

Source DB:  PubMed          Journal:  J Natl Cancer Inst        ISSN: 0027-8874            Impact factor:   13.506


  52 in total

1.  Improved amplification of genital human papillomaviruses.

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2.  The log multinomial regression model for nominal outcomes with more than two attributes.

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3.  Viral load of human papilloma virus 16 as a determinant for development of cervical carcinoma in situ: a nested case-control study.

Authors:  A M Josefsson; P K Magnusson; N Ylitalo; P Sørensen; P Qwarforth-Tubbin; P K Andersen; M Melbye; H O Adami; U B Gyllensten
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4.  ASCUS-LSIL Triage Study. Design, methods and characteristics of trial participants.

Authors:  M Schiffman; M E Adrianza
Journal:  Acta Cytol       Date:  2000 Sep-Oct       Impact factor: 2.319

5.  Effect of cervical cytologic status on the association between human papillomavirus type 16 DNA load and the risk of cervical intraepithelial neoplasia grade 3.

Authors:  Long Fu Xi; Nancy B Kiviat; Denise A Galloway; Xiao-Hua Zhou; Jesse Ho; Laura A Koutsky
Journal:  J Infect Dis       Date:  2008-08-01       Impact factor: 5.226

6.  Type-dependent integration frequency of human papillomavirus genomes in cervical lesions.

Authors:  Svetlana Vinokurova; Nicolas Wentzensen; Irene Kraus; Ruediger Klaes; Corina Driesch; Peter Melsheimer; Fjodor Kisseljov; Mattias Dürst; Achim Schneider; Magnus von Knebel Doeberitz
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7.  Viral DNA load, physical status and E2/E6 ratio as markers to grade HPV16 positive women for high-grade cervical lesions.

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8.  Human papillomavirus DNA integration and messenger RNA transcription in cervical low- and high-risk squamous intraepithelial lesions in Austrian women.

Authors:  M Manavi; G Hudelist; A Fink-Retter; D Gschwantler-Kaulich; K Pischinger; K Czerwenka
Journal:  Int J Gynecol Cancer       Date:  2007-06-22       Impact factor: 3.437

9.  Variation in human papillomavirus type-16 viral load within different histological grades of cervical neoplasia.

Authors:  A N Fiander; K W Hart; S J Hibbitts; G C Rieck; A J Tristram; R W Beukenholdt; N G Powell
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10.  Distribution of human papillomavirus types in different histological subtypes of cervical adenocarcinoma.

Authors:  Ita Hadzisejdć; Maja Krasević; Herman Haller; Blazenka Grahovac
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  14 in total

1.  Changes in DNA Level of Oncogenic Human Papillomaviruses Other Than Types 16 and 18 in Relation to Risk of Cervical Intraepithelial Neoplasia Grades 2 and 3.

Authors:  Long Fu Xi; Mark Schiffman; James P Hughes; Denise A Galloway; Laura A Koutsky; Nancy B Kiviat
Journal:  Cancer Epidemiol Biomarkers Prev       Date:  2019-05-17       Impact factor: 4.254

2.  Increase in viral load, viral integration, and gain of telomerase genes during uterine cervical carcinogenesis can be simultaneously assessed by the HPV 16/18 MLPA-assay.

Authors:  Wendy Theelen; Ernst-Jan M Speel; Michael Herfs; Martin Reijans; Guus Simons; Els V Meulemans; Marcella M Baldewijns; Frans C S Ramaekers; Joan Somja; Philippe Delvenne; Anton H N Hopman
Journal:  Am J Pathol       Date:  2010-09-02       Impact factor: 4.307

3.  Type-dependent association between risk of cervical intraepithelial neoplasia and viral load of oncogenic human papillomavirus types other than types 16 and 18.

Authors:  Long Fu Xi; Mark Schiffman; Yang Ke; James P Hughes; Denise A Galloway; Zhonghu He; Ayaka Hulbert; Rachel L Winer; Laura A Koutsky; Nancy B Kiviat
Journal:  Int J Cancer       Date:  2017-01-24       Impact factor: 7.396

4.  Quantitative human papillomavirus 16 and 18 levels in incident infections and cervical lesion development.

Authors:  Rachel L Winer; Tiffany G Harris; Long Fu Xi; Kathrin U Jansen; James P Hughes; Qinghua Feng; Carolee Welebob; Jesse Ho; Shu-Kuang Lee; Joseph J Carter; Denise A Galloway; Nancy B Kiviat; Laura A Koutsky
Journal:  J Med Virol       Date:  2009-04       Impact factor: 2.327

5.  Six-year regression and progression of cervical lesions of different human papillomavirus viral loads in varied histological diagnoses.

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Journal:  Int J Gynecol Cancer       Date:  2013-05       Impact factor: 3.437

6.  Relationship between cigarette smoking and human papilloma virus types 16 and 18 DNA load.

Authors:  Long Fu Xi; Laura A Koutsky; Philip E Castle; Zoe R Edelstein; Craig Meyers; Jesse Ho; Mark Schiffman
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7.  Human papillomavirus types 16 and 18 DNA load in relation to coexistence of other types, particularly those in the same species.

Authors:  Long Fu Xi; Zoe R Edelstein; Craig Meyers; Jesse Ho; Stephen L Cherne; Mark Schiffman
Journal:  Cancer Epidemiol Biomarkers Prev       Date:  2009-08-18       Impact factor: 4.254

8.  Human Papillomavirus (HPV) type 16 and type 18 DNA Loads at Baseline and Persistence of Type-Specific Infection during a 2-year follow-up.

Authors:  Long Fu Xi; James P Hughes; Zoe R Edelstein; Nancy B Kiviat; Laura A Koutsky; Constance Mao; Jesse Ho; Mark Schiffman
Journal:  J Infect Dis       Date:  2009-12-01       Impact factor: 5.226

9.  HR-HPV viral load quality detection provide more accurate prediction for residual lesions after treatment: a prospective cohort study in patients with high-grade squamous lesions or worse.

Authors:  Lihua Chen; Binhua Dong; Qiaoyu Zhang; Xiaodan Mao; Wenyu Lin; Guanyu Ruan; Yafang Kang; Pengming Sun
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10.  Is human papillomavirus viral load a clinically useful predictive marker? A longitudinal study.

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Journal:  Cancer Epidemiol Biomarkers Prev       Date:  2010-03-03       Impact factor: 4.254

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