Literature DB >> 18771980

Maternal hypercholesterolemia leads to activation of endogenous cholesterol synthesis in the offspring.

Nima Goharkhay1, Esther H Tamayo, Huaizhi Yin, Gary D V Hankins, George R Saade, Monica Longo.   

Abstract

OBJECTIVE: The purpose of this study was to determine the effect of maternal hypercholesterolemia on hepatic cholesterol metabolism in the offspring in a mouse model. STUDY
DESIGN: Male and female wild type and apoE(-/-KO) (knockout for the apoprotein E [apoE]) gene) mice were crossbred to obtain all 4 possible genetic offspring types. The litters were maintained on regular chow and sacrificed at 8 months of age. Liver samples were collected and the mRNA expression levels for SCAP, SREBP-1a, SREBP-2, HMGCR, and LDLR determined using real-time RT-PCR.
RESULTS: We found a significant activation of the transcriptional activity of genes involved in endogenous cholesterol synthesis, as well as LDLR, in the liver of adult mice born to hypercholesterolemic dams.
CONCLUSION: Reprogramming of hepatic cholesterol homeostasis may be the basis for an increased predisposition to hypercholesterolemia and atherosclerosis found in offspring of mice exposed to a high cholesterol environment during early life.

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Year:  2008        PMID: 18771980      PMCID: PMC2574210          DOI: 10.1016/j.ajog.2008.06.064

Source DB:  PubMed          Journal:  Am J Obstet Gynecol        ISSN: 0002-9378            Impact factor:   8.661


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