Literature DB >> 16140998

Lack of association of p53 polymorphisms and haplotypes in high and normal tension open angle glaucoma.

D P Dimasi, A W Hewitt, C M Green, D A Mackey, J E Craig.   

Abstract

BACKGROUND: The final common pathway for open angle glaucoma (OAG) is retinal ganglion cell apoptosis. Polymorphisms in p53, a major regulator of apoptosis, affect the efficiency of cell death induction. Association studies of p53 haplotypes and OAG have had conflicting results.
OBJECTIVE: To examine the association between p53 haplotypes and OAG in a larger white population than in previous reports, and extend the analysis to normal tension glaucoma.
METHODS: 345 unrelated people with OAG were recruited (283 subjects with high tension glaucoma and 62 with normal tension glaucoma) and compared with 178 age matched controls. Genomic DNA was analysed for the p53 codon 72 Arg/Pro polymorphism as well as for the presence or absence of a 16 bp intron 3 insertion.
RESULTS: In this white cohort no association was found between glaucoma (high or normal tension) and either sequence variant or haplotype.
CONCLUSIONS: The p53 codon 72 Arg/Pro polymorphism is not associated with age of onset or severity of glaucoma.

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Year:  2005        PMID: 16140998      PMCID: PMC1736135          DOI: 10.1136/jmg.2005.032458

Source DB:  PubMed          Journal:  J Med Genet        ISSN: 0022-2593            Impact factor:   6.318


  11 in total

1.  WDR36 and P53 gene variants and susceptibility to primary open-angle glaucoma: analysis of gene-gene interactions.

Authors:  Cristina Blanco-Marchite; Francisco Sánchez-Sánchez; María-Pilar López-Garrido; Mercedes Iñigez-de-Onzoño; Francisco López-Martínez; Enrique López-Sánchez; Lydia Alvarez; Pedro-Pablo Rodríguez-Calvo; Carmen Méndez-Hernández; Luis Fernández-Vega; Julián García-Sánchez; Miguel Coca-Prados; Julián García-Feijoo; Julio Escribano
Journal:  Invest Ophthalmol Vis Sci       Date:  2011-10-31       Impact factor: 4.799

2.  The primary open-angle glaucoma gene WDR36 functions in ribosomal RNA processing and interacts with the p53 stress-response pathway.

Authors:  Jonathan M Skarie; Brian A Link
Journal:  Hum Mol Genet       Date:  2008-05-10       Impact factor: 6.150

Review 3.  Complex genetic mechanisms in glaucoma: an overview.

Authors:  Kollu N Rao; Srujana Nagireddy; Subhabrata Chakrabarti
Journal:  Indian J Ophthalmol       Date:  2011-01       Impact factor: 1.848

4.  Association between p53 codon 72 (Arg72Pro) polymorphism and primary open-angle glaucoma in Iranian patients.

Authors:  Hossein Neamatzadeh; Reza Soleimanizad; Aref Atefi; Masoud Zare-Shehneh; Saba Gharibi; Abolfazl Shekari; Amir Bahman Rahimzadeh
Journal:  Iran Biomed J       Date:  2015

5.  Association of polymorphisms in APOE, p53, and p21 with primary open-angle glaucoma in Turkish patients.

Authors:  E Saglar; D Yucel; B Bozkurt; R K Ozgul; M Irkec; A Ogus
Journal:  Mol Vis       Date:  2009-06-30       Impact factor: 2.367

6.  Primary open angle glaucoma in a Caucasian population is associated with the p53 codon 72 polymorphism.

Authors:  Christopher L Daugherty; Hilda Curtis; Tony Realini; Judie F Charlton; Sepideh Zareparsi
Journal:  Mol Vis       Date:  2009-09-22       Impact factor: 2.367

7.  Lack of association between p53 gene polymorphisms and primary open angle glaucoma in the Japanese population.

Authors:  Fumihiko Mabuchi; Yoichi Sakurada; Kenji Kashiwagi; Zentaro Yamagata; Hiroyuki Iijima; Shigeo Tsukahara
Journal:  Mol Vis       Date:  2009-05-20       Impact factor: 2.367

8.  The p53 codon 72 PRO/PRO genotype may be associated with initial central visual field defects in caucasians with primary open angle glaucoma.

Authors:  Janey L Wiggs; Alex W Hewitt; Bao Jian Fan; Dan Yi Wang; Dayse R Figueiredo Sena; Colm O'Brien; Anthony Realini; Jamie E Craig; David P Dimasi; David A Mackey; Jonathan L Haines; Louis R Pasquale
Journal:  PLoS One       Date:  2012-09-26       Impact factor: 3.240

9.  Different WDR36 mutation pattern in Chinese patients with primary open-angle glaucoma.

Authors:  Bao Jian Fan; Dan Yi Wang; Ching-Yu Cheng; Wendy Charles Ko; Shun Chiu Lam; Chi Pui Pang
Journal:  Mol Vis       Date:  2009-04-03       Impact factor: 2.367

10.  Increase in retinal ganglion cells' susceptibility to elevated intraocular pressure and impairment of their endogenous neuroprotective mechanism by age.

Authors:  Hani Levkovitch-Verbin; Shelly Vander; Daria Makarovsky; Fabio Lavinsky
Journal:  Mol Vis       Date:  2013-09-26       Impact factor: 2.367

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