Literature DB >> 15978108

TAFI gene haplotypes, TAFI plasma levels and future risk of coronary heart disease: the PRIME Study.

P E Morange1, D A Tregouet, C Frere, G Luc, D Arveiler, J Ferrieres, P Amouyel, A Evans, P Ducimetiere, F Cambien, L Tiret, I Juhan-Vague.   

Abstract

OBJECTIVES: To evaluate the association of thrombin-activatable fibrinolysis inhibitor (TAFI) gene polymorphisms with the risk of coronary heart disease (CHD) and with TAFI levels measured by a newly developed enzyme-linked immunosorbent assay (ELISA) (TAFI-1B1), shown to be a reliable method for detecting quantitative variations in circulating TAFI. PATIENTS/
METHODS: Six polymorphisms (C-2599G, G-438A, Ala147Thr, Thr325Ile, C + 1542G and T + 1583A) were genotyped and baseline plasma concentrations of TAFI were measured in a nested case-control design as part of the Prospective Epidemiological Study of Myocardial Infarction (PRIME) Study. Participants from France and Northern Ireland who had developed a CHD event during a 5-year follow-up (n = 321) were compared with 645 population- and age-matched control subjects.
RESULTS: In France, the Thr147 allele was more frequent in cases than in controls (0.41 vs. 0.32; P = 0.02), whereas the reverse was observed in Northern Ireland (0.33 vs. 0.38; P = 0.19) (P = 0.01 for interaction). No other polymorphism was associated with CHD risk. Consistent with the results derived from the single-locus analysis, haplotype analysis revealed that the haplotype carrying the Thr147 allele was associated with increased risk of CHD in France while the reverse tended to hold in the Northern Ireland population. Single-locus and haplotype analyses revealed that two polymorphisms, C-2599G and Ala147Thr (or T + 1583A that is in nearly complete association with it), had additive effects on TAFI levels and explained >18% of TAFI variability. This effect was homogeneous in France and Northern Ireland, and in cases and controls who exhibited similar TAFI levels.
CONCLUSIONS: Thrombin-activatable fibrinolysis inhibitor gene polymorphisms are strongly associated to plasma TAFI levels, but the relation to CHD risk is less clear.

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Year:  2005        PMID: 15978108     DOI: 10.1111/j.1538-7836.2005.01486.x

Source DB:  PubMed          Journal:  J Thromb Haemost        ISSN: 1538-7836            Impact factor:   5.824


  11 in total

1.  A genome-wide exploration suggests an oligogenic model of inheritance for the TAFI activity and its antigen levels.

Authors:  Maria Sabater-Lleal; Alfonso Buil; Juan Carlos Souto; Laura Alamsy; Montserrat Borrell; Mark Lathrop; John Blangero; Jordi Fontcuberta; José Manuel Soria
Journal:  Hum Genet       Date:  2008-06-18       Impact factor: 4.132

2.  Lack of association of genetic variants in the LRP8 gene with familial and sporadic myocardial infarction.

Authors:  Wolfgang Lieb; Tanja Zeller; Massimo Mangino; Anika Götz; Peter Braund; Juergen J Wenzel; Christian Horn; Carole Proust; Patrick Linsel-Nitschke; Philippe Amouyel; Petra Bruse; Dominique Arveiler; Inke R König; Jean Ferrières; Andreas Ziegler; Anthony J Balmforth; Alun Evans; Pierre Ducimetière; Francois Cambien; Christian Hengstenberg; Klaus Stark; Alistair S Hall; Heribert Schunkert; Stefan Blankenberg; Nilesh J Samani; Jeanette Erdmann; Laurence Tiret
Journal:  J Mol Med (Berl)       Date:  2008-07-01       Impact factor: 4.599

3.  Low thrombin activatable fibrinolysis inhibitor activity levels are associated with an increased risk of a first myocardial infarction in men.

Authors:  Mirjam E Meltzer; Carine J M Doggen; Philip G de Groot; Joost C M Meijers; Frits R Rosendaal; Ton Lisman
Journal:  Haematologica       Date:  2009-04-18       Impact factor: 9.941

4.  Genetic variation in thrombin-activatable fibrinolysis inhibitor is associated with the risk of diabetic nephropathy.

Authors:  C-W Xu; X-B Wu; X-L Ma; Y-S Wang; B-C Zhang; J-J Zhao; Z-J Wang; J Chen
Journal:  J Endocrinol Invest       Date:  2012-07       Impact factor: 5.467

Review 5.  Carboxypeptidase U (CPU, TAFIa, CPB2) in Thromboembolic Disease: What Do We Know Three Decades after Its Discovery?

Authors:  Karen Claesen; Joachim C Mertens; Dorien Leenaerts; Dirk Hendriks
Journal:  Int J Mol Sci       Date:  2021-01-17       Impact factor: 5.923

6.  Polymorphic variation of genes in the fibrinolytic system and the risk of ovarian cancer.

Authors:  Yaakov Bentov; Theodore J Brown; Mohammad R Akbari; Robert Royer; Harvey Risch; Barry Rosen; John McLaughlin; Ping Sun; Shiyu Zhang; Steven A Narod; Robert F Casper
Journal:  PLoS One       Date:  2009-06-15       Impact factor: 3.240

7.  Data supporting the structural and functional characterization of Thrombin-Activatable Fibrinolysis Inhibitor in breast cancer.

Authors:  Manal S Fawzy; Eman A Toraih
Journal:  Data Brief       Date:  2015-11-14

8.  Ambivalent roles of carboxypeptidase B in the lytic susceptibility of fibrin.

Authors:  András Kovács; László Szabó; Colin Longstaff; Kiril Tenekedjiev; Raymund Machovich; Krasimir Kolev
Journal:  Thromb Res       Date:  2013-09-21       Impact factor: 3.944

9.  A Genome-wide Study of Common and Rare Genetic Variants Associated with Circulating Thrombin Activatable Fibrinolysis Inhibitor.

Authors:  Tara M Stanne; Maja Olsson; Erik Lorentzen; Annie Pedersen; Anders Gummesson; Ann Gils; Katarina Jood; Gunnar Engström; Olle Melander; Paul J Declerck; Christina Jern
Journal:  Thromb Haemost       Date:  2018-01-29       Impact factor: 5.249

10.  Genetic Polymorphisms Associated with Thrombotic Disease Comparison of Two Territories: Myocardial Infarction and Ischemic Stroke.

Authors:  Irma Isordia-Salas; Manuel Martínez-Marino; Paolo Alberti-Minutti; María Tania Ricardo-Moreno; Ricardo Castro-Calvo; David Santiago-Germán; José Antonio Alvarado-Moreno; Cristian Calleja-Carreño; Jesús Hernández-Juárez; Alfredo Leaños-Miranda; Abraham Majluf-Cruz
Journal:  Dis Markers       Date:  2019-10-30       Impact factor: 3.434

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