Literature DB >> 19377074

Low thrombin activatable fibrinolysis inhibitor activity levels are associated with an increased risk of a first myocardial infarction in men.

Mirjam E Meltzer1, Carine J M Doggen, Philip G de Groot, Joost C M Meijers, Frits R Rosendaal, Ton Lisman.   

Abstract

BACKGROUND: Studies on the relation between thrombin activatable fibrinolysis inhibitor (TAFI) and arterial thrombosis have produced conflicting results. TAFI regulates fibrinolysis, but other roles of this inhibitor, including anti-inflammatory properties, have also been demonstrated. DESIGN AND METHODS: We investigated the association between TAFI activity and the risk of myocardial infarction. Additionally, we studied the association of common single nucleotide polymorphisms in the TAFI gene with levels of the TAFI protein and risk of myocardial infarction.We included 554 men under 70 years old with a first myocardial infarction and 643 controls participating in the Study of Myocardial Infarctions Leiden (SMILE), a case-control study.
RESULTS: We found odds ratios (95% confidence intervals) of a first myocardial infarction of 2.4 (1.6-3.6), 3.2 (2.1-4.7) and 3.4 (2.3-5.1) for subjects whose TAFI levels were in the third, second and first quartiles (lowest TAFI levels), respectively, compared with the fourth quartile, after adjusting for arterial disease risk factors. The rare -438A and 1040T alleles were associated with lower, and the rare 505G allele with higher TAFI levels than the common alleles. Carriers of the -438A allele had an increased risk of myocardial infarction (odds ratio 1.6 (1.0-2.5) for AA; odds ratio 1.2 (0.9-1.5) for AG compared with GG). The other single nucleotide polymorphisms were not associated with myocardial infarction.
CONCLUSIONS: Low TAFI activity levels are associated with increased risk of a first myocardial infarction in men. The results on the association between TAFI single nucleotide polymorphisms and myocardial infarction were inconsistent.

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Year:  2009        PMID: 19377074      PMCID: PMC2688572          DOI: 10.3324/haematol.2008.002386

Source DB:  PubMed          Journal:  Haematologica        ISSN: 0390-6078            Impact factor:   9.941


  37 in total

Review 1.  Curiouser and curiouser: recent advances in measurement of thrombin-activatable fibrinolysis inhibitor (TAFI) and in understanding its molecular genetics, gene regulation, and biological roles.

Authors:  Michael B Boffa; Marlys L Koschinsky
Journal:  Clin Biochem       Date:  2006-11-21       Impact factor: 3.281

2.  In vivo regulation of plasminogen function by plasma carboxypeptidase B.

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3.  Two naturally occurring variants of TAFI (Thr-325 and Ile-325) differ substantially with respect to thermal stability and antifibrinolytic activity of the enzyme.

Authors:  Mark Schneider; Michael Boffa; Ronald Stewart; Mona Rahman; Marlys Koschinsky; Michael Nesheim
Journal:  J Biol Chem       Date:  2001-10-29       Impact factor: 5.157

4.  Plasma thrombin-activatable fibrinolysis inhibitor antigen concentration and genotype in relation to myocardial infarction in the north and south of Europe.

Authors:  I Juhan-Vague; P E Morange; H Aubert; M Henry; M F Aillaud; M C Alessi; A Samnegård; E Hawe; J Yudkin; M Margaglione; G Di Minno; A Hamsten; S E Humphries
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6.  Effect of single nucleotide polymorphisms on expression of the gene encoding thrombin-activatable fibrinolysis inhibitor: a functional analysis.

Authors:  Michael B Boffa; Deborah Maret; Jeffrey D Hamill; Nazareth Bastajian; Paul Crainich; Nancy S Jenny; Zhonghua Tang; Elizabeth M Macy; Russell P Tracy; Rendrik F Franco; Michael E Nesheim; Marlys L Koschinsky
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Review 7.  Inflammation in atherosclerosis.

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8.  Absolute risk of venous and arterial thromboembolism in thrombophilic families is not increased by high thrombin-activatable fibrinolysis inhibitor (TAFI) levels.

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Authors:  Eduard J Libourel; Ivan Bank; Johan R Meinardi; Corinne P Baljé -Volkers; Karly Hamulyak; Saskia Middeldorp; Maria M W Koopman; Elisabeth C M van Pampus; Martin H Prins; Harry R Büller; Jan van der Meer
Journal:  Haematologica       Date:  2002-10       Impact factor: 9.941

10.  Crystal structures of TAFI elucidate the inactivation mechanism of activated TAFI: a novel mechanism for enzyme autoregulation.

Authors:  Pauline F Marx; T Harma C Brondijk; Tom Plug; Roland A Romijn; Wieger Hemrika; Joost C M Meijers; Eric G Huizinga
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  12 in total

Review 1.  Fibrinolysis and the control of blood coagulation.

Authors:  John C Chapin; Katherine A Hajjar
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2.  Increase in plasma thrombin-activatable fibrinolysis inhibitor may not contribute to thrombotic tendency in antiphospholipid syndrome because of inhibitory potential of antiphospholipid antibodies toward TAFI activation.

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3.  Thrombin-activatable fibrinolysis inhibitor (TAFI) deficient mice are susceptible to intracerebral thrombosis and ischemic stroke.

Authors:  Peter Kraft; Tobias Schwarz; Joost C M Meijers; Guido Stoll; Christoph Kleinschnitz
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Review 4.  Carboxypeptidase U (TAFIa): a new drug target for fibrinolytic therapy?

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5.  Sex-specific effect of CPB2 Ala147Thr but not Thr325Ile variants on the risk of venous thrombosis: A comprehensive meta-analysis.

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7.  Thrombin-Activatable Fibrinolysis Inhibitor Polymorphisms and Cerebral Venous Thrombosis in Mexican Mestizo Patients.

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8.  Type 2 diabetes as a modifier of fibrin clot properties in patients with coronary artery disease.

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9.  Data supporting the structural and functional characterization of Thrombin-Activatable Fibrinolysis Inhibitor in breast cancer.

Authors:  Manal S Fawzy; Eman A Toraih
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10.  Association between polymorphisms in the flanking region of the TAFI gene and atherosclerotic cerebral infarction in a Chinese population.

Authors:  You Li; Zhiliang Zeng; Jianghao Zhao; Guoda Ma; Lili Cui; Hua Tao; Zhijun Lin; Yanyan Chen; Bin Zhao; Yusen Chen; Keshen Li
Journal:  Lipids Health Dis       Date:  2014-05-13       Impact factor: 3.876

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