Deficits to global motion processing in human amblyopia.

We investigated global motion processing in a group of adult amblyopes using a method that allows us to factor out any influence of the known contrast sensitivity deficit. We show that there are independent global motion processing deficits in human amblyopia that are unrelated to the contrast sensitivity deficit, and that are more extensive for contrast-defined than for luminance-defined stimuli. We speculate that the site of these deficits must include the extra-striate cortex and in particular the dorsal pathway.