Literature DB >> 12438434

Orchestrating the unfolded protein response in health and disease.

Randal J Kaufman1.   

Abstract

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Year:  2002        PMID： 12438434      PMCID： PMC151822          DOI： 10.1172/JCI16886

Source DB:  PubMed          Journal:  J Clin Invest        ISSN： 0021-9738            Impact factor:   14.808


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  87 in total

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Review 2.  Mammalian stress response: induction of the glucose-regulated protein family.

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4.  Regulated translation initiation controls stress-induced gene expression in mammalian cells.

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Journal:  Mol Cell       Date:  2000-11       Impact factor: 17.970

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7.  Overexpression of Pex15p, a phosphorylated peroxisomal integral membrane protein required for peroxisome assembly in S.cerevisiae, causes proliferation of the endoplasmic reticulum membrane.

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8.  Genetic tailoring of N-linked oligosaccharides: the role of glucose residues in glycoprotein processing of Saccharomyces cerevisiae in vivo.

Authors:  C A Jakob; P Burda; S te Heesen; M Aebi; J Roth
Journal:  Glycobiology       Date:  1998-02       Impact factor: 4.313

9.  A lag in intracellular degradation of mutant alpha 1-antitrypsin correlates with the liver disease phenotype in homozygous PiZZ alpha 1-antitrypsin deficiency.

Authors:  Y Wu; I Whitman; E Molmenti; K Moore; P Hippenmeyer; D H Perlmutter
Journal:  Proc Natl Acad Sci U S A       Date:  1994-09-13       Impact factor: 11.205

10.  Inducible membranes in yeast: relation to the unfolded-protein-response pathway.

Authors:  R Menzel; F Vogel; E Kärgel; W H Schunck
Journal:  Yeast       Date:  1997-10       Impact factor: 3.239
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Review 1.  Protein aggregation in disease: a role for folding intermediates forming specific multimeric interactions.

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2.  The cellular response to aggregated proteins associated with human disease.

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Review 3.  Rescuing protein conformation: prospects for pharmacological therapy in cystic fibrosis.

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Review 4.  Translational control in the endoplasmic reticulum stress response.

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Review 8.  Gene Therapy Strategies to Restore ER Proteostasis in Disease.

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9.  Decreased contraction induced by endothelium-derived contracting factor in prolonged treatment of rat renal artery with endoplasmic reticulum stress inducer.

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10.  Translational control of cytochrome c by RNA-binding proteins TIA-1 and HuR.

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