Literature DB >> 10892877

Photodamage to human RPE cells by A2-E, a retinoid component of lipofuscin.

F Schütt1, S Davies, J Kopitz, F G Holz, M E Boulton.   

Abstract

PURPOSE: A fluorescent component of lipofuscin, A2-E (N-retinylidene-N-retinylethanol-amine) has been shown to impair lysosomal function and to increase the intralysosomal pH of human retinal pigment epithelial (RPE) cells. In addition to its lysosomotropic properties A2-E is known to be photoreactive. The purpose of this study was to determine the phototoxic potential of A2-E on RPE cells.
METHODS: A2-E (synthesized by coupling all-trans-retinaldehyde to ethanolamine) was complexed to low-density lipoprotein (LDL) to allow for specific loading of the lysosomal compartment. Human RPE cell cultures were loaded with the A2-E-LDL complex four times within 2 weeks. A2-E accumulation was confirmed by fluorescence microscopy and flow cytometry analysis. Acridine orange staining allowed assessment of lysosomal integrity and intralysosomal pH. The phototoxic properties of A2-E were determined by exposing A2-E-free and A2-E-fed RPE cell cultures to short wavelength visible light (400-500 nm) and assessing cell viability and lysosomal integrity.
RESULTS: Fluorescence microscopy and flow cytometry analysis demonstrated that the intralysosomal accumulation of A2-E in cultured RPE cells increased with the number of feedings. Acridine orange staining confirmed that the A2-E was located in the lysosomal compartment and induced an elevation of intralysosomal pH. Exposure of A2-E-fed cells to light resulted in a significant loss of cell viability by 72 hours, which was not observed in either RPE cells maintained in the dark or A2-E-free cultures exposed to light. Toxicity was associated with a loss of lysosomal integrity.
CONCLUSIONS: A2-E is detrimental to RPE cell function by a variety of mechanisms: inhibition of lysosomal degradative capacity, loss of membrane integrity, and phototoxicity. Such mechanisms could contribute to retinal aging as well as retinal diseases associated with excessive lipofuscin accumulation-for example, age-related macular degeneration and Stargardt's disease.

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Year:  2000        PMID: 10892877

Source DB:  PubMed          Journal:  Invest Ophthalmol Vis Sci        ISSN: 0146-0404            Impact factor:   4.799


  76 in total

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Review 3.  Ageing of the retinal pigment epithelium: implications for transplantation.

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Review 6.  Keypathophysiologic pathways in age-related macular disease.

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7.  Rpe65 Leu450Met variant is associated with reduced levels of the retinal pigment epithelium lipofuscin fluorophores A2E and iso-A2E.

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8.  [RPE transplantation: the challenges and the future].

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Journal:  Ophthalmologe       Date:  2004-09       Impact factor: 1.059

9.  Quantitative measurements of autofluorescence with the scanning laser ophthalmoscope.

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10.  Anthocyanins protect against A2E photooxidation and membrane permeabilization in retinal pigment epithelial cells.

Authors:  Young P Jang; Jilin Zhou; Koji Nakanishi; Janet R Sparrow
Journal:  Photochem Photobiol       Date:  2005 May-Jun       Impact factor: 3.421

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