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Literature DB >> 985444

Leucine and D-3-hydroxybutyrate as lipid precursors in developing rat spinal cord and liver.

Abstract

By using a labelled ketone body, D-3-hydroxy[3-14C]butyrate, or a ketone-body precursor, L-[4,5-3H]leucine, preferential labelling of spinal-cord cholesterol was demonstrated. In contrast, the phospholipid fraction was more heavily labelled in the liver. In both tissues phosphatidylcholine was the major labelled complex lipid. Incorporation ob both substrates into total lipid, on a tissue-weight basis, was appreciably greater for spinal cord.

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Year: 1976 PMID： 985444 PMCID： PMC1163997 DOI： 10.1042/bj1580501

Source DB: PubMed Journal: Biochem J ISSN： 0264-6021 Impact factor: 3.857

18 in total

1. The metabolism of ketone bodies in developing human brain: development of ketone-body-utilizing enzymes and ketone bodies as precursors for lipid synthesis.

Authors: M S Patel; C A Johnson; R Rajan; O E Owen
Journal: J Neurochem Date: 1975-12 Impact factor: 5.372

2. BRAIN CHOLESTEROL. VI. THE EFFECT OF HEREDITARY DYSTROPHIA MUSCULARIS ON (C14)LEUCINE AND (2-3H)ACETATE INCORPORATION.

Authors: J J KABARA
Journal: Tex Rep Biol Med Date: 1964

3. Metabolism of myelin: the persistence of (4-14C) cholesterol in the mammalian central nervous system.

Authors: A N DAVISON
Journal: Lancet Date: 1959-03-28 Impact factor: 79.321

4. A simple method for the isolation and purification of total lipides from animal tissues.

Authors: J FOLCH; M LEES; G H SLOANE STANLEY
Journal: J Biol Chem Date: 1957-05 Impact factor: 5.157

5. Studies on the biosynthesis of cholesterol. III. Purification of C14-cholesterol from perfusions of livers and other organs.

Authors: E SCHWENK; N T WERTHESSEN
Journal: Arch Biochem Biophys Date: 1952-10 Impact factor: 4.013

4. Phosphatidylserine decarboxylase 1 (Psd1) promotes mitochondrial fusion by regulating the biophysical properties of the mitochondrial membrane and alternative topogenesis of mitochondrial genome maintenance protein 1 (Mgm1).

Authors: Eliana Y L Chan; G Angus McQuibban
Journal: J Biol Chem Date: 2012-10-08 Impact factor: 5.157

5. NADP-dependent dehydrogenases in rat liver parenchyma. III. The description of a liponeogenic area on the basis of histochemically demonstrated enzyme activities and the neutral fat content during fasting and refeeding.

Authors: H Rieder
Journal: Histochemistry Date: 1981

5 in total

Leucine and D-3-hydroxybutyrate as lipid precursors in developing rat spinal cord and liver.

1. The metabolism of ketone bodies in developing human brain: development of ketone-body-utilizing enzymes and ketone bodies as precursors for lipid synthesis.

2. BRAIN CHOLESTEROL. VI. THE EFFECT OF HEREDITARY DYSTROPHIA MUSCULARIS ON (C14)LEUCINE AND (2-3H)ACETATE INCORPORATION.

3. Metabolism of myelin: the persistence of (4-14C) cholesterol in the mammalian central nervous system.

4. A simple method for the isolation and purification of total lipides from animal tissues.

5. Studies on the biosynthesis of cholesterol. III. Purification of C14-cholesterol from perfusions of livers and other organs.

Review 6. Metabolism of ketone bodies by the brain.

7. Labelling of lipids by radioactive amino acids in the central nervous system.

8. Ketone bodies as precursors of sterols and fatty acids in the developing rat.

9. Leucine and mevalonate as precursors of serum cholesterol in man.

10. Activities of enzymes of ketone-body utilization in brain and other tissues of suckling rats.

1. Isolation of alveolar type II cells by centrifugal elutriation.

Review 2. The contribution of the cholesterol biosynthetic pathway to intermediary metabolism and cell function.

3. Age-related quantitative changes in enzyme activities of rat brain.

4. Phosphatidylserine decarboxylase 1 (Psd1) promotes mitochondrial fusion by regulating the biophysical properties of the mitochondrial membrane and alternative topogenesis of mitochondrial genome maintenance protein 1 (Mgm1).

5. NADP-dependent dehydrogenases in rat liver parenchyma. III. The description of a liponeogenic area on the basis of histochemically demonstrated enzyme activities and the neutral fat content during fasting and refeeding.