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Literature DB >> 9596782

Secreted aspartyl proteinases and interactions of Candida albicans with human endothelial cells.

A S Ibrahim¹, S G Filler, D Sanglard, J E Edwards, B Hube.

Abstract

The endothelial cell interactions of homozygous null mutants of Candida albicans that were deficient in secreted aspartyl proteinase 1 (Sap1), Sap2, or Sap3 were investigated. Only Sap2 was found to contribute to the ability of C. albicans to damage endothelial cells and stimulate them to express E-selectin. None of the Saps studied appears to play a role in C. albicans adherence to endothelial cells.

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Year: 1998 PMID： 9596782 PMCID： PMC108304 DOI： 10.1128/IAI.66.6.3003-3005.1998

Source DB: PubMed Journal: Infect Immun ISSN： 0019-9567 Impact factor: 3.441

14 in total

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