Literature DB >> 8786067

Angelman syndrome in an inbred family.

J Beuten1, R C Hennekam, B Van Roy, K Mangelschots, J S Sutcliffe, D J Halley, F A Hennekam, A L Beaudet, P J Willems.   

Abstract

Angelman syndrome (AS) is characterized by severe mental retardation, absent speech, puppet-like movements, inappropriate laughter, epilepsy, and abnormal electroencephalogram. The majority of AS patients (approximately 65%) have a maternal deficiency within chromosomal region 15q11-q13, caused by maternal deletion or paternal uniparental disomy (UPD). Approximately 35% of AS patients exhibit neither detectable deletion nor UPD, but a subset of these patients have abnormal methylation at several loci in the 15q11-q13 region. We describe here three patients with Angelman syndrome belonging to an extended inbred family. High resolution chromosome analysis combined with DNA analysis using 14 marker loci from the 15ql1-q13 region failed to detect a deletion in any of the three patients. Paternal UPD of chromosome 15 was detected in one case, while the other two patients have abnormal methylation at D15S9, D15S63, and SNRPN. Although the three patients are distantly related, the chromosome 15q11-q13 haplotypes are different, suggesting that independent mutations gave rise to AS in this family.

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Year:  1996        PMID: 8786067     DOI: 10.1007/bf02185757

Source DB:  PubMed          Journal:  Hum Genet        ISSN: 0340-6717            Impact factor:   4.132


  31 in total

1.  Angelman syndrome.

Authors:  J Clayton-Smith; M E Pembrey
Journal:  J Med Genet       Date:  1992-06       Impact factor: 6.318

2.  Detection of imprinting mutations in Angelman syndrome using a probe for exon alpha of SNRPN.

Authors:  J Beuten; J S Sutcliffe; B M Casey; A L Beaudet; R C Hennekam; P J Willems
Journal:  Am J Med Genet       Date:  1996-05-17

Review 3.  Recurrence risk in the Angelman ("happy puppet") syndrome.

Authors:  P J Willems; I Dijkstra; O F Brouwer; G P Smit
Journal:  Am J Med Genet       Date:  1987-08

4.  Molecular, cytogenetic, and clinical investigations of Prader-Willi syndrome patients.

Authors:  W P Robinson; A Bottani; Y G Xie; J Balakrishman; F Binkert; M Mächler; A Prader; A Schinzel
Journal:  Am J Hum Genet       Date:  1991-12       Impact factor: 11.025

5.  Dinucleotide repeat polymorphism of D15S10 in the Prader-Willi chromosome region (PWCR).

Authors:  R Lindeman; S Kouts; T Woodage; A Smith; R J Trent
Journal:  Nucleic Acids Res       Date:  1991-10-11       Impact factor: 16.971

6.  Imprinting mutations suggested by abnormal DNA methylation patterns in familial Angelman and Prader-Willi syndromes.

Authors:  A Reis; B Dittrich; V Greger; K Buiting; M Lalande; G Gillessen-Kaesbach; M Anvret; B Horsthemke
Journal:  Am J Hum Genet       Date:  1994-05       Impact factor: 11.025

7.  Molecular diagnosis of the Prader-Willi and Angelman syndromes by detection of parent-of-origin specific DNA methylation in 15q11-13.

Authors:  B Dittrich; W P Robinson; H Knoblauch; K Buiting; K Schmidt; G Gillessen-Kaesbach; B Horsthemke
Journal:  Hum Genet       Date:  1992-11       Impact factor: 4.132

8.  Deletions of a differentially methylated CpG island at the SNRPN gene define a putative imprinting control region.

Authors:  J S Sutcliffe; M Nakao; S Christian; K H Orstavik; N Tommerup; D H Ledbetter; A L Beaudet
Journal:  Nat Genet       Date:  1994-09       Impact factor: 38.330

9.  Linkage analysis with chromosome 15q11-13 markers shows genomic imprinting in familial Angelman syndrome.

Authors:  E J Meijers-Heijboer; L A Sandkuijl; H G Brunner; H J Smeets; A J Hoogeboom; W H Deelen; J O van Hemel; M R Nelen; D F Smeets; M F Niermeijer
Journal:  J Med Genet       Date:  1992-12       Impact factor: 6.318

10.  Functional imprinting and epigenetic modification of the human SNRPN gene.

Authors:  C C Glenn; K A Porter; M T Jong; R D Nicholls; D J Driscoll
Journal:  Hum Mol Genet       Date:  1993-12       Impact factor: 6.150

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  2 in total

1.  The Angelman syndrome-associated protein, E6-AP, is a coactivator for the nuclear hormone receptor superfamily.

Authors:  Z Nawaz; D M Lonard; C L Smith; E Lev-Lehman; S Y Tsai; M J Tsai; B W O'Malley
Journal:  Mol Cell Biol       Date:  1999-02       Impact factor: 4.272

2.  Sporadic imprinting defects in Prader-Willi syndrome and Angelman syndrome: implications for imprint-switch models, genetic counseling, and prenatal diagnosis.

Authors:  K Buiting; B Dittrich; S Gross; C Lich; C Färber; T Buchholz; E Smith; A Reis; J Bürger; M M Nöthen; U Barth-Witte; B Janssen; D Abeliovich; I Lerer; A M van den Ouweland; D J Halley; C Schrander-Stumpel; H Smeets; P Meinecke; S Malcolm; A Gardner; M Lalande; R D Nicholls; K Friend; A Schulze; G Matthijs; H Kokkonen; P Hilbert; L Van Maldergem; G Glover; P Carbonell; P Willems; G Gillessen-Kaesbach; B Horsthemke
Journal:  Am J Hum Genet       Date:  1998-07       Impact factor: 11.025

  2 in total

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