Literature DB >> 8410619

The role of the host response in periodontal disease progression: implications for future treatment strategies.

T E Van Dyke1, M A Lester, L Shapira.   

Abstract

In this paper, we review the relevant aspects of host responses in periodontal diseases as we understand them today. Discussion will focus on neutrophil function, lymphocytes and the immune response, macrophage function, cytokines and complement, fibroblasts and growth factors, and regeneration. Recent literature and concepts will be presented with an emphasis on future directions and application to treatment regimens.

Mesh：

Year:  1993        PMID： 8410619     DOI： 10.1902/jop.1993.64.8s.792

Source DB:  PubMed          Journal:  J Periodontol        ISSN： 0022-3492            Impact factor:   6.993


  25 in total

1.  Actinobacillus actinomycetemcomitans serotype b-specific polysaccharide antigen stimulates production of chemotactic factors and inflammatory cytokines by human monocytes.

Authors:  N Yamaguchi; Y Yamashita; D Ikeda; T Koga
Journal:  Infect Immun       Date:  1996-07       Impact factor: 3.441

Review 2.  Diabetic periodontitis: a model for activated innate immunity and impaired resolution of inflammation.

Authors:  Hamdy Nassar; Alpdogan Kantarci; Thomas E van Dyke
Journal:  Periodontol 2000       Date:  2007       Impact factor: 7.589

3.  Role of the capsular polysaccharide-like serotype-specific antigen in resistance of Actinobacillus actinomycetemcomitans to phagocytosis by human polymorphonuclear leukocytes.

Authors:  N Yamaguchi; M Kawasaki; Y Yamashita; K Nakashima; T Koga
Journal:  Infect Immun       Date:  1995-12       Impact factor: 3.441

Review 4.  Antibacterial and immunomodulatory properties of azithromycin treatment implications for periodontitis.

Authors:  P M Bartold; A H du Bois; S Gannon; D R Haynes; R S Hirsch
Journal:  Inflammopharmacology       Date:  2013-02-28       Impact factor: 4.473

5.  Strain-dependent activation of monocytes and inflammatory macrophages by lipopolysaccharide of Porphyromonas gingivalis.

Authors:  L Shapira; C Champagne; T E Van Dyke; S Amar
Journal:  Infect Immun       Date:  1998-06       Impact factor: 3.441

6.  Shifting the paradigm from inhibitors of inflammation to resolvers of inflammation in periodontitis.

Authors:  Thomas E Van Dyke
Journal:  J Periodontol       Date:  2020-06-20       Impact factor: 6.993

7.  Photodynamic therapy decrease immune-inflammatory mediators levels during periodontal maintenance.

Authors:  Priscila Vivas da Cruz Andrade; Vanessa Tubero Euzebio Alves; Verônica Franco de Carvalho; Michelle De Franco Rodrigues; Claudio Mendes Pannuti; Marinella Holzhausen; Giorgio De Micheli; Marina Clemente Conde
Journal:  Lasers Med Sci       Date:  2016-10-04       Impact factor: 3.161

8.  Use of defined mutants to assess the role of the Campylobacter rectus S-layer in bacterium-epithelial cell interactions.

Authors:  B Wang; E Kraig; D Kolodrubetz
Journal:  Infect Immun       Date:  2000-03       Impact factor: 3.441

9.  Serum antibodies to Porphyromonas gingivalis block the prostaglandin E2 response to lipopolysaccharide by mononuclear cells.

Authors:  B W Bainbridge; R C Page; R P Darveau
Journal:  Infect Immun       Date:  1997-11       Impact factor: 3.441

10.  Myeloperoxidase content is a marker of systemic inflammation in a chronic condition: the example given by the periodontal disease in rats.

Authors:  Celso Martins Queiroz-Junior; Cinthia Mara da Fonseca Pacheco; Allyson Henrique Fonseca; André Klein; Marcelo Vidigal Caliari; Janetti Nogueira de Francischi
Journal:  Mediators Inflamm       Date:  2009-12-31       Impact factor: 4.711
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