Literature DB >> 8301098

High blood pressure and metabolic disorders are associated in the Lyon hypertensive rat.

M Vincent1, E H Boussaïri, R Cartier, M Lo, A Sassolas, C Cerutti, C Barrès, M P Gustin, G Cuisinaud, N J Samani.   

Abstract

OBJECTIVE: A large population of F2 rats, obtained from a cross between male Lyon hypertensive (LH) rats and female Lyon normotensive (LN) rats, was studied in order to assess the relationship between increased body weight, hyperlipidaemia and high blood pressure which characterize LH rats.
METHODS: Mean arterial pressure (MAP) was recorded in male, conscious, freely moving LH, LN, F1 and F2 rats aged 30 weeks. Plasma total cholesterol, high-density lipoprotein-, low-density lipoprotein- and very low-density lipoprotein-cholesterol, phospholipids, triglycerides, insulin and glucose were measured.
RESULTS: In the F2 cohort it was observed that high MAP was a recessive trait that depends on several genes and was unrelated to body weight. The left ventricular weight, corrected for tibia length, was correlated with MAP. Plasma total and high-density lipoprotein-cholesterol and phospholipids concentrations were lower in the F1 rats than in the LN rats, suggesting an overdominance of the LN alleles. In the F2 rats MAP was related to total, high-density lipoprotein- and low-density lipoprotein-cholesterol. Plasma triglycerides, insulin and the insulin:glucose ratio, which were higher in the LH rats than in the LN rats, were also correlated with MAP in the F2 cohort. Using stepwise multiple regression analysis, MAP remained correlated with plasma total cholesterol, insulin and the insulin:glucose ratio, but not with triglycerides.
CONCLUSIONS: Hypertension in LH rats is a recessive trait that is independent of body weight. In addition, the cosegregation of blood pressure with plasma cholesterol and, to a lesser degree, with insulin levels, which was observed in the present study provides the first direct evidence that these phenotypes are associated and are not due simply to genetic drift in the Lyon model.

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Year:  1993        PMID: 8301098

Source DB:  PubMed          Journal:  J Hypertens        ISSN: 0263-6352            Impact factor:   4.844


  11 in total

1.  Quantitative trait loci influencing cholesterol and phospholipid phenotypes map to chromosomes that contain genes regulating blood pressure in the spontaneously hypertensive rat.

Authors:  A Bottger; H A van Lith; V Kren; D Krenová; V Bílá; J Vorlícek; V Zídek; A Musilová; M Zdobinská; J M Wang; B F van Zutphen; T W Kurtz; M Pravenec
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2.  Rat Models of Metabolic Syndrome.

Authors:  Anne E Kwitek
Journal:  Methods Mol Biol       Date:  2019

3.  Systems biology with high-throughput sequencing reveals genetic mechanisms underlying the metabolic syndrome in the Lyon hypertensive rat.

Authors:  Jinkai Wang; Man Chun John Ma; Amanda K Mennie; Janette M Pettus; Yang Xu; Lan Lin; Matthew G Traxler; Jessica Jakoubek; Santosh S Atanur; Timothy J Aitman; Yi Xing; Anne E Kwitek
Journal:  Circ Cardiovasc Genet       Date:  2015-01-08

Review 4.  Opportunities and limitations of genetic analysis of hypertensive rat strains.

Authors:  Juan M Saavedra
Journal:  J Hypertens       Date:  2009-06       Impact factor: 4.844

5.  Implication of chromosome 13 on hypertension and associated disorders in Lyon hypertensive rats.

Authors:  Sophie Gilibert; Alain Bataillard; Juerg Nussberger; Jean Sassard; Anne E Kwitek
Journal:  J Hypertens       Date:  2009-06       Impact factor: 4.844

6.  Measurement of absolute copy number variation reveals association with essential hypertension.

Authors:  Francine Z Marques; Priscilla R Prestes; Leonardo B Pinheiro; Katrina Scurrah; Kerry R Emslie; Maciej Tomaszewski; Stephen B Harrap; Fadi J Charchar
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7.  Genome variation and conserved regulation identify genomic regions responsible for strain specific phenotypes in rat.

Authors:  David Martín-Gálvez; Denis Dunoyer de Segonzac; Man Chun John Ma; Anne E Kwitek; David Thybert; Paul Flicek
Journal:  BMC Genomics       Date:  2017-12-22       Impact factor: 3.969

8.  Contribution of independent and pleiotropic genetic effects in the metabolic syndrome in a hypertensive rat.

Authors:  Man Chun John Ma; Janette M Pettus; Jessica A Jakoubek; Matthew G Traxler; Karen C Clark; Amanda K Mennie; Anne E Kwitek
Journal:  PLoS One       Date:  2017-08-08       Impact factor: 3.240

9.  Cervical vagal nerve stimulation impairs glucose tolerance and suppresses insulin release in conscious rats.

Authors:  Harald M Stauss; Hubert Stangl; Karen C Clark; Anne E Kwitek; Vitor A Lira
Journal:  Physiol Rep       Date:  2018-12

10.  Genomic structure of nucleotide diversity among Lyon rat models of metabolic syndrome.

Authors:  Man Chun John Ma; Santosh S Atanur; Timothy J Aitman; Anne E Kwitek
Journal:  BMC Genomics       Date:  2014-03-14       Impact factor: 3.969

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