Literature DB >> 8221661

Absence of p53 gene mutations in primary neuroblastomas.

K Vogan1, M Bernstein, J M Leclerc, L Brisson, J Brossard, G M Brodeur, J Pelletier, P Gros.   

Abstract

Neuroblastoma is a common childhood malignancy of the sympathetic nervous system. Mutations in p53, a tumor suppressor gene located on the short arm of chromosome 17, are one of the most common genetic lesions in human cancers. The evidence for trisomies of 17q with loss of 17p in some cases of neuroblastoma led us to consider whether p53 mutations might contribute to the onset and progression of this malignancy. In this study, primary tumors from 38 neuroblastoma patients were screened for mutations within the coding exons of the p53 gene by single-strand conformation polymorphism analysis, and potential mutations were further analyzed by nucleotide sequence analysis. Previously described sequence variations were detected in many of the tumors, including a silent polymorphism at codon 213 (CGA to CGG) and the nontransforming Pro to Arg substitution at codon 72 (CCC to CGC). However, no other sequence variations were detected within the coding portions of the p53 gene. This finding suggests that p53 mutations do not contribute to the etiology of neuroblastoma and that the chromosome 17 alterations observed in neuroblastoma involve genes which are distinct from the p53 locus.

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Year:  1993        PMID: 8221661

Source DB:  PubMed          Journal:  Cancer Res        ISSN: 0008-5472            Impact factor:   12.701


  55 in total

1.  N-myc promotes survival and induces S-phase entry of postmitotic sympathetic neurons.

Authors:  Kirmo Wartiovaara; Fanie Barnabe-Heider; Freda D Miller; David R Kaplan
Journal:  J Neurosci       Date:  2002-02-01       Impact factor: 6.167

2.  The cyclin-dependent kinase inhibitor p21 (WAF1) is required for survival of differentiating neuroblastoma cells.

Authors:  W Poluha; D K Poluha; B Chang; N E Crosbie; C M Schonhoff; D L Kilpatrick; A H Ross
Journal:  Mol Cell Biol       Date:  1996-04       Impact factor: 4.272

3.  Polyphyllin D, a steroidal saponin in Paris polyphylla, induces apoptosis and necroptosis cell death of neuroblastoma cells.

Authors:  Shunsuke Watanabe; Tatuya Suzuki; Fujio Hara; Toshihiro Yasui; Naoko Uga; Atuki Naoe
Journal:  Pediatr Surg Int       Date:  2017-03-04       Impact factor: 1.827

Review 4.  Therapeutic targets for neuroblastomas.

Authors:  Garrett M Brodeur; Radhika Iyer; Jamie L Croucher; Tiangang Zhuang; Mayumi Higashi; Venkatadri Kolla
Journal:  Expert Opin Ther Targets       Date:  2014-01-06       Impact factor: 6.902

Review 5.  Patterns of oncogene activation in human neuroblastoma cells.

Authors:  R Corvi; L Savelyeva; M Schwab
Journal:  J Neurooncol       Date:  1997-01       Impact factor: 4.130

6.  N-myc amplification and its relationship to experimental therapy.

Authors:  A Livingstone; R J Mairs
Journal:  J Neurooncol       Date:  1997-01       Impact factor: 4.130

7.  Translocation involving 1p and 17q is a recurrent genetic alteration of human neuroblastoma cells.

Authors:  L Savelyeva; R Corvi; M Schwab
Journal:  Am J Hum Genet       Date:  1994-08       Impact factor: 11.025

8.  MicroRNA-125b is a novel negative regulator of p53.

Authors:  Minh T N Le; Cathleen Teh; Ng Shyh-Chang; Huangming Xie; Beiyan Zhou; Vladimir Korzh; Harvey F Lodish; Bing Lim
Journal:  Genes Dev       Date:  2009-03-17       Impact factor: 11.361

9.  Wild-type p53 protein undergoes cytoplasmic sequestration in undifferentiated neuroblastomas but not in differentiated tumors.

Authors:  U M Moll; M LaQuaglia; J Bénard; G Riou
Journal:  Proc Natl Acad Sci U S A       Date:  1995-05-09       Impact factor: 11.205

10.  A novel 1p36.2 located gene, APITD1, with tumour-suppressive properties and a putative p53-binding domain, shows low expression in neuroblastoma tumours.

Authors:  C Krona; K Ejeskär; H Carén; F Abel; R-M Sjöberg; T Martinsson
Journal:  Br J Cancer       Date:  2004-09-13       Impact factor: 7.640

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