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Literature DB >> 8094122

Viral genetic variation: hepatitis B virus as a clinical example.

W Carman¹, H Thomas, E Domingo.

Abstract

Entities: Species

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Year: 1993 PMID： 8094122 DOI： 10.1016/0140-6736(93)90146-8

Source DB: PubMed Journal: Lancet ISSN： 0140-6736 Impact factor: 79.321

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15 in total

Review 1. Viral quasispecies evolution.

Authors: Esteban Domingo; Julie Sheldon; Celia Perales
Journal: Microbiol Mol Biol Rev Date: 2012-06 Impact factor: 11.056

2. Use of PCR in resolving diagnostic difficulties potentially caused by genetic variation of hepatitis B virus.

Authors: F J van Deursen; K Hino; D Wyatt; P Molyneaux; P Yates; L A Wallace; B C Dow; W F Carman
Journal: J Clin Pathol Date: 1998-02 Impact factor: 3.411

3. Hepatitis B virus genomes of chronic hepatitis patients do not contain specific mutations related to acute exacerbation.

Authors: W Li; H Ikematsu; T K Yamaji; Y Chong; J Hayashi; S Kashiwagi
Journal: Dig Dis Sci Date: 2001-10 Impact factor: 3.199

4. In vitro resistance to interferon of hepatitis B virus with precore mutation.

Authors: Yan Wang; Lai Wei; Dong Jiang; Xu Cong; Ran Fei; Jiang Xiao; Yu Wang
Journal: World J Gastroenterol Date: 2005-02-07 Impact factor: 5.742

5. Nucleotide sequence analysis of precore and proximal core regions in patients with chronic hepatitis B treated with interferon.

Authors: T Laskus; J Rakela; D H Persing
Journal: Dig Dis Sci Date: 1995-01 Impact factor: 3.199

Viral genetic variation: hepatitis B virus as a clinical example.

Review 1. Viral quasispecies evolution.

2. Use of PCR in resolving diagnostic difficulties potentially caused by genetic variation of hepatitis B virus.

3. Hepatitis B virus genomes of chronic hepatitis patients do not contain specific mutations related to acute exacerbation.

4. In vitro resistance to interferon of hepatitis B virus with precore mutation.

5. Nucleotide sequence analysis of precore and proximal core regions in patients with chronic hepatitis B treated with interferon.

6. The enhancer I core region contributes to the replication level of hepatitis B virus in vivo and in vitro.

7. Long term protection after hepatitis B vaccination.

8. Nucleotide sequence analysis of the precore region in patients with spontaneous reactivation of chronic hepatitis B.

9. Antisense therapy of hepatitis B virus infection.

10. Detection of S-HBsAg Mutations in Patients with Hematologic Malignancies.