Literature DB >> 7983640

A comparison of the performance of different methods of disease classification for rheumatoid arthritis. Results of an analysis from a nationwide twin study.

A J MacGregor1, S Bamber, A J Silman.   

Abstract

OBJECTIVE: We have assessed the importance of the distinction between classification criteria for rheumatoid arthritis (RA) that recognize the presence of currently active disease from those that, in addition, incorporate evidence of past disease activity in ascertaining disease occurrence. We applied 7 classification schemes to a population of twins with inflammatory arthritis to determine (a) the number of individuals classified as RA positive by each scheme and hence the effect on the estimate of disease concordance in the twins and (b) their performance in correctly assigning a diagnosis compared with a physician's opinion.
METHODS: The schemes assessed were the 1958 ARA (Rome) criteria which detect active disease, the 1966 New York (using both the 2/4 and 3/4 published cutoffs) which detect "ever" disease and 4 variants of the 1987 ARA criteria. These were the 4/7 and decision tree approaches applied on the basis of the relevant features (1) being present at the time of the study and (2) being present ever and allowing current joint deformity to substitute for absent joint swelling.
RESULTS: In all, 283 individuals with a history of joint swelling were assessed, 255 of whom were considered to have RA by their physician. Criteria used to recognize "current" RA identified only about 70% of those which recognized RA ever. These differences in ascertainment level produced a marked effect on the monozygotic twin RA concordance estimates with percentages ranging from 10 to 18%. The results from receiver operating curves confirmed that criteria used to assess only current RA were too insensitive to be of value. Of the criteria that recognized RA status ever the 1987 ARA performed best overall.
CONCLUSION: The use of classification methods that incorporate past as well as current evidence of disease activity is essential to avoid important misclassification in epidemiological and family studies. The 1987 criteria, applied retrospectively and allowing joint deformity to substitute for swelling, are of enhanced value over other existing schemes.

Entities:  

Mesh:

Year:  1994        PMID: 7983640

Source DB:  PubMed          Journal:  J Rheumatol        ISSN: 0315-162X            Impact factor:   4.666


  39 in total

1.  Epidemiology of rheumatoid arthritis in Turkey.

Authors:  Nurullah Akkoc; Servet Akar
Journal:  Clin Rheumatol       Date:  2006-01-06       Impact factor: 2.980

2.  Linkage of cytokine genes to rheumatoid arthritis. Evidence of genetic heterogeneity.

Authors:  S John; A Myerscough; A Marlow; A Hajeer; A Silman; W Ollier; J Worthington
Journal:  Ann Rheum Dis       Date:  1998-06       Impact factor: 19.103

3.  The prevalences of some rheumatic diseases in western Turkey: Havsa study.

Authors:  Necati Cakır; Ömer Nuri Pamuk; Emine Derviş; Neşe Imeryüz; Haşim Uslu; Ömer Benian; Edip Elelçi; Genco Erdem; Fatma Oğuz Sarvan; Mustafa Senocak
Journal:  Rheumatol Int       Date:  2011-01-13       Impact factor: 2.631

4.  Unexpected HLA haplotype sharing in dizygotic twin pairs discordant for rheumatoid arthritis.

Authors:  D Jawaheer; A J MacGregor; P K Gregersen; A J Silman; W E Ollier
Journal:  J Med Genet       Date:  1996-12       Impact factor: 6.318

5.  Relative importance of genetic effects in rheumatoid arthritis: historical cohort study of Danish nationwide twin population.

Authors:  Anders J Svendsen; Niels V Holm; Kirsten Kyvik; Per Hyltoft Petersen; Peter Junker
Journal:  BMJ       Date:  2002-02-02

6.  Use of supplementary phenotype to identify additional rheumatoid arthritis loci in a linkage analysis of 342 UK affected sibling pair families.

Authors:  Bamidele O Tayo; Yulan Liang; Arpad Kelemen; Austin Miller; Maurizio Trevisan; Richard S Cooper
Journal:  BMC Med Genet       Date:  2009-12-21       Impact factor: 2.103

7.  Investigation of potential non-HLA rheumatoid arthritis susceptibility loci in a European cohort increases the evidence for nine markers.

Authors:  Darren Plant; Edward Flynn; Hamdi Mbarek; Philippe Dieudé; François Cornelis; Lisbeth Arlestig; Solbritt Rantapää Dahlqvist; George Goulielmos; Dimitrios T Boumpas; Prodromos Sidiropoulos; Julia S Johansen; Lykke M Ørnbjerg; Merete Lund Hetland; Lars Klareskog; Andrew Filer; Christopher D Buckley; Karim Raza; Torsten Witte; Reinhold E Schmidt; Jane Worthington
Journal:  Ann Rheum Dis       Date:  2010-05-24       Impact factor: 19.103

8.  Newborn infant characteristics and risk of future rheumatoid arthritis: a twin-control study.

Authors:  Anders J Svendsen; Kirsten O Kyvik; Gunnar Houen; Christian Nielsen; René Holst; Axel Skytthe; Peter Junker
Journal:  Rheumatol Int       Date:  2013-11-05       Impact factor: 2.631

9.  Identification of AF4/FMR2 family, member 3 (AFF3) as a novel rheumatoid arthritis susceptibility locus and confirmation of two further pan-autoimmune susceptibility genes.

Authors:  Anne Barton; Steve Eyre; Xiayi Ke; Anne Hinks; John Bowes; Edward Flynn; Paul Martin; Anthony G Wilson; Ann W Morgan; Paul Emery; Sophia Steer; Lynne J Hocking; David M Reid; Pille Harrison; Paul Wordsworth; Wendy Thomson; Jane Worthington
Journal:  Hum Mol Genet       Date:  2009-04-09       Impact factor: 6.150

10.  Combined effects of three independent SNPs greatly increase the risk estimate for RA at 6q23.

Authors:  Gisela Orozco; Anne Hinks; Steve Eyre; Xiayi Ke; Laura J Gibbons; John Bowes; Edward Flynn; Paul Martin; Anthony G Wilson; Deborah E Bax; Ann W Morgan; Paul Emery; Sophia Steer; Lynne Hocking; David M Reid; Paul Wordsworth; Pille Harrison; Wendy Thomson; Anne Barton; Jane Worthington
Journal:  Hum Mol Genet       Date:  2009-05-05       Impact factor: 6.150

View more

北京卡尤迪生物科技股份有限公司 © 2022-2023.